genetic and neural explanations of OB Flashcards

1
Q

what are the different components of the genetic explanation

A

twin studies (inc adoption studies)
diathesis stress and epigenetics
candidate gene studies

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2
Q

what do genetic exps assume: that criminality is the result of …

A

an inherited characteristic
proposing that one or more genes predispose individuals to criminal B

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3
Q

what are the examples of twin studies

A

lange
christiansen
raine

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4
Q

lange twin study: investigated … MZ and …. DZ twins, where one of the twins…

they then explored how many of them…

and what did they find and what does this conclude

A

13mz
17dz

where one of the twins in each pair had served time in prison

and then explored how many of them had a co-twin who had also spent time in prison (concordance rate)

found no with cotwin in prison:
10 for mz
2 for dz

therefore suggests that genetic factors must play a predominant role in OB AND the more genes shared the more similarity in conviction

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5
Q

what are limitations of lange’s twin study

A

the sample size is small and so there is a lack of generalisability

they judged whether the twins were mz/dz based off appearance and not off a dna test and so this could lead to a lack of validity. (there are some mz twins who dont look the same and so could be thought of as dz)

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6
Q

christiansen explored the concordance rate of offending in mz and dz twin pairs

they had …. mz pairs and …. dz pairs
concordance rates were…

and what does this support the view of…
but what is the problem with these results

A

87 mz pairs , ccr = 33%
147 dz pairs, ccr = 12%

supports he view that OB may have a genetic component

BUT you would expect mz twins to have a higher if not 100% ccr and so this suggests that other factors have a role and it isnt due to genetics entirely

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7
Q

raines twin evidence, raine reviewed delinquent B of twins and found what ccr for mz and dz

what are limitations of this

A

mz ccr- 52%
dz ccr- 21%

delinquent b is very broad and so this was not operationalised, so the difference in rates could be due to type of delinquency/how it was measured, delinquent not necessarily convicted so the difference could be due to convicted vs not

the sample was also very small so cant necessarily generalise

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8
Q

what are general lims of twin studies: cant separate

A

the role of genetics and the E and MZ have a more similar E and so this doesnt represent how others see the world, dz because they look different they may be treated different and so that could be a reason the ccr was less as they have more different experiences

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9
Q

what are the examples of adoption studies

A

crowe
mednick

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10
Q

mednick studied … adoptees and looked at their risk of OB

what was the % of adopted sons who went on to be criminals

A

with both adoptive and bio parents having criminal convictions: 24.5%

bio crim parents- 20%

crim ad parents- 15%

neither bio nor ad parents had criminal convictions- 13.5%

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11
Q

what are lims of mednick

A

they didnt look at daughters and so by not separating this is could act as confounding variable, ie results could be because sons offend more than daughters.

only petty crime was studied so cant generalise to serious crime

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12
Q

what did crowe find- adoption study

adoptees risk of having a criminal record by age 18 for having either a biological or adoptive parent with a criminal record

A

biological- 50% greater risk
adoptive- 5% risk

suggests that inherited genes are a marginally more significant factor than E influences

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13
Q

what are the lims of adoption studies

A

adoption could happen at any ages, the reason for adoption could be because the parent is in prison, could still be eg visiting biological parent, more traumatic when older &raquo_space; E factors

cant separate all factors so assumes the role of genetics and the E

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14
Q

what is research into candidate genes all about

A

searching genome wide, not family based, for certain genes that predispose an individual to ob

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15
Q

who provides evidence for candidate genes

A

brunner
tilhonen

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16
Q

tilhonen- a finnish study of …. offenders found evidence of abnormalities (…..) on two genes that may be associated with …….

what are the genes and what do they control in relation to ob

A

900 offenders
low activity
violent crime

MAOA gene- which controls serotonin and dopamine and is thought to be linked to aggressive B

CDH13 gene- thought to be linked to substance abuse and attention deficit disorder > impulsive > dont think about consequences/lack of inhibition > ob

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17
Q

tilhonen- those with …… were …. times more likely to have a history of repeated …

A

defective genes
13x
violent behaviour

high risk combo

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18
Q

tilhonen- estimated that between … - … % of all violent crime in finland may be due to …..

A

5-10%

abnormalities in the MAOA and CDH13 genes

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19
Q

limitation of tilhonen:

A

not be replicated so dont know whether its valid or not

20
Q

brunner: researched … male members of a … family with a history of .. and … criminal Bs such as … and ….

brunner analysed the … of these men and found that they shared a particular gene that led to abnormally low levels of …

A

28
dutch
impulsive and violent
rape and attempted murder
dna
MAOA

21
Q

what does the diathesis stress model suggest with regard to OB

A

it is the modern understanding of genetic influence

no longer 1 or few genes on their own will determine ob. tendency towards ob may come through the combo of genetic predisposition and bio or psych trigger

eg being raised in a dysfunctional E or having criminal role models

22
Q

what is the role of epigenetics in the diathesis stress model of crime

A

proposes an interplay where genes are “switched on or off” by epigenomes- which in turn have been affected by E factors

this can exp lack of CCR in mz

23
Q

where does evidence for diathesis stress model come from

A

caspi and mednick

24
Q

caspi used data from a …. study in NZ that has followed …. people from where they were babies in the ….
they assessed …..B at age 26

they found …% of those men with low ……. had experienced ….. when they were babies but were responsible for …..% of violent convictions

A

longitudinal
100
1970s

antisocial B
12%
low MAOA genes
exp maltreatment
44%

so maltreatment is an e trigger that causes maoa gene to be switched off

25
general lims of genetic explanations
lims with supporting research evidence for violent crime but not non violen criminality not 100% explained
26
apply determinism to genetic exp
determinist- no free will, legal system based on the premise that criminals have a personal and moral responsibility for their crimes, and only in extreme mental cases can someone plead they werent acting under their own free will. this raises ethical issues with what soc does with people who are suspected of carrying criminal genes and implications for sentencing
27
apply reductionism to genetic exp
reductionism- wrong to reduce down to genetics because it is actually multiple factors interacting, criminality is complex, criminality does appear to run in families, but so does emotional instability and mental illness for eg, this makes it difficult to untangle the effects of genes and neural influences from other possible factors
28
what are the components of the neural exp of OB
regions of the brain mirror neurones neurotransmitters
29
what is APD
antisocial personality disorder (formerly psychopathy) and most evidence for neural exp comes from offender with ADP. characterised by lack of empathy, reduced emotional responses, and it characterises many criminals
30
what is the evidence regions of the brain: raine cited ... brain imaging studies showing ... also raine found ...% reduction in prefrontal cortex
71 showing offenders (murderers, psychopaths, violent individuals) have decreased functioning in the prefrontal cortex 11% reduction in volume of grey matter in prefrontal cortex of people with APD compared to controls
31
what is the prefrontal cortex involved in
regulating emotion and controlling moral B, a decrease in activity = impulsiveness and loss of control
32
what is the evidence regions of the brain: raine found that ... who were found .... limbic system
murderers who were found not guilty by insanity, compared to control, were found to have abnormal asymmetries in limbic system, especially the amygdala having reduces activity on the left and increased on the right
33
what is the limbic system responsible for
it is a set of subcortical structures including the hypothalamus, amygdala. it is linked to emotion and motivation
34
what are mirror neurones
an exp that says that criminals with APD can exp empathy but more sporadically then everyone else christian keysers found that criminals could empathsise when asked to (when shown a video of a person in "pain") and this activates an empathy reaction suggesting APD can have a neural switch controlled by mirror neurones whereas a normal brain has empathy permanently on
35
what is the evidence for neurotransmitters: serotonin, seo suggested that ..
low serotonin levels predispose individuals to impulsive aggression > ob, this is because low S > lack of inhibition of impulsive aggressive urges by the prefrontal cortex dopamine hyperactivity may enhance this effect
36
what is the evidence for neurotransmitters: noradrenaline
very high OR very low noradrenaline levels is associated with aggression, violence and criminality (Wright) very high: causes activation of the sns and the f/fr which leads to aggression very low: reduces ability to react to perceived threats
37
what is the evidence about the relationship between brain injury and criminality
is it commonly observed that criminals report having some head injury (in general 8.5/% of the US pop have a brain injury compared to 60% in US prisons (harmon) therefore brain differences may be due to nurture, or they may be inherited (nature)
38
strength of neural exp: rwa, methods of treatment
if low serotonin levels are related to increased aggressiveness in criminals, could be given special diets that enhance their serotonin and so aggression is decreased artificial sweeteners are high in phenylalanine and low in tryptophan both of which make the production of serotonin difficult therefore this suggests that drugs and or changes in diet could be used to help some individuals
39
limitation of neural exp: it is based on research into aggression rather than OB (what are the impacts of this generally in terms of the use of the research)
eg for studies into neurotransmitters it is aggressiveness being studied NOT criminality so validity is reduced. also some studies use non-human animals which undermines the potential relevance of the info in relation to understanding OB also with genetic evidence, there is not 100% correspondence with any of the brain or neurotransmitter. therefore the data cannot be used to predict who might become an offender and the research cannot be generalised to non-violent crimes
40
limitation of neural exp: issues about c +e, one issue is about whether abnormalities
one issue is about whether abnormalities in regions of the brain or levels of neurotransmitters are the cause of OB, the result of OB or just an intervening variable. research only highlights a correlation between head injuries and later criminality. eg someone who grew up in a violent household might be more likely to suffer head injury, so the link between head injury and OB could be because of a violent childhood therefore neural exps only highlight an association not a causal link with crime
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