Myocardial infarction III Flashcards

(32 cards)

1
Q

do arrhythmias occur during STEMIs, NSTEMIs, or both?

A

both

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2
Q

what is the most common type of arrhythmia seen with MI?

A

ventricular premature

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3
Q

what is done for PVCs?

A

correct electrolytes

beta blockers

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4
Q

when is accelerated idioventricular rhythm observed?

A

shortly after successful reperfusion

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5
Q

accelerated idioventricular rhythm is a result of what type of conduction abnormality?

A

enhanced automaticity of purkinje fibers

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6
Q

VT induced by ischemia has what morphology?

A

polymorphic

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7
Q

VT induced by scarring has what morphology?

A

monomorphic

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8
Q

what is the significance of having late VT/VF (after 48 hours) following MI?

A

increased risk of sudden cardiac death

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9
Q

what are the high risk factors for sudden cardiac death?

A

late VT/VF

EF less than 35% (strongest factor)

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10
Q

what is the strongest risk factor for sudden cardiac death?

A

EF less than 35% when leaving the hospital

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11
Q

inferior MIs typically result from a block at what level?

A

AV node

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12
Q

what are three causes of inferior MIs due to AV nodal block?

A

high parasympathetic tone
local accumulation of K, adenosine
AV node ischemia

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13
Q

what is the best management option for acute MI phase of heart failure?

A

vasodilators (NTG unless hypotensive)

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14
Q

what are the three management options for post acute phase of heart failure?

A

diuretic
ACE inhibitor
aldosterone antagonists (spironolactone, epleronone)

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15
Q

what is the treatment for cardioembolism?

A

anticoagulation with warfarin

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16
Q

what type of pain characterizes pericarditis?

A

pleuritic, can radiate to trapezius

17
Q

what is the treatment for pericarditis?

A

aspirin (650 mg for 4-6 hours)

18
Q

what treatments should be avoided in pericarditis?

A

NSAIDs, steroids

19
Q

the syndrome for late pericarditis goes by what name?

A

dressler’s syndrome

20
Q

what is dressler’s syndrome?

A

(late) pericarditis plus systemic symptoms (malaise, arthralgias, fever, pleural / pericardial effusions, high sedimentation rate, CRP)

21
Q

what is the mechanism type for dressler’s syndrome?

22
Q

what is the treatment for dressler’s syndrome?

A

aspirin, colchicine

23
Q

how does auscultation of a VSD differ from mitral regurg?

A

in VSD the murmur is found at the LLSB as opposed to the apex and typically louder

24
Q

acute mitral regurg is found primarily in the setting of inferior MI involving which papillary muscle?

A

posteromedial

25
what is the best diagnostic test for mitral regurg?
echo or TEE
26
how can VSD be differentiated from papillary muscle rupture?
VSD - loud holosystolic murmr as LSB MR - may be faint, more commonly at apex VSD - can lie flat MR - cannot lie flat
27
what are the risk factors for free wall rupture?
``` older female history of HTN first MI absent reperfusion ```
28
echo will show what features for free wall rupture?
pericardial effusion, tamponade
29
true aneurysm
all three layers are present | bulges out
30
which layer holds a pseudoaneurysm in place?
epicardium
31
which type of aneurysm has a higher risk of rupture?
pseudoaneurysm
32
what are the goals of an intra-aortic pump?
1. augments coronary blood flow during diastole 2. decreases afterload during systole by deflating at onset of systole 3. reduces myocardial ischemia by both mechanisms