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Flashcards in Neuro Deck (155)
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31

What happens in posterior cord syndrome?

Loss of light touch, pressure, vibration and propioception from dermatomes below level of lesion but other sensory and motor functions remain intact

32

What occurs in large central cord lesion?

Loss of everything but sacral region can be spared.

33

Where are the second neruon cell bodies located in the DC/ML system?

Caudal medulla, axons dessucate as internal arcuate fibers
axons form medial leminiscus through rostral medulla pons ad midbrain
terminate in VPL

34

What occurs with lesion of medial leminiscus?

Loss of light touch, pressure, vibrationa nd propioception from dermatomes below lesion on cotnralateral side of body (medial medullary system)

35

The third neuron for DC/ML system are located where?

Cell body in VPL(ventral posterior lateral nucleus) of thalamus
axons pass through posterior limb of interal capsule
terminates in SI cortex

36

What is teh difference between VPL ad VPM in the THalamus?

VPL(ventral posterior lateral nuclues) sensation from body
VPM (Ventral posterior medial nucleus) sensation from body

37

Somatotopy occurs how in hte the thalamus?

most medial is face goes to foot as move laterally

38

How do axons from thalamus fan out?

As the corona radiata, fiibers from VPL and VPM pass to SI cortex

39

What occus with lesions of thalamus or SI cortex?

Loss of sensation from contralateral half of body

40

What brodmann area is responsible for limb movement?

area 3a

41

What brodmann area is responsible for basic tactile information

area 3b

42

What brodmanna rea is responsible for motion and direction of movment of objects?

area 1

43

What brodmann area is responsible for limb position, shapes of objects

area 2

44

What is the relationship between SI and SII cortex?

Located along upper border of Sylvian fissure and insular cortex, SI neurons project to SII cortex

45

Parietal association corticiess, what is a unimodal association cortex?

vision, auditory or somatosensory and a lesion causes agnosias (inability recognize object)

46

what is multimodal parietal association cortices?

combine sensation with motivation, attention, relevane
lesions lead to contralateral neglect

47

Anesthesia means what?

lack of sensation

48

analgesia means?

lack of pain

49

hyperalgesia means what?

increased pain from normally painful stimulus

50

allodynia

pain froma normally non-painful stimulus

51

hypoalgesia means what?

Decreased pain sensation

52

Pruritus means what?

itching

53

nociceptive pain is what type of pain?

Pain resulting from tissue damage
well localized, throbbing quality
typically responds to NSAIDS

54

What type of pain is neuropathic pain?

Pain directly from damage to nerves
often has burning, lancinating, electrical qualty
resistant to NSAIDS and opoids

55

What sensation does the anterolateral system transmit?

Pain and temperature sensation to higher brain levels

56

What is the input to the anterolateral system?

noxious mechanical, thermal, or chemical stimulus to free nerve endings of Adelta or C fibers of nociceptors

57

Adelta fibers mediate what type of pain?

Mediate initial pain which is immediate, short-lasting, pricking quality

58

C fibers mediate what type of pain?

delayed, long lasting, burning quality

59

Where do central processors of nociceptors enter as part of teh anterolateral system?

Enter spinal cord dorsal horn, and synapse onto second order spinal neurons in lamina I/II. Some nociceptors synapse in lateral endge of spinal cord and a few near central canal.

60

What chemical synapse is involved at the second order spinal neruon synapse in anterolateral system?

Chemical synapses involves glutamage and substance P