Path 2 Flashcards by Trent Tamate

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“bread & butter”, adhesions

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Fibrinous pericarditis

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hemorrhagic pericarditis (malignancy, TB)

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bacteria, fungi

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dilated cardiomyopathy, most common,

etiology: etoh & drugs, genetics

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fibrous scar formation in cardiomyopathy

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myocytes arranged haphazardly

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hypertrophic cardiomyopathy

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restrictive cardiomyopathy (amyloidosis)

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restrictive cardiomyopathy (hemochromatosis)

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acute bacterial endocarditis, staph aureus & other virulent bacteria, large friable vegetations on valve cusps

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subacute bacterial endocarditis, strep viridans & other less virulent bacteria

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vegetations on valve leaflets (bacterial endocarditis)

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clinical presentation of bacterial endocarditis

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clinical presentation of bacterial endocarditis

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small vegetations on valve cusps in RF

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aschoff bodies in myocardium (Acute RHD)

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aschoff bodies, myocarditis in acute RHD

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endocarditis dominates in chronic RHD

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fishmouth mitral stenosis in chronic RHD

fishmouth valves, mitral stenosis in chronic RHD

Non Bacterial Thrombotic Endocarditis - d/t hypercoagulable states e.g. cancer, chr illness, trauma from indwelling catheter - less # of vegetations, non destructive, but friable

Non Bacterial Thrombotic Endocarditis -pink fibrin, no inflammation or bacteria

Libman-Sacks Endocarditis d/t SLE -small sterile vegetations, trigger inflammation, can occur anywhere

Types of vegetations