Posterior Pituitary - Unit 1 Flashcards

1
Q

What hormone comes from the posterior pituitary?

A

Antidiuretic Hormone (ADH/Vasopressin)

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2
Q

What does ADH do?

A

Increases water reabsorption/prevent water loss through urination.

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3
Q

When is ADH released?

A

Osmoreceptors in the hypothalamus sense increased osmotic pressure and baroreceptors (heart and vessels) sense volume depletion.

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4
Q

When too much ADH is secreted by the post.. pit gland, what happens?

A

Excess fluid is reabsorbed into the vascular system.

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5
Q

What happens with SIADH?

A

Water retention, increased vascular volume and decreased sodium level.

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6
Q

What physical assessment things might be find with someone who has SIADH?

A

Crackles, confusion, lethargy, concentrated urine because water is in the body.

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7
Q

What are some interventions for SIADH?

A

Fluid restriction, promote water excretion (sodium sparing diuretics), sodium replacement, teaching and safety, providing oral care for dry mucous membranes, I/O

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8
Q

When preparing to administer meds through an NG tube, the nurse should dissolve the meds in water?

A

Saline!

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9
Q

What is a patient with ADH deficiency at risk for?

A

Dehydration!

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10
Q

What is diabetes insipidus?

A

Excreting large amounts of urine!

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11
Q

Diabetes Insipidus - primary and secondary. What are they?

A

Primary - hypothalamus or pituitary defect causing decreased ADH production or release.

Secondary - Other processes that reduce ADH production (trauma, tumor, infection, surgery, etc.)

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12
Q

What are some assessment findings for DI?

A

Water loss, decreased vascular volume, tacky for sure, low BP, dilute urine, etc.

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13
Q

What are some interventions for DI? (drugs, etc)

A

Drugs (Chlorpropamide, which works with ADH in the body, and Desmopressin Acetate, which is synthetic ADH in blood.)

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14
Q

What are some interventions for DI that nurses do?

A

Accurate I&O, daily weight, aim for oral intake = urine output, IV intake, monitor for neuro changes.

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