Respiratory Flashcards

(42 cards)

1
Q

The most important part of the system is the

A

alveoli. That’s where there’s gas exchange. Everything else is just a conduit to the alveoli.

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2
Q

alveoli: there are Type 1 and type 2

Type 2 cells are

A

especially important

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3
Q

Surfactant is to

A

decrease surface tension so that the alveoli can expand, otherwise they’d be stuck together.

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4
Q

Right side of heart has much lower pressure than left side, because

A

it just has to push blood to the lungs which shouldn’t have a lot of pressure (unless there’s disease).

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5
Q

Oxygen does not go into solution, that’s why we need

A

hemoglobin

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6
Q

CO2 however can go into

A

solution

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7
Q

Under normal conditions alveoli and capillaries are in

A

direct contact.

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8
Q

Cyanosis is a very very

A

late sign

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9
Q

Dyspnea is subjective,

A

patient tells you if it happens.

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10
Q

Orthopnea

A

dyspnea when they’re laying down.

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11
Q

Paroxysmal nocturnal dyspnea-

A

wake up at night short of breath.

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12
Q

Assessing for minor heart failure

A

You’re laying down and then all the blood comes up from your legs and can cause minor heart failure, so ask how many pillows do you need to sleep with and do you wake up short of breath.

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13
Q

Kussmaul-

A

sign of metabolic acidosis - blowing off CO2

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14
Q

Cheyne-stokes -

A

can see it in very old and all ____?

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15
Q

Hyperthermia ____ the bond between hemoglobins and O2

A

weakens

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16
Q

Larger the A-a gradient, the less likely it is the

A

O2 is gonna help them.

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17
Q

tension pneumothorax.

A

This is when not only is the one lung shrinking, but it keeps getting pushed because the amount of air keep building which squashes the good lung and the great vessels. Trachea also shifts over. BP drops. Can happen in COPD, ventilator patients. treatment is to release the air that’s causing the continued tension.

18
Q

Friction rub is usually

A

inflammation of the pleura

19
Q

Flail chest

A

broken ribs causes a section of the chest to move parodoxically

20
Q

ARDS-

A

fluid accumulation to the lungs between alveolus and capillary, no surfactant production, so you have trouble ventilating, you can’t have O2 exchange.

21
Q

ARDS- you initially have

A

resp alkalosis but later get resp acidosis. There’s also a bunch of other manifestations.

22
Q

Absent BS on one side, shock, distended jugulars, tracheal deviation

A

Tension pneumothorax

23
Q

Multiple causes including nasal or tonsillar hypertrophy, obesity, genetic predisposition

A

obstructive sleep apnea

24
Q

Several contiguous ribs broken 2-3 or more places

25
Indication of LV failure, CHF
rales
26
Due to decrease RR or decreased tidal volume, increased pCO2 (and hypoxia)
Hypoventilation
27
Can cause respiratory failure (MG, GB, ALS, SCI, etc)
Neuromuscular patho
28
Post-op atelectasis, aspiration pneumonia, Ventilator-associated pneumonia
Considered iatrogenic
29
Interstitial edema, shunting, decreased compliance, decreased surfactant, refractory hypoxemia
Patho of ARDS
30
Night sweats, cough, weight loss, hemoptysis, fever
Tuberculosis
31
Air, blood, exudate between pleural layers
“Collapsed” lung
32
heard first on expiration, worse if inspiration
Wheezing
33
PE, CHF, hypoventilation, pneumonia, shock
Examples of V/Q mismatch
34
Hypoxia, shock, fever, anxiety, pain, acidosis
Causes of hyperventilation
35
Bronchial responsiveness increased, inflammation
Asthma
36
Blood: right ventricle to left ventricle without adequate O2 uptake
Pulmonary shunting (physiologic)
37
Increased mucus formation, air trapping in alveoli
Emphysema
38
Bronchodilation, increased volume
SNS pulmonary effects
39
Pulmonary fibrosis, COPD, ARDS, pneumonia, age
Cause decreased lung compliance
40
Can be idiopathic or associated with lung diseases
Pulmonary hypertension
41
Check ET tube placement; check perfusion
EtCO2 (capnography)
42
200 times more affinity to Hbg than O2
Carbon monoxide (CO)