Supraventricular Tachycardia Flashcards

1
Q

another name for supraventricular tachycardia

A

narrow complex tachycardia

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2
Q

is SVT a diagnosis

A

no
SVT is an umbrella term for any cause of rapid heart rate originating from above or within the atrioventricular node

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3
Q

two main categories of SVT

A

focal tachycardais
re-entrant tachycardias

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4
Q

focal tachycardias

A

a tachycardia that originates from a single point (or points) in the atrium or AV node

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5
Q

sinus tachycardia

A

this counts as a focal tachycardia
the ‘focus’ is the sinoatrial node
usually regular

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6
Q

atrial tachycardia

A

a different focus in the atrium takes over from the sinoatrial node resulting in abnormal p waves preceding QRS complexes
often seen in patients with chronic lung disease
normally > 100bpm and regular
p waves will be positive in the inferior leads as they originate from the top of the atrium and propagate towards the inferior lead

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7
Q

atrial tachycardia is often seen in patients with

A

chronic lung disease

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8
Q

multifocal atrial tachycardia

A

the p waves will have different morphologies as the atrial focus changes from beat to beat

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9
Q

junctional rhythms

A

the impulse originates from the AV node and propagates to the atrium and the ventricles simultaneously
p waves are not visible because they are buried in the QRS complex

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10
Q

management of inappropriate sinus tachycardia

A

can be slowed using bet blockers or ivabradine (selective sinus node blocker), however it is usually best left alone

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11
Q

management of appropriate sinus tachycardia

A

may be due to concurrent sepsis, anaemia, thyrotoxicosis, pain
should be left alone and the underlying cause treated

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12
Q

management of atrial tachycardias

A

can usually be rate controlled with beta-blockers or calcium channel blockers

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13
Q

re-entry tachycardias - atrial flutter

A

well known for it’s sawtooth baseline
classified as a macro-re-entrant tachycardia
this means there is a single re-entry circuit around the atrium which stimulates the AV node every time it passes

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14
Q

Typical vs. atypical atrial flutter

A

typical atrial flutter runs nit-clockwise around the right atrium and across the cavotricuspid valve isthmus
atypical flutter can be clockwise in the right atrium, in the left atrium or around sites of previous surgery and can be difficult to identify as they lack the typical sawtooth appearance

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15
Q

the sawtooth appearance of atrial tachycardia in best seen in

A

the inferior leads becaause it is caused by the circiut alternately heading toward and way from the leads as it speeds round the atrium

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16
Q

atrial fibrillation

A

micro-re-entrant tachycardia
lots of tiny circuits contributing to chotic random fibrillation of tha atrium
irregularly regular

17
Q

AVNRT (atrio-ventricular node re-entrant tachycardia)

A

activates ventricles and atria almost simultaneously
pseudo R wave: retrograde p wave superimposed on the QRS complex
rapid regular tachycardia
treatment involves complete transient AV node blockade

18
Q

AVRT (atrio-ventricular re-entrant tachycardia)

A

this requires two pathways: the normal AV conduction system and an accessory pathway (AP)
accessory pathways my conduct antegrade (atria to ventricles) retrograde (ventricle to atria

19
Q

orthodromic AVRTs

A

down the AV node and up the accessory pathway

20
Q

antidromic AVRTs

A

down the accessory pathway and up the normal AV conduction

21
Q

management of AVRTs

A

electrically cardiovert the patient back to sinus rhythm
flecanide can be used to prevent AVRTs but ablation is usually the mainstay of treatment given risk of VF and sudden death

22
Q

what is the treatment of choice in a haemodynamically unstable patient due to the underlying SVT

A

cardioversion

23
Q

rate control for atrial fibrillaation and atrial flutter

A

beta blockers
calcium channel blockers
digoxin

24
Q

rhythm control for atrial fibrillation and atrial flutter

A

chemical cardioversion (amiodarone, flecanide)
electrical cardioversion (DC shock)

25
Q

ablation for atrial fibrillation and atrial flutter

A

more likely to succeed in atrial flutter than atrial fib
more likely to succeed in paroxysmal AF than persistent AF

26
Q

anticoagulation in atrial fibrillation and atrial flutter

A

according to stroke risk vs bleeding risk (CHADS2 VASc score)

27
Q

AF patients who should not be considered for AF

A

AF is not life-threatening
a patient with well-controlled AF and normal LV function has the same life expectancy as someone in sinus rhythm
symptomatic patients will well rate controlled AF will not feel better or life longer by returning them to sinus rhythm

28
Q

patients who should be considered for cardioversion

A

new-onset AF
symptomatic AF
LV dysfunction secondary to AF
patients with a reversible cause of AF (eg. post-surgical, post-infection)
pateints who have a reasonable chance of maintaining sinus rhythm

29
Q

patients who have a reasonable chance of maintaining sinus rhythm will

A

previous successsful cardioversion - although each subsequent cardioversion becomes less successful
minimal dilatation of the left atrium
no signiificant mitral valve disease

30
Q

management of AVNRT

A

induce AV node blockade
mechanical: vasovagal manouvres, carotid sinus massage
chemical: adenosine, beta blockers, verapamil

31
Q

management of AVRT

A

avoid AV node blockade as this can promote conduction down the AP and AF can convert to VF
electrical cardioversion is the best initial option although flecanide can be used to chemically cardiovert
ablation of the pathway is the curative option

32
Q

what is adenosine

A

a purine nucleoside that, administered intravenously, causes transient complete AV node blockade
it is a useful diagnostic tool as well as a treatment in the setting of narrow complex tahcycardia

33
Q

is adenosine safe in pregnancy

A

yes (its endogenous)

34
Q

can adenosine be used in asthma

A

usually can be used safetly in asthmatic unless they have brittle severe asthma

35
Q

effects of adenosine

A

you will see a few seconds of total AV block

36
Q

if you administer adenosine and you do not see AV block

A

one of two things have occurred:
1. you have not administered the adenosine properly and the adnosine has not reached the heart in time given its very short half life
2. the rhythm is not involving the AV node - it is therefore, ventricular tachycardia