cardio 3 Flashcards

(14 cards)

1
Q

CAD and cause

A

coronary artery diensease caused by atherosclerosis. narrowing/bloackage of CA —>ischemia

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2
Q

what happens when blockage of coronary Artery

A

myocardial infarction —> myocardial ischemia —> hypoxia and tissue damage –> myocardial necrosis —> cells breakdown —> leakage of cellular components —-> impaired ventricular function —> decreased ejection fraction

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3
Q

what happens when narrowing of coronary artery

A

causes angina which is chest pain when O2 demand is higher than supple. can be stable and unstable. when unstable its unpredictable and can be indicator of potential MI.

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4
Q

surronding tissue effects

A

stunning - temp loss
hibernation - longer loss of muscle cells contractility
remodelling - permanent loss

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5
Q

S/S, what to monitor and complications of MI

A

s/s - pain in chest, neck, shoulders, jaw, back and right arm. fatigue, feeling of anxiety, sweating, pallor, nausea, SOB, dyspnoea
monitor - changes in ECG, HR, heart sounds, blood test increase in HR and BP
complications - arrythmia, angina, reinfraction, heart failure

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6
Q

STEMI and NSTEMI

A

ST elevation MI - ST elevated on ECG means there was a full blockage in coronary artery so transmural infarction
Non ST elevation MI - means no ST Elevation on ECG so there is only Patrica occlusion or perfusion restored before necrosis. so subendocardial infarction

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7
Q

management of MI

A

obs
MONA
morphine, oxygen, nitrate and aspirin
ecg
repercussion therapy
- thrombolysis
percutaneous coronary intervention
angiogram and angiplasty
bypass surgery

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8
Q

what causes heart failure and difference between systolic and systolic

A

cardiac dysfunction
systolic - cant contract properly
diastolic - cant relax properly

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9
Q

right sided heart failure and s/s

A

decrease CO into pulmonary circuit –> systemic venous congestion —< venous HT —> peripheral oedema —> kidney enlarged —> liver failure
s/s - juglar vein distention, oedema, pulmary dysfunction

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10
Q

left sided heart failure and s/s

A

decrease CO into systemic circuit —> pulmonary HT and oedema —> impaired gas exchange
s/s - dypnoea and SOB due to HT, CAD and cardiomyopathy

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11
Q

artial fibrillation

A

atria quivers which causes rapid and irregular HR

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12
Q

ventricular tachycardia

A

abnormal rapid signals in ventricle which causes very high HR and decrease CO and BP

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13
Q

ventricular fib

A

vents quiver not contract
no CO so go into cardiac arrest. can defibrillator

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14
Q

asystole

A

no electrical signals and pumping. no shock only cpr

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