Ch3 Neurophysiology Flashcards

(53 cards)

1
Q

What structures form the BBB?

A

Endothelial tight junctions (within the capillaries)

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2
Q

What is the effect of steroids on BBB?

A

Reduce the penetration of small hydrophilic molecules

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3
Q

Which areas are the BBB deficient? (6 areas)

A

Choroid plexus Posterior pituitary Tuber cinereum Area postrema Pineal gland Pre-optic recess

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4
Q

What are the 3 types of oedema?

A

Cytotoxic (no enhancement) e.g. TBI Vasogenic (enhancement) e.g. tumour Ischaemic (delayed enhancement) e.g. stroke

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5
Q

What is the triple flexor response?

A

Seen in UMN lesion in addition to babinski reflex, the knee and hip also flex.

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6
Q

What is the anatomy of the Babinski reflex?

A

S1 dermatome stimulation > tibial nerve > spinal cord L4 to S2 > common peroneal nerve > toe extension

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7
Q

What other tests can be used in addition to Babinski?

A

Schaeffer = pinch achilles; Gordon = pinch calf; Oppenheim = slide knuckles down shin Stronsky = pull 5th toe and allow it to snap back

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8
Q

What does a Hoffmann’s reflex represent?

A

Disinhibition of a C8 reflex i.e. lesion above C8

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9
Q

What proportion of patients with cervical myelopathy have a positive Hoffmann’s?

A

2/3

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10
Q

Where is the primary centre for bladder function?

A

Locus coeruleus of the pons

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11
Q

What is the function of the locus coeruleus?

A

Synchronises bladder contraction with urethral relaxation

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12
Q

How is voluntary cortical micturation controlled?

A

Mesial frontal lobes and genu of CC > pontine locus coeruleus > detrusor contraction and relaxation of the internal urethral sphincter under parasympathetic control. The external sphincter sympathetic tone relaxes through the pudendal nerve.

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13
Q

When is the Babinski reflex negative in SCI?

A

During spinal shock

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14
Q

Where do the bladder efferents run in the spinal cord?

A

Lateral to the dorsal columns

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15
Q

Where are the parasympathetic preganglionic cell bodies?

A

S2-4. The fibres then run via the pelvic splanchnics nerves

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16
Q

Where are sympathetic cell bodies located?

A

T1-L2

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17
Q

Which tract do bladder sensory fibres run in?

A

Spinothalamic tract

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18
Q

What is a neurogenic bladder / areflexia?

A

Lack of sensory input from the bladder wall leads to stretching and overflow incontinence without feeling

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19
Q

What is detrusor hyperreflexia

A

Efferent lesions from cortex to sacrum cause loss of cortical inhibition and reflex contraction of the bladder

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20
Q

How does a cortical lesion affect bladder function?

A

Loss of inhibition of the pontine voiding reflex. Results in involuntary reflex bladder contraction and voiding as sensation is intact. Treat with anticholinergics

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21
Q

How does a complete spinal cord lesion affect bladder function?

A

Initially - spinal shock causes an areflexic bladder and retention. After this the bladder becomes hyperreflexic. Treat with intermittent catheterisation and anticholinergics

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22
Q

When are bulbocavernosus and anal wink reflexes lost?

A

With cauda equina lesion

23
Q

How can the bladder function be evaluated?

A

Urodynamic testing, sphincter EMG and micturating cystourethrogram.

24
Q

How is detrusor hyperreflexia treated?

A

antimuscarinics e.g. oxybutynin, these relax the bladder wall and increase capacity so should be combined with a timed voiding regime.

25
How should you manage patients bladder function post CES?
Intermittent self catheterisation / indwelling. Start Tamsulosin. Check post-void residuals. If \<75ml then no need for catheter.
26
What clinical features are associated with large frontal lobe lesion?
Apathy, abulia, eye deviation if frontal eye fields involved (looks to the side of the lesion!). Involvement of the prefrontal region results in impulsiveness and disinhibited behaviour.
27
What clinical features are seen with a dominant parietal lobe lesion?
Gerstmann's syndrome, aphasia and bilateral astereognosis (inability to identify objects by touch), hemianopsia and neglect
28
What clinical features are seen with a non-dominant parietal lobe lesion?
Spatial memory loss, loss of self awareness (anosognosia) and dressing apraxia, hemianopsia and neglect
29
What are the clinical features of a cerebellar lesion?
Hemispheric = Ataxia in ipsilateral limbs Vermis = Ataxia in trunk
30
What is Gerstmann's syndrome?
Agraphia without alexia (can't write but can read) Digital agnosia (cannot name fingers) Left - right confusion Acalculia Tactile agnosia (cannot tell what objects are in hand) Ideomotor apraxia (cannot carry out a series of verbal commands for a motor actions)
31
What is astereognosis?
Inability to identify objects by feel
32
What is agraphesthesia?
Cannot interpret numbers written by hand. Associated with postcentral gyrus lesions.
33
What is Anton-Babinski syndrome?
Unilateral asomatagnosia - hemibody neglect and lack of awareness of one half of the body. Characterised by: 1. Anosognosia (unaware of deficits) 2. Apathy 3. Allocheiria (unilateral stimulus perceived bilaterally) 4. Dressing apraxia 5. Extinction (unaware of contralateral stimulus when bilateral stimulus applied) 6. Inattention to hemi-visual field
34
How does Tamsulosin work?
Alpha a1 R-antagonist (works for both men and women with bladder outlet obstruction)
35
What tests can be performed for a parietal lobe syndrome?
Ask to read Ask to write Clock face drawing task 3 stage commands (ideomotor apraxia) Ask what finger (digital agnosia) and right or left Ask what object is placed in hands (astereognosis) Kimura Box Test (apraxia) Two-Point Discrimination Test (somatosensory)
36
What is Weber syndrome?
CN3 palsy with contralateral hemiparesis
37
What is Benedikt's syndrome?
CN3 with hemiparesis but inclusion of the red nucleus causing ataxia, coarse intention tremor and hyperkinesia
38
What is Millard-Gubler syndrome?
CN6 and 7 palsy with contralateral hemiparesis due to a lesion in the base of the pons
39
What is Parinaud's syndrome?
Up-gaze palsy Convergence-retraction nystagmus Pupillary near light dissociation (pseudo-Argyll robertson pupil)
40
What is the syndrome of the sylvian aqueduct?
Parinaud's syndrome combined with a downgaze palsy
41
What is an internuclear opthalmoplegia?
Damage to the medial longitudinal fasciculus disconnecting CN3 from CN6; One eye looks laterally but the opposite eye cannot look medially.
42
What is Vernet syndrome?
CN 9, 10 and 11
43
What is Villaret syndrome?
CN 9, 10, 11, 12 and sympathetics
44
What is Collet-Siccard syndrome?
CN 9, 10, 11 and 12 (not sympathetics)
45
What is Jackson syndrome?
CN 10, 11 and 12 palsy
46
What are the causes of jugular foramen syndromes?
Condylar and Jefferson fractures, Cartoid dissection, Skull base tumours and fibromuscular dysplasia
47
What are the parameters that effect ICP?
MAP JVP (neck collar / intrathoracic pressure / paralysis) PaCO2 PaO2 Haematocrit Temperature CMR02 CSF drainage Elevation of the head
48
Draw a graph of CBF vs PaO2 and PaCO2
As pCo2 rises the CBF and ICP rises As pO2 rises the CBF and ICP fall
49
What is the action of Nitrous oxide?
Vasodilation causing increased CBF; DO NOT give nitrous oxide to patients with pneumocephalus as it will cause tension pneumocephalus
50
What is the action of Desflurane on CBF?
Increases CBF and ICP, but also decreases CMR02
51
What is the action of Sevoflurane on CBF?
Increases CBF and ICP, also reduced CMR02; Problem is that it also reduces cardiac output
52
What is the CBF?
50ml/100g/min of brain tissue
53
What proportion of CO goes to the brain?
12%