CVS Session 7 Flashcards

0
Q

Why do electrodes outside the cell ‘see’ two signals with each systole?

A

Can only ‘see’ changing membrane potential therefore one signal for depolarisation and one for repolarisation

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1
Q

In which direction does the large changing electrical field generated by the myocardium travel?

A

Radiating away from the myocytes through the body to the skin

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2
Q

What does an ECG show?

A

Effects of depolarisation
Effects of repolarisation
Spread of electrical field alteration over the heart

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3
Q

How does conduction spread over the atria?

A

Starts at SAN
Spreads over atria to AVN
AVN delays conduction for 120 ms

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4
Q

How does spread of excitation cause ventricular depolarisation?

A

Activity spread down septum then out over ventricular myocardium
Endocardial –> epicardial

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5
Q

What causes depolarisation to spread in the epicardial direction?

A

A uniform thin layer of depolarisation in the endocardial surface

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6
Q

Where does the last signal seen originate from?

A

Activity of cells at base of valves

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7
Q

How does repolarisation occur?

A

After 280 ms it spreads in the opposite direction to depolarisation

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8
Q

Why does repolarisation occur in the opposite direction to depolarisation?

A

To unravel the fibres arranged in a figure of eight pattern that have twisted during depolarisation

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9
Q

What is a view in relation to an ECG?

A

Imaginary direction in which you are looking at the heart depending on the position of the electrode relative to the spread of activity

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10
Q

What creates an upward signal on an ECG?

A

Depolarisation moving towards the electrode

Repolarisation moving away from the electrode

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11
Q

What causes a downward signal on an ECG?

A

Depolarisation moving away from the electrode

Repolarisation moving towards the electrode

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12
Q

What does the amplitude of the signal depend on?

A

How much muscle is depolarising

Vector of movement of excitation

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13
Q

Considering the apex view, why does atrial depolarisation cause a small upwards deflection?

A

Small amount of muscle

Moving towards electrode but not directly

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14
Q

How does excitation spread from the septum?

A

Spreads ~1/2 down septum then out across the axis of the heart

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15
Q

What restricts the amplitude of the signal caused by spread from the septum given that a large amount of muscle is depolarising?

A

Its relative direction

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16
Q

How does excitation spread through the ventricular myocardium?

A

Through ventricular muscle along an axis slightly to the left of the septum

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17
Q

Why does the epicardium repolarise first?

A

Cells in epicardium happen to spontaneously repolarise first

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18
Q

Why is the area under the R wave equal to that under the T wave?

A

Same cells are involved

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19
Q

What does the QRS complex show?

A

Spread of excitation to endocardium and subsequent spread across ventricles

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20
Q

What does the P wave show?

A

Atrial systole

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21
Q

What does the Q wave show?

A

Septal depolarisation spreading to ventricle

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22
Q

What does the R wave show?

A

Main ventricular depolarisation

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23
Q

What does the S wave show?

A

End of ventricular depolarisation

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24
What does the T wave show?
Ventricular depolarisation
25
Why is atrial repolarisation not seen on an ECG?
It is lost in the QRS complex as the signal is small and swamped by ventricular changes
26
Starting from the apex view, as you move the view clockwise around the heart, what happens to the R wave?
Amplitude decreases --> becomes -ve --> becomes more -ve --> neutral --> small +ve --> large +ve
27
How does the amplifier in an ECG utilise a +ve and -ve electrode?
Invert -ve electrode | Add signal to +ve input making signal detectable
28
In which direction does lead I look from?
Left side
29
In which direction does lead II view the heart?
Towards apex
30
In which direction does lead III view the heart?
From the bottom
31
In which plane do the augmented leads measure electrical activity?
Vertical
32
Using clock positions, what views do the augmented leads have of the heart?
``` aVL = 2 o' clock aVF = 6 o' clock aVR = 10 o' clock ```
33
Which plane do the chest leads give views in?
Horizontal
34
Where are the electrodes for V1-3 placed?
``` V1 = 4th intercostal space, right eternal edge V2 = 4th intercostal space, left eternal edge V3 = 5th rib b/w V2 and V4 ```
35
Where are the electrodes for V4-V6 placed?
``` V4 = 5th intercostal space, mid-clavicular line V5 = 5th intercostal space, b/w V4 and V6 V6 = 5th intercostal space, mid axillary line ```
36
What standard rate do all ECG machines run at?
300 squares per minute
37
How can heart rate be calculated?
300/ squares of RR interval | ~12 beats present normally
38
How do you asses the rhythm of a heart trace?
Choose lead where relevant components are most visible to see if R waves are regular/irregular
39
How is the axis determined?
Estimate the direction of arrow that generates the R wave by looking at the most positive leads
40
When are P waves absent?
Atrial fibrillation
41
How long should a normal PR interval be?
~120 ms
42
What 3 different PR interval abnormalities can be seen and what do they indicate?
Prolonged = first degree heart block Erratic = second degree heart block No relationship b/w P and R = third degree heart block
43
What is heart block?
Something that prevents transmission b/w atria and ventricles
44
What type of damage can the QRS complex be used to identify?
Ventricular
45
What does the QT interval indicate?
Length of systole
46
What is the T wave examined for?
To see if repolarisation is in the right direction
47
Why are intervals between R waves irregular in atrial fibrillation?
Pacemaker other than SAN is controlling contraction at a slower than usual rate
48
Why is atrial fibrillation dangerous if the heart continues to function?
Pooling occurs in corners of atria which increases the risk of a clot being pumped into the arteries
49
If the R waves are further apart than the P waves, what does this indicate?
Atria are contracting more frequently than ventricles
50
How is the P wave seen in third degree heart block?
Normal shape but not related to the QRS complex
51
What causes natural axis deviation?
Size of person
52
Why is the normal axis of the heart a single vector pointing slightly left?
Combination of R and L ventricular depolarisation with thicker ventricle wall on left pulling net arrow left
53
How do the limb leads appear in right axis deviation?
``` I = -ve II = small III = +ve ```
54
How do the limb leads appear in left axis deviation?
``` I = +ve II = small III = -ve ```
55
If the net deflection is 0, what does this tell you about the axis?
At right angles to that view
56
What causes bundle branch block?
Damage to conducting pathways altering route of spread of depolarisation
57
What changes are detected on an ECG of a patient with bundle branch block?
QRS complex changes shape - almost always increased width | 'Bunny ears' may be present
58
What causes 'bunny ears' in bundle branch block?
Spread of depolarisation turns around and moves back along path it has just taken due to block --> two R waves generated
59
How is damage to the myocardium identified on an ECG?
Changes to the ST segment
60
When does damage to the myocardium occur?
If there are problems w/perfusion to the myocardium - if it is stressed, dying or dead
61
What does damage to the myocardium affect?
Spread of electrical activity during systole
62
What do extra ST signals indicate?
Extra electrical current generated in systole due to stressed/dying/dead myocardium
63
What does ST depression indicate?
Transient hypoxia
64
Why will the heart rate often be high on an ECG displaying ST depression?
It is investigated by exercise test
65
What does ST elevation indicate?
Dying tissues generate injury currents due to lack of oxygen
66
What three ECG changes may be identified during myocardial infarction?
ST elevation (sometimes) Pathological Q waves Inverted T waves
67
What causes pathological Q waves in MI?
Dead myocardium replaced by fibrous tissue that electrical activity has to flow around
68
How are pathological Q waves identitified on an ECG?
>0.04s wide (1 small square)
69
How would the ECG of someone who has not presented with an Mi show that they had in fact suffered a previous MI?
Pathological Q waves - they persist after other changes are resolved
70
In myocardial infarction, what does the view with the most prominent abnormality help identify?
Which coronary artery is blocked | Whether full/partial ventricular wall thickness affected
71
What type of MI are pathological Q waves seen in?
Full thickness
72
Which leads most commonly show the most prominent abnormality in an MI?
Chest leads