Duplex/Color flow venous imaging Flashcards

(32 cards)

1
Q

Sources for false positive?

A
  • extrinsic compression: tumors, ascites, and pregnancy
  • peripheral arterial disease: decreased venous filling
  • chronic obstructive pulmonary disease: elevated central venous P
  • improper Doppler angle or probe pressure
  • superior vena cava syndrome
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2
Q

Sources of false negative studies?

A

prox obstruction

technically limited studies

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3
Q

May be difficult to thoroughly evaluate what peripheral veins in the lower extremity secondary to vessel size, depth, and course?

A

infra-popliteal

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4
Q

Difficult to thoroughly evaluate what peripheral upper extremity veins secondary to bony structures?

A

subclavian and brachiocephalic/innominate

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5
Q

What is the patient positioning for upper extremity peripheral vein?

A

supine or low fowlers position

arm in pledge position

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6
Q

What settings can be adjusted to maximize color filling and flow patterns?

A
  • adjust color scale to detect slower velocities
  • change wall filters
  • increase color gains
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7
Q

What is another word for compressibility?

A

coaptation

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8
Q

What are the possible venous flow patterns?

A

spontaneous, phasic, augment with distal compression, augment with proximal release

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9
Q

Signal immediately heard at all sites except what vein?

A

posterior tibial

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10
Q

what is the lower extremity phasicity?

A

increase with expiration and decreases with inspiration

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11
Q

what is the upper extremity phasicity?

A

decreases with expiration and increases with inspiration

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12
Q

pulsatile venous flow pattern evident with fluid overload or what?

A

congestive heart failure

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13
Q

What is a technique to collapse the subclavian and innominate?

A

a quick breath through pursed lips should collapse vein

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14
Q

Flow reversal usually in response to a valsalva maneuver or during prox manual compression indicates what?

A

venous refulx

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15
Q

Reflux is identified when reversed flow last more than how long?

A

1 sec

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16
Q

Deep inspiration causes the abdominal and pelvic veins what?

17
Q

Phasic, bi-directional/pulsatile Doppler signals are in what veins?

A

IVC, renal, and hepatic

18
Q

minimally phasic, continuous Doppler signals are in what veins?

A

portal, splenic, and mesenteric

19
Q

There is minimal flow fluctuation in the portal vein and flow is variable in the hepatic veins when?

A

during inspiration

20
Q

If flow is not spontaneous at the CFV, FV, and/or Pop V what could be the cause?

A

obstruction distal to or at the site

21
Q

If flow is not phasic, but rather continuous, what could be the result?

A

a proximal obstruction

22
Q

Where might an obstruction be if the is no augmentation with distal compression seen?

A

obstruction may be between where you are compressing and where you are listening, or slightly more proximal

23
Q

If there is no augmentation with proximal release, where might an obstruction be?

24
Q

If flow increases during proximal compression, what does that signify?

A

venous reflux

25
A compressible vessel with evidence of rauleau formation on B-mode could be what?
normal or suggest proximal obstruction
26
What is the appearance of chronic clot?
- highly echogenic - visible collateralization or recanalization may be evident - vessel not dilated; may retract over time
27
Flow characteristics with chronic clot?
- abnormal Doppler venous signals may be evident, such as continuous, decreased phasicity, or no augmentation - venous reflux lasting >1 sec
28
Where is an IVC interruption device usually placed?
below renal veins and may appear as bright echogenic lines
29
With systemic venous hypertension what is evident?
persistent dilated vessels
30
Increased portal venous pressure can result in what flow alterations?
- reversed flow in portal vein (hepato-fugal) | - collateral development
31
What is Budd-Chiari Syndrome?
- results from hepatic occlusion | - primary site of obstruction may be hepatic vein, sinusoids, or IVC
32
What are the clinical findings of Budd-Chiari Syndrome?
hepatomegaly, abdominal pain, sudden onset of ascites