FEd State Fasting and Starvation 11-4-13 Flashcards Preview

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Flashcards in FEd State Fasting and Starvation 11-4-13 Deck (28):
1

The macronutrients that are used for energy generation are (carbs/protien/fats)

carbs, fats

2

_____bodies are an alternative fuel utilized during starvation.

ketone

3

how many hours go by until your body goes into a fasting state?

greater than4

4

How many days goes by until your body is in a starvation state?

3 days

5

(Fatty acids/proteins/carbs) can also be converted into ketone bodies by the liver.

fatty acids

6

(glucose/carbs/ketone bodies) is the preferred fuel for all organs'tissues

glucose

7

(brain/liver/skeletal muscle/reb blood cells) o can only utilize glucose and therefore need a constant supply of glucose for ATP production.

red blood cells

8

(brain/liver/skeletal muscle/reb blood cells)can utilize glucose and ketone bodies.

brain

9

(brain/liver/skeletal muscle/reb blood cells)can utilize glucose and fatty acids.

liver

10

(brain/liver/skeletal muscle/reb blood cells)can utilize glucose, fatty acids and ketone bodies.

skeletal muscle

11

(brain/liver/skeletal muscle/adipose tissue) utilizes glucose and fatty acids

adipose tissue

12

in teh liver when glycogen stores are saturated:
-fatty acids are converted to (VLDLS/TAGs) then packaged as (VLDLs/TAGs) and deposited into the circulation

(VLDL/TAGs) are taken up by the adipose tissue where tehy are stored as (VLDL/TAGs)

TAGS, VLDLs


VLDL, TAGs

13

T/F liver glygocen stores have a point of saturation but adipose stores do not

T, they are limitless

14

blood glucose levels are maintained by degradation of (liver/adipose tissue) glycogen primary source . an to a lesser extent (carbs/amino acids/fatty acids)

liver , amino acids

15

(cabs/protiens/fats) are broken down to amino acids

protiens

16

1) storage of glucose as glycogen,
2) fatty acid synthesis,
3) storage of fatty acids as TAGs, and
4) protein synthesis in skeletal muscle and other tissues. Through the metabolic effects of insulin, elevated blood glucose levels (induced by a meal) return back to the normal range.
--Is promoted by (glucose/insulin)

insulin

17

1) glucose is used for ATP production (through glycolysis),
2) excess glucose is stored as glycogen (through glycogenesis),
3) when liver glycogen stores are saturated, excess glucose is converted to fatty acids (these fatty acids are then converted into TAGs, packaged into VLDLs and deposited into the circulation for storage as fat in adipose tissue).
--describes metabolic process in the (liver/skeletal muslces/adipose tissue)

liver

18

1) storage of dietary glucose as glycogen (glycogenesis),
2) oxidation of glucose to produce ATP (through glycolysis), and
3) the synthesis of fatty acids (from excess dietary glucose).

--describes what insulin stimulates in the (liver/skeletal muscle/adipose tissue)

liver

19

1) glucose is used for ATP production (glycolysis),
2) excess glucose is stored as glycogen (glycogenesis; these stores can only be used by muscle during exercise), and
3) proteins are synthesized from dietary aminoacids.
--describes metabolic process in the (liver/skeletal muslces/adipose tissue)

skeletal muscle

20

1) transport of plasma glucose into muscle cells,
2) storage of dietary glucose as glycogen (glycogenesis),
3) transport of dietary aminoacids into muscle cells and
4) protein synthesis.

--describes what insulin stimulates in the (liver/skeletal muscle/adipose tissue)

skeletal muscle

21

what are teh two alternative fuels for tissues

fatty acids, ketone bodies

22

what does glucagon do?

1) degradation of glycogen stores (glycogenolysis) and
2) gluconeogenesis.

23

Glucagon doesn't have effect on (muscle/liver) metabolism

muslce

24

does glucagon have effect on brain or RBC?

NO ONLY IN LIVER

25

During starvation and fasting what is the primary source of glucose and what is the secondary ?

primary source is glycogenolysis
secondary source is gluconeogenesis

26

(hyperglycemia/hypoglycemia) This is due to either the absence of insulin, or to abnormal insulin signaling

hyperglycemia

27

(hyperglycemia/hypoglycemia) epinephrine is released and gives rise to adrenergic symptoms (palpitations, tremors, anxiety).

hypoglycemia

28

(RBC/brain/skeletal muslce/adipose tissue) can only use glucose

RBC