5: GINGIVITIS, PERIODONTITIS, PERICORONITIS, PERI-IMPLANTITIS, PERI-IMPLANT MUCOSITIS Flashcards

(105 cards)

1
Q

______
Inflammatory conditions affecting the gums and structures supporting the teeth(gingiva, periodontal ligament, cementum, and alveolar bone) . They range from mild, reversible gingivitis to severe, irreversible periodontitis that can lead to tooth loss.

A

PERIODONTAL DISEASE

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2
Q

OVERVIEW OF MICROBIAL ETIOLOGY OF PERIODONTAL DISEASE
• Caused mainly by ______ at the ______

A

plaque buildup, gumline

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3
Q

Healthy gum crevice
- mildly ______(low oxygen)and protected by ______. GCF contains immune cells like

A

anaerobic, gingival crevicular fluid (GCF), polymorphonuclear leukocytes (PMNLs)

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4
Q

Plaque makes the environment more ______ and ______

A

anaerobic, alkaline

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5
Q

Favors growth of proteolytic bacteria:
• ______
• ______
• ______
These bacteria colonize tooth surfaces and gums which triggers ______ and destroys ______

A

Porphyromonas gingivalis, Prevotella spp., Peptococcus micros, inflammation, supporting tissues

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6
Q

______
• is a reversible inflammation of the gums caused by plaque buildup, which triggers an immune response leading to swelling, redness, and bleeding.

A

GINGIVITIS

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7
Q

GINGIVITIS

PATHOGENESIS
THE ______ LESION
• Develops within 4 days of plaque accumulation

A

INITIAL

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8
Q

GINGIVITIS

PATHOGENESIS
THE ______ LESION
• Shows acute inflammation, blood vessel changes (______), collagen breakdown, increased crevicular fluid, white blood cell (______) infiltration

A

INITIAL, vasculitis, polymorphonuclear leukocyte

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9
Q

GINGIVITIS

PATHOGENESIS
THE ______ LESION
• No clinical signs are visible at this stage

A

INITIAL

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10
Q

GINGIVITIS

PATHOGENESIS
THE ______ LESION
• Dominated by Gram-positive and facultative bacteria, mainly ______

A

INITIAL, streptococci

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11
Q

GINGIVITIS

PATHOGENESIS
THE ______ LESION
• Appears after 7 days

A

EARLY

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12
Q

GINGIVITIS

PATHOGENESIS
THE ______ LESION
• Visible gingival inflammation, dense lymphocyte infiltration, and collagen breakdown

A

EARLY

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13
Q

GINGIVITIS

PATHOGENESIS
THE ______ LESION
• Increased Actinomyces spp., Capnocytophaga, and some Gram-negative anaerobes

A

EARLY

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14
Q

GINGIVITIS

PATHOGENESIS
THE ______ LESION
• Develops over a variable period

A

ESTABLISHED

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15
Q

GINGIVITIS

PATHOGENESIS
THE ______ LESION
• Pocket formation, increased plasma cells, B cells, and neutrophils; lesion may persist for years without deeper tissue damage if plaque remains

A

ESTABLISHED

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16
Q

GINGIVITIS

PATHOGENESIS
THE ______ LESION
• Increased black-pigmented ______ (e.g., Porphyromonas gingivalis, Prevotella intermedia) and ______

A

ESTABLISHED, anaerobes, spirochetes

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17
Q

GINGIVITIS

MANAGEMENT
Treatment is by thorough removal of ______ and ______ deposits, all plaque-retentive factors, and the introduction of good oral hygiene

A

plaque, calculus

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18
Q

GINGIVITIS

MISCELLANEOUS FORMS OF GINGIVITIS:

A

• PREGNANCY GINGIVITIS
• DIABETES MELLITUS–RELATED GINGIVITIS
• ACUTE STREPTOCOCCAL GINGIVITIS
• HIV DISEASE ASSOCIATED GINGIVITIS
• ACUTE HERPETIC GINGIVOSTOMATITIS

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19
Q

GINGIVITIS

MISCELLANEOUS FORMS OF GINGIVITIS:

______
• Hormone-related gingivitis

A

PREGNANCY GINGIVITIS

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20
Q

GINGIVITIS

MISCELLANEOUS FORMS OF GINGIVITIS:

______
• Usually seen in the second trimester of pregnancy

A

PREGNANCY GINGIVITIS

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21
Q

GINGIVITIS

MISCELLANEOUS FORMS OF GINGIVITIS:

______
• Linked to increased numbers of anaerobes, especially Prevotella intermedia

A

PREGNANCY GINGIVITIS

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22
Q

GINGIVITIS

MISCELLANEOUS FORMS OF GINGIVITIS:

______
• Associated with changes in steroid hormone levels in crevicular fluid

A

PREGNANCY GINGIVITIS

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23
Q

GINGIVITIS

MISCELLANEOUS FORMS OF GINGIVITIS:

______
• Commonly seen in poorly controlled diabetics

A

DIABETES MELLITUS–RELATED GINGIVITIS

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24
Q

GINGIVITIS

MISCELLANEOUS FORMS OF GINGIVITIS:

______
• Diseased sites show increased levels of Capnocytophaga

A

DIABETES MELLITUS–RELATED GINGIVITIS

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25
GINGIVITIS MISCELLANEOUS FORMS OF GINGIVITIS: ______ • Also associated with periodontopathogens like ______ and ______
DIABETES MELLITUS–RELATED GINGIVITIS, Porphyromonas gingivalis, Treponema denticola
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GINGIVITIS MISCELLANEOUS FORMS OF GINGIVITIS: ______ • Caused by Streptococcus pyogenes
ACUTE STREPTOCOCCAL GINGIVITIS
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GINGIVITIS MISCELLANEOUS FORMS OF GINGIVITIS: ______ • Often follows a streptococcal sore throat and presents as a severe disease with fever
ACUTE STREPTOCOCCAL GINGIVITIS
28
GINGIVITIS MISCELLANEOUS FORMS OF GINGIVITIS: ______ • Gingivae appear inflamed, red (______), and swollen (______)
ACUTE STREPTOCOCCAL GINGIVITIS, erythema, edema
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GINGIVITIS MISCELLANEOUS FORMS OF GINGIVITIS: ______ • Often linked to immune suppression caused by the virus
HIV DISEASE ASSOCIATED GINGIVITIS
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GINGIVITIS MISCELLANEOUS FORMS OF GINGIVITIS: ______ • Caused by atypical and opportunistic bacteria
HIV DISEASE ASSOCIATED GINGIVITIS
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GINGIVITIS MISCELLANEOUS FORMS OF GINGIVITIS: ______ • Common bacteria include Atypical mycobacteria, Porphyromonas gingivalis, and Treponema denticola
HIV DISEASE ASSOCIATED GINGIVITIS
32
GINGIVITIS MISCELLANEOUS FORMS OF GINGIVITIS: ______ • Caused by Human Herpesviruses 1 and 2 (HHV-1, HHV-2)
ACUTE HERPETIC GINGIVOSTOMATITIS
33
GINGIVITIS MISCELLANEOUS FORMS OF GINGIVITIS: ______ • Often in early childhood (usually subclinical or mistaken for teething); more severe if first contracted in adulthood
ACUTE HERPETIC GINGIVOSTOMATITIS
34
GINGIVITIS MISCELLANEOUS FORMS OF GINGIVITIS: ______ • Painful, inflamed, bleeding gums, oral ulcers, fever; incubation ~______ days; spread through skin lesions or saliva
ACUTE HERPETIC GINGIVOSTOMATITIS, 5
35
______ • is an inflammatory or infectious disease of the ______ — the tissues that surround and support the teeth (gingiva, periodontal ligament, cementum, and alveolar bone).
PERIODONTITIS, periodontium
36
______ • It is characterized by sore, swollen, and bleeding gums and, in severe cases, can cause tooth loosening or tooth loss.
PERIODONTITIS
37
PERIODONTITIS DISTINGUISHING FEATURES Progressive destruction of the tooth-supporting structures (______, ______, ______, ______).
gingiva, periodontal ligament, cementum, alveolar bone
38
PERIODONTITIS DISTINGUISHING FEATURES Presence of ______ and ______ (visible on X-rays) — key diagnostic markers.
Clinical Attachment Loss (CAL), Alveolar Bone Loss
39
PERIODONTITIS DISTINGUISHING FEATURES Formation of ______ (deep spaces between tooth and gum due to tissue detachment).
periodontal pockets
40
PERIODONTITIS DISTINGUISHING FEATURES ______ bleeding — especially when ______.
Gingival, probing
41
PERIODONTITIS DISTINGUISHING FEATURES Symptoms include: • ______, ______, or ______ gums. • ______ gums (teeth appear longer). • ______ teeth or tooth ______ in advanced cases. • Persistent ______ and sometimes ______.
Swollen, tender, bleeding Receding Loose, loss bad breath, pus discharge
42
______ • Most prevalent form of periodontal disease worldwide.
CHRONIC PERIODONTITIS
43
______ • A chronic, multifactorial, inflammatory condition associated with the progressive destruction of the tooth-supporting apparatus.
CHRONIC PERIODONTITIS
44
______ • Characterized by slow progression, primarily affecting adults.
CHRONIC PERIODONTITIS
45
______ CLINICAL FEATURES • Gross gingival inflammation, fibrosis, and shrinkage.
CHRONIC PERIODONTITIS
46
______ CLINICAL FEATURES • Periodontal pockets >3 mm with bleeding.
CHRONIC PERIODONTITIS
47
______ CLINICAL FEATURES • Tooth mobility and migration.
CHRONIC PERIODONTITIS
48
______ CLINICAL FEATURES • Irregular alveolar bone loss & gingival recession.
CHRONIC PERIODONTITIS
49
______ CLINICAL FEATURES • Halitosis and offensive taste.
CHRONIC PERIODONTITIS
50
______ CLINICAL FEATURES • Usually painless, may or may not be linked to systemic diseases.
CHRONIC PERIODONTITIS
51
CHRONIC PERIODONTITIS MICROBIOLOGY • Dominated by ______.
Gram-negative anaerobes
52
CHRONIC PERIODONTITIS MICROBIOLOGY Key Pathogens:
• Porphyromonas gingivalis • Prevotella intermedia • Fusobacterium nucleatum • Tannerella forsythia • Aggregatibacter actinomycetemcomitans • Selenomonas spp. • Capnocytophaga spp. • Spirochaetes
53
______ • A rapidly progressing form of periodontal disease.
AGGRESSIVE PERIODONTITIS
54
______ • Affects primarily young individuals, often around puberty.
AGGRESSIVE PERIODONTITIS
55
______ • Historically classified as “juvenile periodontitis” or “early-onset periodontitis.”
AGGRESSIVE PERIODONTITIS
56
______ CLINICAL FEATURES • Onset around puberty (~13 years).
AGGRESSIVE PERIODONTITIS
57
______ CLINICAL FEATURES • Rapid bone loss with intermittent active and quiescent phases.
AGGRESSIVE PERIODONTITIS
58
______ CLINICAL FEATURES • Minimal plaque and calculus in periodontal pockets, despite severe destruction.
AGGRESSIVE PERIODONTITIS
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______ CLINICAL FEATURES • May or may not show signs of gingivitis.
AGGRESSIVE PERIODONTITIS
60
______ CLINICAL FEATURES • Often discovered through radiographs rather than symptoms.
AGGRESSIVE PERIODONTITIS
61
AGGRESSIVE PERIODONTITIS TYPES & MORBIDITY • ______: Primarily affects incisors and first permanent molars.
Localized
62
AGGRESSIVE PERIODONTITIS TYPES & MORBIDITY • ______: Affects multiple teeth beyond molars and incisors.
Generalized
63
AGGRESSIVE PERIODONTITIS TYPES & MORBIDITY • Relatively rare: ~______% in young white populations, more common in ______ and ______.
0.1, West Africans, Asians
64
AGGRESSIVE PERIODONTITIS TYPES & MORBIDITY • ______ is common; may involve genetic or immune defects.
Familial clustering
65
AGGRESSIVE PERIODONTITIS MICROBIOLOGY & IMMUNOLOGY Key Pathogens:
• Aggregatibacter actinomycetemcomitans • Capnocytophaga spp. • Porphyromonas gingivalis • Prevotella intermedia
66
AGGRESSIVE PERIODONTITIS MICROBIOLOGY & IMMUNOLOGY • Impaired immune response: Peripheral blood ______ show reduced ______ response.
lymphocytes, chemotactic
67
AGGRESSIVE PERIODONTITIS MICROBIOLOGY & IMMUNOLOGY Strong association with ______ due to: • High incidence in ______. • High ______ levels that drop after treatment.
A. actinomycetemcomitans, lesional sites, antibody
68
AGGRESSIVE PERIODONTITIS MANAGEMENT • Mechanical ______ therapy combined with strict ______.
periodontal, oral hygiene
69
AGGRESSIVE PERIODONTITIS MANAGEMENT • Adjunctive ______ therapy (e.g., ______ 250 mg 3x/day for 4 weeks) is often effective.
antibiotic, tetracycline
70
AGGRESSIVE PERIODONTITIS MANAGEMENT • Successful treatment leads to regression of ______ and reduction of ______.
disease, A. actinomycetemcomitans
71
______ • Defined as inflammation of the soft tissues covering or immediately subjacent to the crown of a partially erupted tooth.
PERICORONITIS
72
______ • The condition is frequently seen in the operculum and the soft tissues in erupting lower third molars of young adults.
PERICORONITIS
73
PERICORONITIS CLINICAL FEATURES ______: • sudden onset and short lived
Acute pericoronitis
74
PERICORONITIS CLINICAL FEATURES ______: • with significant symptoms such as trismus and extreme pain, especially when opposing tooth causes additional trauma
Acute pericoronitis
75
PERICORONITIS CLINICAL FEATURES ______: • repeated episodes of acute pericoronitis
Chronic or recurrent
76
PERICORONITIS CLINICAL FEATURES ______: • presents with varying degrees of inflammation of the pericoronal flap and adjacent structures, as well as systemic complications.
Chronic or recurrent
77
PERICORONITIS MICROBIOLOGY & IMMUNOLOGY Key Pathogens Strict anaerobes:
• Prevotella intermedia • Fusobacterium species • Anaerobic streptococci
78
PERICORONITIS MICROBIOLOGY & IMMUNOLOGY Key Pathogens Other periodontopathic organisms:
• Aggregatibacter actinomycetemcomitans • Tannerella forsythia
79
PERICORONITIS MANAGEMENT Mild cases: • local ______ irrigation, especially underneath the ______
saline, operculum
80
PERICORONITIS MANAGEMENT Mild cases: • ______ relief from the opposing tooth, if any.
trauma
81
PERICORONITIS MANAGEMENT Moderate to severe cases: • antibiotic therapy with ______ either with or without ______ and ______
penicillin, clindamycin, metronidazole
82
______ • IS A REVERSIBLE INFLAMMATORY CONDITION THAT AFFECTS THE SOFT TISSUES (MUCOSA) SURROUNDING A DENTAL IMPLANT WITHOUT ANY LOSS OF SUPPORTING BONE. IT IS CONSIDERED THE PRECURSOR TO PERI-IMPLANTITIS
PERI-IMPLANT MUCOSITIS
83
______ • Inflammation limited to soft tissues surrounding the implant
PERI-IMPLANT MUCOSITIS
84
______ • No radiographic evidence of bone loss
PERI-IMPLANT MUCOSITIS
85
______ • Bleeding on probing (BoP) is the most reliable clinical sign
PERI-IMPLANT MUCOSITIS
86
______ • Caused by bacterial plaque accumulation
PERI-IMPLANT MUCOSITIS
87
______ • Can be reversed with improved oral hygiene and professional care
PERI-IMPLANT MUCOSITIS
88
PERI-IMPLANT MUCOSITIS KEY PATHOGENS:
Streptococcus spp. Fusobacterium nucleatum Prevotella intermedia Actinomyces spp.
89
PERI-IMPLANT MUCOSITIS KEY PATHOGENS: • ______ - Early colonizers; initiate biofilm formation on implant surfaces
Streptococcus spp.
90
PERI-IMPLANT MUCOSITIS KEY PATHOGENS: • ______ - Gram-negative anaerobe; promotes inflammatory response
Fusobacterium nucleatum
91
PERI-IMPLANT MUCOSITIS KEY PATHOGENS: • ______ - Gram-negative anaerobic rod; associated with soft tissue inflammation
Prevotella intermedia
92
PERI-IMPLANT MUCOSITIS KEY PATHOGENS: • ______ - Gram-positive rods; often found in early-stage peri-implant mucositis
Actinomyces spp.
93
______ CLINICAL IMPLICATIONS • Acts as an early warning sign of peri-implant disease
PERI-IMPLANT MUCOSITIS
94
______ CLINICAL IMPLICATIONS • Progresses to peri-implantitis if not treated
PERI-IMPLANT MUCOSITIS
95
______ CLINICAL IMPLICATIONS • No bone loss — key to distinguish from peri-implantitis
PERI-IMPLANT MUCOSITIS
96
______ CLINICAL IMPLICATIONS • Indicates poor plaque control, possibly poor prosthesis design or patient hygiene habits
PERI-IMPLANT MUCOSITIS
97
______ CLINICAL IMPLICATIONS • Routine monitoring and maintenance are essential
PERI-IMPLANT MUCOSITIS
98
PERI-IMPLANT MUCOSITIS INTERVENTION:
• Mechanical Debridemen • Antimicrobial Therapy • Oral Hygiene Instruction • Maintenance and Monitoring
99
______ • is a biofilm-associated inflammatory condition affecting the peri-implant mucosa, with loss of supporting bone.
PERI IMPLANTITIS
100
______ • Additional clinical signs of inflammation, including redness, swelling, or the presence of a draining sinus, in addition to progressive bone loss.
PERI IMPLANTITIS
101
______ • Lesions extend beyond the pocket epithelium, have a larger size inflammatory cell infiltrate, and large proportions of plasma cells.
PERI IMPLANTITIS
102
PERI IMPLANTITIS KEY PATHOGENS Same with peri-implant mucositis but with high numbers of:
Staphylococcus aureus Peptostreptococci Enteric rods Yeasts
103
PERI IMPLANTITIS Signs and symptoms:
• Swelling or redness of gums around the implant • Bleeding on probing • Suppuration (pus) • Pain or tenderness in the gums around the implant. • A bad taste in the mouth. • Loosening of the implant.
104
______ CLINICAL IMPLICATIONS • Implant design and surface roughness influence biofilm accumulation.
PERI IMPLANTITIS
105
PERI IMPLANTITIS INTERVENTION:
• Patients should maintain meticulous oral hygiene • Professional dental management • Surgical treatment