descending motor pathways Flashcards

(37 cards)

1
Q

motor system evolution

A
  1. reticular formation
  2. vestibular system
  3. tectum
  4. red nucleus
  5. cerebral cortex
  6. (later) pyramidal tract
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2
Q

reticular formation

A

random network of neurons that has a reticulospinal pathway that modifies excitability of neurons in the spinal cord

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3
Q

vestibular system

A

semicircular canals give us position in space, in relation to gravity
(up vs down)

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4
Q

tectum

A

contain superior and inferior colliculus for visual and auditory inputs

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5
Q

red nucleus

A
  • primitive corticospinal tract
  • has excitatory and controlling affects on more distal muscles
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6
Q

cerebral cortex

A

controlled lower stations in the motor pathway

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7
Q

somatotopic organization of ventral horn

A

more medial the muscle, the more medial the neurons are in gray matter

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8
Q

descending pathways

A
  • lateral pathways
  • medial pathways
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9
Q

lateral pathways control

A

distal musculature
- include: lateral corticospinal tract
skilled muscle movement

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10
Q

medial pathways control

A

axial and proximal musculature
- include: vestibulospinal, reticulospinal, tectospinal tracts

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11
Q

cells of motor cortex

A

giant pyramidal cells

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12
Q

physiology of motor cortex

A
  • pyramidal cell fires impulses prior to muscle movement
  • organized to produce simple movements of individual body parts
  • determines direction, amplitude, speed and force of movement
  • not organized to control individual muscles
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13
Q

somatotopy of motor cortex

A

motor area given to parts of the body that perform more precise and delicate movements compared to those that perform less precise movements
- movements not muscles!!

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14
Q

efferent pathway

A
  • axons thru white matter
  • to internal capsule
  • to crus cerebri/cerebellar peduncle
  • pyramidal fibers pierce the pons
  • pyramids of medulla
  • lateral corticospinal tract
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15
Q

other parts of the cerebral cortex that is involved with motor function

A
  • premotor cortex
  • supplementary motor cortex
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16
Q

premotor cortex

A
  • affects motor cortex
  • organizes neurons into more complex units of movement
  • triggers movement of response to visual and other stimuli
  • directs reaching and grasping movements
    “preparation of movement”
17
Q

supplementary motor cortex

A
  • higher level of motion control
  • concerned with mvmt that is self generated
  • bilateral movements
18
Q

corticobulbar tract

A
  • cell bodies in motor cortex (lateral convexity)
  • fibers leave through internal capsule
  • exit in midbrain, pons, and medulla
  • innervate cranial nerve motor nuclei
  • crossed and uncrossed fibers
    (motor cortex to cranial nerve nuclei)
19
Q

corticobulbar tract interacts with these nuclei:

A
  • trigeminal motor nucleus
  • nucleus ambiguus
  • facial motor nucleus (upper face)
  • facial motor nucleus (lower face)
  • hypoglossal nucleus
20
Q

which nuclei have completely crossed tracts? (control contralateral sides)

A
  • facial motor nucleus (lower face)
  • hypoglossal nucleus
21
Q

abducens and facial nerves

22
Q

bell’s palsy symptoms

A
  • droopy eyelid, dry eye, or excessive tears
  • facial paralysis, twitching, weakness
  • drooping corner of mouth, dry mouth, impaired taste
23
Q

bell’s palsy can be caused by

A

lesion of CN VII
- lesion on left side = paralysis of left side of face

24
Q

lower face nucleus receive input from

A

opposite side motor cortex face area only

25
the upper face gets inputs form
same side AND opposite side motor cortex
26
what if there is a lesion of motor cortex on opposite side?
- the axons will degenerate - lost control of lower part of the face - still have control of the upper half of the face bc it also receives same side signals
27
why is it bad if only the lower half of the face lost motor function?
- indicative of a stroke in dangerous area - disrupted the cranial nerve (7?) - motor cortex damages
28
jacksonian march
the spreading of seizure from primary location to other parts of the body (I.e. from thumb to face)
29
epilepsy is the result of
excessive activity of neurons - opposite of a lesion
30
upper motor neuron lesion
- slight muscle atrophy - no fasciculation - hyperreflexia - increased/spastic muscle tone
31
lower motor neuron lesion
- severe muscle atrophy - fasciculation - hyporeflexia - decreased/flaccid muscle tone
32
spastic hemiplegia cause
stroke of left hemisphere
33
symptoms of spastic hemiplegia
- arm is flexed upward - knee is straight out - fingers are curled due to hyperactivity of stretch reflexes - excess tone due to UMN lesion (no descending inhibition to blow out unnecessary reflexes
34
Babinksi Response Test
- in babies less than 1 year - run finger along bottom of foot - toes will flare and withdraw foot
35
when is the Babinski reflex suppressed?
- after 1 year of age - helps ability to walk - not present in adults (curl toes instead)
36
grasp reflex
- when anything touches a baby's palm, fingers close - reflex is outgrown unless damage to motor cortex
37