descending motor pathways Flashcards

1
Q

motor system evolution

A
  1. reticular formation
  2. vestibular system
  3. tectum
  4. red nucleus
  5. cerebral cortex
  6. (later) pyramidal tract
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2
Q

reticular formation

A

random network of neurons that has a reticulospinal pathway that modifies excitability of neurons in the spinal cord

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3
Q

vestibular system

A

semicircular canals give us position in space, in relation to gravity
(up vs down)

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4
Q

tectum

A

contain superior and inferior colliculus for visual and auditory inputs

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5
Q

red nucleus

A
  • primitive corticospinal tract
  • has excitatory and controlling affects on more distal muscles
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6
Q

cerebral cortex

A

controlled lower stations in the motor pathway

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7
Q

somatotopic organization of ventral horn

A

more medial the muscle, the more medial the neurons are in gray matter

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8
Q

descending pathways

A
  • lateral pathways
  • medial pathways
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9
Q

lateral pathways control

A

distal musculature
- include: lateral corticospinal tract
skilled muscle movement

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10
Q

medial pathways control

A

axial and proximal musculature
- include: vestibulospinal, reticulospinal, tectospinal tracts

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11
Q

cells of motor cortex

A

giant pyramidal cells

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12
Q

physiology of motor cortex

A
  • pyramidal cell fires impulses prior to muscle movement
  • organized to produce simple movements of individual body parts
  • determines direction, amplitude, speed and force of movement
  • not organized to control individual muscles
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13
Q

somatotopy of motor cortex

A

motor area given to parts of the body that perform more precise and delicate movements compared to those that perform less precise movements
- movements not muscles!!

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14
Q

efferent pathway

A
  • axons thru white matter
  • to internal capsule
  • to crus cerebri/cerebellar peduncle
  • pyramidal fibers pierce the pons
  • pyramids of medulla
  • lateral corticospinal tract
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15
Q

other parts of the cerebral cortex that is involved with motor function

A
  • premotor cortex
  • supplementary motor cortex
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16
Q

premotor cortex

A
  • affects motor cortex
  • organizes neurons into more complex units of movement
  • triggers movement of response to visual and other stimuli
  • directs reaching and grasping movements
    “preparation of movement”
17
Q

supplementary motor cortex

A
  • higher level of motion control
  • concerned with mvmt that is self generated
  • bilateral movements
18
Q

corticobulbar tract

A
  • cell bodies in motor cortex (lateral convexity)
  • fibers leave through internal capsule
  • exit in midbrain, pons, and medulla
  • innervate cranial nerve motor nuclei
  • crossed and uncrossed fibers
    (motor cortex to cranial nerve nuclei)
19
Q

corticobulbar tract interacts with these nuclei:

A
  • trigeminal motor nucleus
  • nucleus ambiguus
  • facial motor nucleus (upper face)
  • facial motor nucleus (lower face)
  • hypoglossal nucleus
20
Q

which nuclei have completely crossed tracts? (control contralateral sides)

A
  • facial motor nucleus (lower face)
  • hypoglossal nucleus
21
Q

abducens and facial nerves

A
22
Q

bell’s palsy symptoms

A
  • droopy eyelid, dry eye, or excessive tears
  • facial paralysis, twitching, weakness
  • drooping corner of mouth, dry mouth, impaired taste
23
Q

bell’s palsy can be caused by

A

lesion of CN VII
- lesion on left side = paralysis of left side of face

24
Q

lower face nucleus receive input from

A

opposite side motor cortex face area only

25
Q

the upper face gets inputs form

A

same side AND opposite side motor cortex

26
Q

what if there is a lesion of motor cortex on opposite side?

A
  • the axons will degenerate
  • lost control of lower part of the face
  • still have control of the upper half of the face bc it also receives same side signals
27
Q

why is it bad if only the lower half of the face lost motor function?

A
  • indicative of a stroke in dangerous area
  • disrupted the cranial nerve (7?)
  • motor cortex damages
28
Q

jacksonian march

A

the spreading of seizure from primary location to other parts of the body (I.e. from thumb to face)

29
Q

epilepsy is the result of

A

excessive activity of neurons
- opposite of a lesion

30
Q

upper motor neuron lesion

A
  • slight muscle atrophy
  • no fasciculation
  • hyperreflexia
  • increased/spastic muscle tone
31
Q

lower motor neuron lesion

A
  • severe muscle atrophy
  • fasciculation
  • hyporeflexia
  • decreased/flaccid muscle tone
32
Q

spastic hemiplegia cause

A

stroke of left hemisphere

33
Q

symptoms of spastic hemiplegia

A
  • arm is flexed upward
  • knee is straight out
  • fingers are curled due to hyperactivity of stretch reflexes
  • excess tone due to UMN lesion (no descending inhibition to blow out unnecessary reflexes
34
Q

Babinksi Response Test

A
  • in babies less than 1 year
  • run finger along bottom of foot
  • toes will flare and withdraw foot
35
Q

when is the Babinski reflex suppressed?

A
  • after 1 year of age
  • helps ability to walk
  • not present in adults (curl toes instead)
36
Q

grasp reflex

A
  • when anything touches a baby’s palm, fingers close
  • reflex is outgrown unless damage to motor cortex
37
Q
A