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4/18 Flashcards

1
Q

digoxin MOA

A

decrease extracellular Na

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2
Q

Fick’s principle

A

CO = rate of O2 used/(arterial o2-venous o2)

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3
Q

most common cause of secondary HTN

A

Renal DZ

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4
Q

what med is contraindicated in bilateral renal artery stenosis

A

ACEi-ischemia

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5
Q

what do alpha blockers end in

A

azosin

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6
Q

DM best HTN med

A

ACEi-delays kindness damage

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7
Q

DM contraindicated HTN meds

A

Thiazides, Beta blockers-impaired glucose tolerance

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8
Q

CHF best HTN meds

A

ACEi/ARB
Aldosterone agonist
B blocker

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9
Q

CHF contraindicated HTN meds

A

Ca2+ blockers-reduced rate and contractility

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10
Q

Post MI best HTN meds

A

ACEi/ARB
Aldosterone agonist
B blocker

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11
Q

BPH HTN med

A

alpha 1 blocker

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12
Q

migranes HTN med

A

verapamil, beta blocker

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13
Q

osteoporosis HTN med

A

thiazides-maintains Ca

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14
Q

pregnancy HTN meds allowed

A

Hydralazine
methyldop
LABATELOL
Nifedipine

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15
Q

who gets a statin

A

High ASCVD 40-75 7.5%
diabetes 40-75
ldl>190

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16
Q

MONABASH

A 
Morphine
O2
Nitro
Aspirin
BB
ACEi
Statin
Heparin
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17
Q

V2-4 changes

A

Anterior LAD

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18
Q

V1-3 changes

A

septal (LAD)

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19
Q

2,3, avf

A

inferior (Posterior descending)

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20
Q

1, avl, V4-V6

A

Lateral (LAD or circumflex)

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21
Q

when do ventricular wall ruptures happen post MI

A

4-8 days

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22
Q

when are arrhythmia post MI

A

2-4 days

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23
Q

when is papillary necrosis post mi

A

5-10days

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24
Q

pericarditis post MI days

A

2-4

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25
Dresslers post MI timeframe
2-8 weeks
26
Afib tx options
``` Anticoagulation BB Cardioversion Ca2+ blockers Digoxin ```
27
Class 1a MOA, Examples, Use
NA+ channel blocker prolonged action potential, Quinidine, Procanamide PSVT, Afib, Aflutter, Vtach
28
Class 1B MOA, Examples, Use
NA+ channel blocker shortens action potential, Lidocaine, tocainide Vtach
29
Class 1C MOA, Examples, Use
NA+ channel blocker no effect on action potential, flecainide propafenone PSVT,Afib, Aflutter,
30
Class 2 MOA, Examples, Use
BB, -lol, PVC,PSVT,Afib, Aflutter,Vtach
31
Class 3 MOA, Examples, Use
K+ blockers, Amioderone, stall, bretylium, | Afib, Aflutter, Vtach (not bretylium)
32
Class 4 MOA, Examples, Use
Ca2+ blockers, verapamil, diltiazam, | PSVT, MAT,Afib, Aflutter
33
Adenosine MOA, Use
K+ activation ➡️ decreased cAMP | PSVT
34
COPD ❤️ thing
cor pulmonale
35
most common HF sign
S3
36
JONES criteria
``` Joints ❤️ Nodules Erythema Sydenham Chorea ```
37
besides st elevation what else is seen on pericarditis egg
PR depression
38
beck's triad for for tamponade
hypotension, distended neck, distant heart sounds
39
Major Duke's criteria
Serial blood cultures + vegetation or access on echo new onset regurg blood cultures + coxiella burnetii
40
Acute endocarditis organism
staph aureus step pneumonia strep pyogenes neisseria
41
Subacute endocarditis organisms
viridian's strep Enterococus fungi staph epididymus
42
endocarditis abs length
4-6 weeks
43
PVD only meds
pentoxyfylline and cilostazol
44
what dz associated with poly arthritis nodoasa
hep b and c
45
what system is spared in poly arthritis nodes
pulm
46
poly arthritis nodes marker
p-ANCA
47
Positive Sudan stain normal d xylose test
Assess for pancreatic insufficiency
48
Abnormal d xylose
Get small bowl biopsy Sprue Whipple Bacterial overgrowth
49
Malabsorption low stool ph
Lactase deficiency
50
Malnutrition normal stool ph
Pernicious arena Ileal dz Bacterial overgrowth
51
Tropical sprue tx
Tetracyclines
52
Whipple sx
Arthralgia Dementia Other neuro abdominal pain
53
Whipple tx
Bactrim or cefteiaxone for 1 year
54
What marker sometime + in UC
PANCA
55
Peutz jeghers syndrome
Polyps and hamaratoma. Mucocutaneous pigmentation of mouth hand and genitalia
56
Grey turner sign | Cullen sign in pancreatitis
Ecchymosis of flank | Periumbilical ecchymosis
57
Exocrine pancreases tumor markets
CEA CA19-9
58
Liver lab PBC
⬆️ Alk phos ⬆️ cholesterols ⬆️bili Normal AST ALT +ANA
59
PSC associated dz
UC
60
Hemochromatosis tx
Phlebotomy | Deferoxamine
61
Ceruloplasmin levels wilsons dz
⬇️
62
Wilson’s tx
Trientine penicillamine Zinc B6
63
Budd–Chiari syndrome
occlusion of the hepatic veins that drain the liver. It presents with the classical triad of abdominal pain, ascites, and liver enlargement.

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