A&P of cardiovascular system Flashcards

(41 cards)

1
Q

Endocardium
Myocardium
Epicardium

A

Endo-surrounds the heart(inner)
Myo-muscle (middle layer)
Epi-outer cardial layer

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2
Q

Endocarditis

A

Infection of the heart caused by a bacteria, usually strep (effects valves of the heart) (vegitation on the valve)

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3
Q

Pericarditis

A

Inflammation of the pericardium

Pinpoint, sharp, chest pain

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4
Q

Myocarditis

A

Inflammation of the heart caused by viral infection. If left untreated, heart failure.

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5
Q

Bicuspid

A
Mitral valve (Left)
AV valve
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6
Q

Semilunar valve

A

Pulmonary and Aortic Valve

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7
Q

Tricuspid

A

Right AV valve

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8
Q

Stenosis

A

Hardening and narrowing

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9
Q

Coronary perfuses during

A

Diastole

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10
Q

Papilary muscles

A

muscles inside the chambers of the heart

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11
Q

Chordae Tendineae

A

Tendens of the coronary valves

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12
Q

Anastomoses

A

Collateral Circulation (branch off above the blockage)

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13
Q

ANP

A

(atrial natriuretic peptide)
Produced, stored and released in atria
Responds to atrial distention and sympathetic stimulation

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14
Q

BNP

A

(brain natriuretic peptide)
Produced, stored and released in ventricular muscle and also the brain
Responds to ventricular dilation and sympathetic stimulation

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15
Q

angiotensin II

A

Potent Vasoconstrictor

converted from angiotensin I

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16
Q

Sympathetic nerves

A

intervate the entire heart

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17
Q

Parasympathetic nerves

A

Only intervate the atria

18
Q

Parasympathetic control of HR

A

Atria

Slows HR

19
Q

Sympathetic control of HR

A

Atria & Ventricles
Adrenals
Increases Heart Rate

20
Q

Cardiac Cycle

A

Diastole – Resting Phase of L Ventricle
Systole
Ejection Fraction - % of blood ejected from L Ventricle (55-70%)
Stroke Volume – Amt of blood discharged from heart
Preload – Amt of blood enters heart
Cardiac Contractility – How hard heart contracts
Afterload – Amount of blood that leaves the L Ventricle
Cardiac Output – Heart Rate x Stroke Volume, based per minute.

21
Q

How can we improve stroke volume?

A

Increase HR, Increase Contractility and change amout of fluid entering and exiting heart

22
Q

How does calcium effect the heart?

A

Contractility

23
Q

Starling law

A

The Greater the stretch, the greater the fill, the greater the contraction (within reason) – You need to make sure the patient’s heart can take the greater fill, (can be done via meds) because if it cannot, you can injure the heart.

24
Q

Cardiac muscles (atrial)

A

Atrial- SA Node 60-100

25
Cardiac Muscle (ventricular)
AV Junction 40-60
26
Cardiac Muscle Excitatory and conductive fibers
``` Intercalated discs (400x faster) Syncytium ```
27
Cardiac Muscle Atrioventricular Bundle
Bundle of HIS
28
Syncytium
Atrial or Ventricular | ALL OR NONE
29
Excitability
Waiting to respond
30
Conductivity
propagate impulse, pass it on
31
Automaticity
self-depolarization All cardiac cells have
32
Contractility
contract or shorten the level of contraction
33
Rhythmicity
Ability to beat in a regular, rhythmic fashion
34
2 types of cardiac cells
Mechanical- Contraction and relaxation: have the ability to pace but usually will not Pacemaker- Depolarization and repolarization
35
Cardiac Cycle
End of one heartbeat to the end of the next heart beat
36
S1
AV Valves Closing during ventricular systole, “lubb”
37
S2
semilunar valves closing at the beginning of ventricular diastole, “dubb”
38
S3
heard after S2, may indicate heart failure
39
S4
heard late in diastole just before S1, may be heard in patients with CHF.
40
Tunica Intima Tunica Media Tunica Adventitia
``` Tunica Intima innermost epithelial layer Tunica Media middle smooth muscle layer Tunica Adventitia outermost connective tissue layer ```
41
Baroreceptors
Blood pressure | Aortic arch and Carotid bodies