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Medical Block > Ventricular Rythms > Flashcards

Ventricular Rythms Flashcards

1
Q

QRS

A

> .12 if initiated in the ventricles

a beat >.12 isn’t necessarily initiated in the ventricles

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2
Q

PVC

A

Wide and bizarre >.12 QRS

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3
Q

Interpolated PVC

A

doesn’t affect rythm. falls in the middle of 2 beats

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4
Q

Compensatory pause

A

PVC interrupts normal rhythm

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5
Q

sinus brady with bigeminal PVC’s

A

electrical twice the mechanical

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6
Q

Run of PVC’s (or burst)

A

3 or more PVC’s in a row

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7
Q

Couplet of PVC’s in a row

A

2 PVC’s in a row

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8
Q

electrical doesn’t match mechanical pulse

A

Pulsus deficit

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9
Q

Frequent PVC’s are = to

A

6 or more per minute

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10
Q

Ventricular escape beats

A

A. Ectopic occurs after pause in which supraventricular pacemaker fails to initiate impulse
B. Protective mechanism

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11
Q

VTAK

A

Heart extremely irritable
Ventricular focus could speed up and over ride pacer sites
Polymorphic vs. monomorphic

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12
Q

Sustained VTAK

A

> 30 seconds

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13
Q

Nonsustained VTAK

A

<30 seconds

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14
Q

6 characteristics of VTAK

A
  1. Rate is 150-250
  2. Rhythm is regular
  3. P wave none usually, if there doesn’t conduct
  4. PRI—none
  5. QRS
    a. wide bizarre
    b. > .12 sec
  6. T wave—–not seen
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15
Q

> 40 IVR

A

accelerated idioventricular rhythm

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16
Q

5 H’s

A 
Hypovolemia
Hypoxia
Hydrogen-ion(acidosis)
Hypo/hyperkalemia
Hypothermia
17
Q

5 T’S

A 
Tablets (OD)
Tamponade
Tension Pheumo
Thrombosis (coronary)
Thrombosis (emboli)
18
Q

LIDO

A

protocol

19
Q

NACO3

A

protocol

20
Q

Amiodarone

A

protocol

21
Q

S127

A

protocol

22
Q

etiology of PVC

A
  1. Damage to His-Purkinje system
  2. Increased sympathetic tone
  3. Hypoxia or Acidosis
  4. AMI or myocardial ischemia or CHF
  5. Electrolyte imbalances (K, Ca++, Mg)
  6. Digitalis toxicity
  7. Use of cyclic antidepressants, phenothiazines
  8. Stimulate uses
23
Q

Etiology of VTAK

A 
Same causes as PVC's
Ischemia
Hypoxia
Electrolytes
Acid-base problems
Increased sympathetic tone
24
Q

Polymorphic VTAK

A

Torsades de Pointes

25
Q

Torsades Itiology

A

Drug induced
Eating disorder
Electrolyte imbalances (magnesium, potassium, calcium)

26
Q

V flutter

A

often degenerates to vfib

27
Q

V Flutter symptoms

A

Palpitations Dyspnea
Dizziness Anxiety
Diaphoresis Angina
Seizures

28
Q

V Fib Etiology

A
  1. Usually preceded by ventricular irritability
  2. Ventricular damage
  3. Hypoxia
  4. Electrolyte disturbances
  5. Electric shock
  6. Malfunctioning pacemaker
  7. Drugs
29
Q

Often first rhythm following defibrillation

A

Idioventricular

Medical Block (20 decks)

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