Conceptual Models Flashcards

1
Q

General Adaptation Syndrome

A

Function – emotion, movement, & cognition

Location – autonomic nervous system (ANS) + hypothalamus

Chemicals - +ACTH, +epinephrine, +norepinephrine, +cortisol

Stages – Selye’s model of stress:
* (Initial) Alarm Reaction Stage- increased sympathetic nervous system & activated hypothalamus to release hormones from the pituitary and adrenal glands.
* (Persists) Resistance Stage- increased parasympathetic nervous system to return to normal functioning, but cortisol lingers leaving the body at high energy levels.
* (Prolonged) Exhaustion Stage- pituitary and adrenal glands can’t maintain hormone levels and physiological processes begin to breakdown.

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2
Q

McEwen’s Allostatic Load Model

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Function – physiological & psychological stress are processed differently; allostasis varies based on genetics, resilience, and locus of control of the stressor.
* processes allow the body to achieve stability by adapting to change (allostasis).
* brief allostatic state is fine, but when extended due to chronic/repeated stress it wears on the body & brain (allostatic load).
* if stressor lasts for days, weeks, or longer, it becomes an allostatic overload which effects one’s physical & psychological health.

Location – amygdala, hippocampus, & prefrontal cortex

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3
Q

Stages of Sleep

A

Function – sleep; Eysenck’s theories: Recovery/Restoration views the purpose of sleeps to be repaired damage that occurs when awake; Adaptive/Evolutionary views the purpose of sleep to conserve energy.

Stages – each having different EEG patterns
* Stage 1: transitional stage between wakefulness & sleep; low frequency, high amplitude alpha waves (drowsy state) are replaced by low frequency, low amplitude theta waves
* Stage 2: theta waves continue but are interrupted by sleep spindles (sudden bursts of moderately fast waves) & K-complexes (large, slow waves)
* Stage 3: after being asleep for 20min, there are low frequency, high amplitude delta waves(slow-wave, deep sleep)
* Stage 4: delta waves continue at a higher amplitude (slow-wave, deep sleep)
* Stage 5: rapid eye movement (REM) after being asleep for 80-90min, similar EEG pattern to stage 1; paradoxical sleep since the brain is active + physiologically aroused while major muscle groups are nearly paralyzed

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4
Q

Lifespan Changes in Sleep

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Function – sleep changes with age increase

Stages –
* Newborns sleep longest, spend most time in REM; start in active sleep, then go to quiet (non-REM) sleep
* 3mos of age, this pattern reverses with the four stages of non-REM evident by 6mos
* Total sleep time decreases from 14-16hrs to 8hrs in adulthood
* Older adults struggle to fall/sleep asleep + spend less time in stage 4 & develop circadian phase advance (sleeping earlier in the night, waking earlier in the morning)

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5
Q

Bottom-Up Processing

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Function – data driven; processing begins with sensory input and continues upward to the brain where it is perceived, interpreted, & stored.

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6
Q

Top-Down Processing

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Function – concept driven; processing begins in the brain with preexisting knowledge and expectations to interpret incoming sensory input.

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7
Q

Vision

A

Function – light waves absorbed in the retina by photoreceptors (cones & rods).
* Cones – work best in bright light, responsible for visual acuity, color perception
* Rods – no color, crucial for peripheral vision, most sensitive to light & responsible for vision in dim light

Theories of Color Vision –
* Trichromatic theory: retina contains 3 color receptors (red, blue, green); focused on initial retina processing.
* Opponent-process theory: opponent-process cells contain 3 types (red/green, blue/yellow, white/black); focused on beyond-retina processing & explains afterimages + colorblindness.

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8
Q

Color Blindness

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Function – seeing the incorrect color
* red/green most common; due to genetic mutation (recessive gene on X- chromosome; most common in males because they inherit only one X-chromosome, so in female, both parents must have the mutated gene) or injury/disease (diabetes, MS).
* blue/yellow caused by autosomal (non-sex) dominant gene, affecting males/females equally.

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9
Q

Depth Perception

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Function – perceive depth on a combination of binocular and monocular cues.
* Binocular cues (both eyes), perception of relative closeness; includes retinal disparity (closer the object, greater the disparity) & convergence (tendency for eyes to turn inward as an object gets closer & vice-versa)
* Monocular cues (one eye), used for greater distances; includes relative size, interposition (overlapping), linear perspective, texture, & relative motion (motion parallax)

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10
Q

Pain

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Function – extreme negative sensory input; extreme temperatures, mechanical pressure, electrical stimulation; moderated by emotional state & past experiences with pain.

Theory –
* Gate control: major theory of pain perception; distinguishes between two spinal cord fibers, the small unmyelinated fibers (responsible for transmitting pain signal to the brain) open the gate to pain signals & white larger myelinated fibers (transmit other sensory signals) closes the gate using other sensory signals (ex: relieving pain through use of heat or cold, distractions, hypnosis).

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11
Q

Synesthesia

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Function – sensations in one sensory modality spontaneously trigger as associated sensational in another modality; most seen as number or letters are associated with specific colors. Genetic component & is due to increased cross-activation & cross-connectivity between sensory areas.

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12
Q

Psychophysics

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Function – study of the relationship between magnitude of physical stimuli & psychological sensations; Weber’s Law, Fechner’s Law, Steven’s power law, & signal detection theory.

Theories –
* Weber’s Law: just noticeable differences (JND) for a stimulus is at a constant proportion (ex: to notice a difference in weight, it is either 2% lighter or heavier).
* Fechner’s Law: (Fechner-Weber) the JND grows to an increasingly greater degree with each increment of intensity; more applicable than Weber’s.
* Steven’s Power Law: relationship between physical stimuli & psychological sensations differs for different stimuli; more accurate than Weber’s & Fechner’s.
* Signal Detection Theory (SDT): assumes that the signal of stimuli is the result of both sensory & decision-making processes & is always accompanied by uncertain (presence of background neural activity like motivation, fatigue, environmental distractions). decision-making is affected by sensitivity (ability to distinguish between stimulus & background noise) and decision criterion (decision bias & response bias; willingness to say the stimulus is present in ambiguous situations). The greater the sensitivity, the greater the accuracy, but the willingness to say a stimulus exists influences the decision being made.

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13
Q

Theories of Emotion

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Function – theories of emotions include James-Lange, Cannon-Bard, Schachter & Singer Two-Factor, LeDoux’s Two-System Theory, & Lazarus Cognitive Appraisal.

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14
Q

James-Lange Theory of Emotion

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Theory - emotions follow physiological arousal; facial feedback hypothesis supports this (ex: smiling makes us happy).

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15
Q

Cannon-Bard Theory of Emotion

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Theory - emotions & physiological arousal occur simultaneously; the thalamus sends signals to the cerebral cortex & sympathetic nervous system simultaneously; all emotions involve similar physiological arousals thus emotional reactions cannot be attributed to differences in physiological arousal.

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16
Q

Schachter & Singer’s Two-Factor Theory of Emotion

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Thoery - (cognitive arousal theory) emotion follows physiological arousal & attribution (cognitive label); we look to our environment to interpret our sensations (epinephrine studies generated research of misattribution of arousal, mislabeling arousal when causes are ambiguous).

17
Q

Zillman’s 1971 Excitation Transfer Theory

A

Theory - generated from the epinephrine studies, it explains how physical arousal caused by one event can be transferred to & intensified by a later unrelated event. It assumes three things:
1) Physical arousal associated with emotions decays slowly & lingers following the event.
2) Residual arousal can be intensified by a subsequent, unrelated event.
3) People have limited insight into physical arousal causes & can misattribute their intense arousal solely to the subsequent event.

18
Q

Lazarus’s Cognitive Appraisal Theory

A

Theory - differences in emotional reactions to events are due to different appraisals of those events; two people can experience the same event but respond differently; assumes physiological arousal follows cognitive appraisal.

  • Primary Appraisal – evaluating the event to determine if its stressful/threatening
  • Secondary appraisal – when event is determined stressful/threatening, coping options are identified
  • Reappraisal – after monitoring the event, changes the primary/secondary appraisal decision
19
Q

LeDoux’s Two-System Theory of Emotion

A

Theory - focuses on fear & it consists of two interacting systems that respond to threatening stimuli (subcortical & cortical)
* Subcortical system – (survival system/low road) reacts quickly/automatically with physiological & defensive behavioral responses (amygdala is a major component)
* Cortical system – (conscious emotional system/high road) processes info from the senses, subcortical system, & episodic & semantic memory to generate a conscious feeling of fear (visual, prefrontal, & parietal cortex)

20
Q

Papez Circuit

A

Function – 1937 originally thought emotions are linked to certain brain areas, but recent research found this circuit is more involved in memory.

21
Q

Lewinsohn’s Social Reinforcement Theory

A

(1974) operant-conditioning
* Depression is the results of low response-contingent reinforcement for social behaviors due to lack of reinforcement. This leads to social isolation, low self-esteem, pessimism that further decrease likelihood of positive reinforcement.

22
Q

Seligman’s Learned Helplessness Model

A

(1974) This model links depression to repeated exposure to uncontrollable negative life events resulting in a sense of helplessness and attributing negative life events to stable, internal, & global factors.

(1989) Hopelessness Theory, this updating version states that exposure of negative events and negative cognitive style causes the sense of hopelessness (a proximal & sufficient cause of depression).

23
Q

Beck’s Cognitive Theory

A

(1974) attributes depression to a negative cognitive triad of negative thoughts about oneself, the world, & the future.

24
Q

Mowrer’s Two-Factor Theory

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Mowrer’s Two-Factor Theory: related to a combo of classical & operant conditioning, a person first experiences classical conditioning at which they find something scary & then experience operant conditioning by avoiding that scary thing. The person’s avoidant behavior in negatively reinforced.