Famous People/Laws/Definitions Flashcards

(62 cards)

1
Q

Subluxations are from abnormal biomechanics caused by muscle imbalance in a weight bearing spine. Pelvic distortion model (Basic distortion of the spine starts in the pelvis)

A

Carver

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2
Q

Developed a protocol for the tx of lumbar disc protrusion, spondylolisthesis, facet syndrome, subluxation, & scoliotic curves

A

Cox/McManus (Flex/dist)

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3
Q

Developed SOT. CSF flow through the pumping action of the sacrum & the cranial dura mater

A

DeJarnette

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4
Q

Fixation theory of joint hypermobility

A

Gillet/Fave

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5
Q

Subluxations are all pos. w/ disc wedging

A

Gonstead

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6
Q

Upper cervical specific; dentate lig.

A

Grostic

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7
Q

Found that disc herniations, exostoses, or subluxations may produce pressure on the dorsal nerve root

A

Hadley

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8
Q

Father of homeopathy (tx’s pts w/ heavily diluted preparations which are thought to cause effects similar to the symptoms presented)

A

Hahnemann

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9
Q

Father of modern medicine; first to manipulate

A

Hippocrates

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10
Q

Joint hypermobility; described SI movement & pelvic dynamics. Discovered & tested SI ligs.

A

Illi

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11
Q

3 phase model of instability (dysfunction, unstable, stabilization); Breaking up spinal adhesions on an injured segment

A

Kirkaldy/Willis

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12
Q

Segmental Facilitation Theory. Established the concept of subluxation creating a hyperactive nervous system, rather than a decrease in nerve impulses. Muscle is central to his theory

A

Korr

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13
Q

Wrote the 1st chiro. textbook in 1906; people started to research & incorporate motion instead of “bone out of place” idea

A

Langworthy, Smith, & Paxson

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14
Q

Revised Faye’s concept of the VSC

A

Skip Lantz

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15
Q

Founder of technique where sacrum is keystone of spine, first to use heel lifts

A

Logan (Logan Basic Technique)

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16
Q

World renown scientists in the field of biomechanics of the spine

A

Panjabi & White

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17
Q

General Adaptation Syndrome; under optimum conditions the body can respond to stressors

A

Selye

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18
Q

Wrote chiro textbook. Identified 33 principles of chiro. & “safety pin” cycle

A

Stephenson

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19
Q

Founder of osteopathy in 1856; utilized the circulatory system

A

Andrew Taylor Still

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20
Q

Came up with name “chiropractic”

A

Samuel Weed

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21
Q

Steady deformation d/t sustained axial pressure

A

Creep

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22
Q

Energy loss from loading & unloading disc (heat)

A

Hysteresis

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23
Q

Long standing creep w/ hysteresis event

A

Buckling syndrome

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24
Q

Deformation increases in proportion to the load applied

A

Hooke’s Law

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25
Opposing translation of two body parts
Sheer
26
Any recoverable deformation
Elastic deformation
27
Property of a material or structure that returns it to its original form following the removal of the deforming load
Elasticity
28
Nonrecoverable deformation
Plastic deformation
29
Property of a material to permanently deform when it is loaded beyond its elastic range
Plasticity
30
Property of returning to the former shape or size after distortion
Resilience
31
Property of a material showing sensitivity to the rate of loading or deformation, two basic components are viscosity & elasticity. Slowly deforms, slowly reforms
Viscoelasticity
32
Property of materials to resist loads that produce shear. Strain delay
Viscosity
33
Balance of compression & tensile forces on connective tissue
Tensegrity
34
Increase epiphyseal plate pressure = decreased growth | Decreased epiphyseal plate pressure = increased growth
Heuter Volkman
35
Bone (structure) responds to stress (function)
Wolff
36
Soft tissue responds to stress
Davis' law
37
A nerve which supplies a joint, supplies the muscle, skin, & tissue around it (axoplasmic aberration)
Hilton's law
38
Pain threshold, pain managment
Algometry
39
Nitrogen release during adjustment (joint capsule seal)
Cavitation
40
Skin rolling over spine for dx & tx
Roulement
41
Prox. neurological irritation causing peripheral neuropathies
Double Crush
42
CNS remapping, reorganizing, & brain expansion. PNS synapse (?)
Neuroplasticity
43
Opiates from peri-aqueductal grey descend to bathe dorsal horn & reduce incoming pain messages
Descending inhibition
44
Facilitation d/t acute nerve compression bombardment of dorsal horn, "endogenous opiates" (PAG) release shakes off pain for body to respond to immediate threat
Descending modulation
45
Gate control theory (Wall's), fast conducting proprioceptor & mechanoreceptor messages close the gate of slow pain receptors to CNS
Inhibiting system
46
Distraction principle such as: rubbing elbow, TENS unit. Proprioception is faster than nociception
Afferent inhibition
47
Deep joint, thin, myelinate A-beta fibers
Group III nociceptors
48
Deep joint, unmyelinated, C fibers
Group IV nociceptors
49
Functional disturbance of pain pathway
Neuropathic pain
50
Reversible inappropriate neurological response to environment, errors in = errors out (safety pin cycle gone bad)
Dysponesis
51
Pain d/t stimulus which doesn't normally provoke pain
Allodynia
52
Pressure on nerve (direct or indirect)
Neurothlipsis
53
Autoimmune attack on C1 transverse ligs (adjustments help)
Grisel syndrome
54
Distorted or impaired voluntary movement
Dyskinesia
55
Impairment of viscera d/t nervous system, can by measured by skin temp. differentiation
Dysautonomia
56
Neuropathic, radicular, deep pain, even felt on skin, but long lasting
Deep nociception
57
Fast, quick, short bursts of pain, not recognized in long standing conditions
Cutaneous nociception
58
Simultaneous pain in structures innervated by shared spinal segment
Referred pain
59
Brain interprets pain from multiple tissues. Rationale for referred pain, such as cervicogenic headache
Central convergence projection
60
Transection of nerve causing multiple synapse recovery
Traumatic neuroma
61
Chronic instability resulting in mechanoreceptors morphing into nociceptors. When the injury has healed & motion is restored, the pain will subside b/c nociceptors revert back to proprioceptors
Wide dynamic receiver (WDR)
62
What are autocoids?
Chemical mediators released by connective tissue that cause pain, joint morphology, & decrease motion