Famous People/Laws/Definitions Flashcards Preview

Part II/III Board Review > Famous People/Laws/Definitions > Flashcards

Flashcards in Famous People/Laws/Definitions Deck (62):
1

Subluxations are from abnormal biomechanics caused by muscle imbalance in a weight bearing spine. Pelvic distortion model (Basic distortion of the spine starts in the pelvis)

Carver

2

Developed a protocol for the tx of lumbar disc protrusion, spondylolisthesis, facet syndrome, subluxation, & scoliotic curves

Cox/McManus (Flex/dist)

3

Developed SOT. CSF flow through the pumping action of the sacrum & the cranial dura mater

DeJarnette

4

Fixation theory of joint hypermobility

Gillet/Fave

5

Subluxations are all pos. w/ disc wedging

Gonstead

6

Upper cervical specific; dentate lig.

Grostic

7

Found that disc herniations, exostoses, or subluxations may produce pressure on the dorsal nerve root

Hadley

8

Father of homeopathy (tx's pts w/ heavily diluted preparations which are thought to cause effects similar to the symptoms presented)

Hahnemann

9

Father of modern medicine; first to manipulate

Hippocrates

10

Joint hypermobility; described SI movement & pelvic dynamics. Discovered & tested SI ligs.

Illi

11

3 phase model of instability (dysfunction, unstable, stabilization); Breaking up spinal adhesions on an injured segment

Kirkaldy/Willis

12

Segmental Facilitation Theory. Established the concept of subluxation creating a hyperactive nervous system, rather than a decrease in nerve impulses. Muscle is central to his theory

Korr

13

Wrote the 1st chiro. textbook in 1906; people started to research & incorporate motion instead of "bone out of place" idea

Langworthy, Smith, & Paxson

14

Revised Faye's concept of the VSC

Skip Lantz

15

Founder of technique where sacrum is keystone of spine, first to use heel lifts

Logan (Logan Basic Technique)

16

World renown scientists in the field of biomechanics of the spine

Panjabi & White

17

General Adaptation Syndrome; under optimum conditions the body can respond to stressors

Selye

18

Wrote chiro textbook. Identified 33 principles of chiro. & "safety pin" cycle

Stephenson

19

Founder of osteopathy in 1856; utilized the circulatory system

Andrew Taylor Still

20

Came up with name "chiropractic"

Samuel Weed

21

Steady deformation d/t sustained axial pressure

Creep

22

Energy loss from loading & unloading disc (heat)

Hysteresis

23

Long standing creep w/ hysteresis event

Buckling syndrome

24

Deformation increases in proportion to the load applied

Hooke's Law

25

Opposing translation of two body parts

Sheer

26

Any recoverable deformation

Elastic deformation

27

Property of a material or structure that returns it to its original form following the removal of the deforming load

Elasticity

28

Nonrecoverable deformation

Plastic deformation

29

Property of a material to permanently deform when it is loaded beyond its elastic range

Plasticity

30

Property of returning to the former shape or size after distortion

Resilience

31

Property of a material showing sensitivity to the rate of loading or deformation, two basic components are viscosity & elasticity. Slowly deforms, slowly reforms

Viscoelasticity

32

Property of materials to resist loads that produce shear. Strain delay

Viscosity

33

Balance of compression & tensile forces on connective tissue

Tensegrity

34

Increase epiphyseal plate pressure = decreased growth
Decreased epiphyseal plate pressure = increased growth

Heuter Volkman

35

Bone (structure) responds to stress (function)

Wolff

36

Soft tissue responds to stress

Davis' law

37

A nerve which supplies a joint, supplies the muscle, skin, & tissue around it (axoplasmic aberration)

Hilton's law

38

Pain threshold, pain managment

Algometry

39

Nitrogen release during adjustment (joint capsule seal)

Cavitation

40

Skin rolling over spine for dx & tx

Roulement

41

Prox. neurological irritation causing peripheral neuropathies

Double Crush

42

CNS remapping, reorganizing, & brain expansion. PNS synapse (?)

Neuroplasticity

43

Opiates from peri-aqueductal grey descend to bathe dorsal horn & reduce incoming pain messages

Descending inhibition

44

Facilitation d/t acute nerve compression bombardment of dorsal horn, "endogenous opiates" (PAG) release shakes off pain for body to respond to immediate threat

Descending modulation

45

Gate control theory (Wall's), fast conducting proprioceptor & mechanoreceptor messages close the gate of slow pain receptors to CNS

Inhibiting system

46

Distraction principle such as: rubbing elbow, TENS unit. Proprioception is faster than nociception

Afferent inhibition

47

Deep joint, thin, myelinate A-beta fibers

Group III nociceptors

48

Deep joint, unmyelinated, C fibers

Group IV nociceptors

49

Functional disturbance of pain pathway

Neuropathic pain

50

Reversible inappropriate neurological response to environment, errors in = errors out (safety pin cycle gone bad)

Dysponesis

51

Pain d/t stimulus which doesn't normally provoke pain

Allodynia

52

Pressure on nerve (direct or indirect)

Neurothlipsis

53

Autoimmune attack on C1 transverse ligs (adjustments help)

Grisel syndrome

54

Distorted or impaired voluntary movement

Dyskinesia

55

Impairment of viscera d/t nervous system, can by measured by skin temp. differentiation

Dysautonomia

56

Neuropathic, radicular, deep pain, even felt on skin, but long lasting

Deep nociception

57

Fast, quick, short bursts of pain, not recognized in long standing conditions

Cutaneous nociception

58

Simultaneous pain in structures innervated by shared spinal segment

Referred pain

59

Brain interprets pain from multiple tissues. Rationale for referred pain, such as cervicogenic headache

Central convergence projection

60

Transection of nerve causing multiple synapse recovery

Traumatic neuroma

61

Chronic instability resulting in mechanoreceptors morphing into nociceptors. When the injury has healed & motion is restored, the pain will subside b/c nociceptors revert back to proprioceptors

Wide dynamic receiver (WDR)

62

What are autocoids?

Chemical mediators released by connective tissue that cause pain, joint morphology, & decrease motion