Modalities Ch. 6 Flashcards

(46 cards)

1
Q

orthopedic injury model

A

normal tissue

  • cells
  • two blood vessels
  • nerve
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

immediate ultrastructural change

A

primary traumatic damage
damage to the nerve
breakdown of the injured cells

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

hemorrhage

A

few minutes only

clot forms, stopping hemorrhage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what happens of a clot forms

A

hematoma forms

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

how does a clot form

A

fibrins form a network

-platelets are caught and it created a plug

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

pain from damaged nerve

-results

A

muscle spasm and more pain
neural inhibition
-leads to decreases in strength and ROM

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

the damaged cells release…

A

chemical mediators as a signal to the body that an injury has taken place

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

result of broken blood vessels

A

extravascular swelling

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

chemical mediators released from dying cells cause…

A
hemodynamic changes
permeability changes
-histamine
-kinins
-serotonin
leukocyte migration
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

what happens at this point?

A

secondary enzymatic injury

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

hemodynamic changes

A
blood flow slows down
OR
blood flow ceases
result
-tissue oxygen decreases
--hypoxia
--metabolic changes
-secondary hypoxic injury soon seen
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

phagocytosis

A

as cells are broken down free proteins are released

free proteins signal edema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

pressure on undamaged nearby pain fibers cause additional…

A

pain

muscle spasm and inhibition

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

immediate care

A

subset of acute care

first 12 hours

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

RICES

A
Rest
Ice
Compression
Elevation
Stabilization
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

why RICES?

A
protection from further damage
decrease or minimize the development of
-swelling
-pain
-muscle spasm
-neural inhibition
-secondary injury
-total injury (because of decreased secondary injury)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

time course of swelling

A
immediate swelling
-comes from hemorrhaging
edema
-begins minutes to hours after injury
-continues to develop over many hours
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

secondary injury and edema

A

secondary injury leads to edema
increased edema increases secondary injury
two mechanisms
-increased distance between blood vessel and tissue cells
-edema can compress the blood vessel
both decrease circulation
-decreased oxygen to tissues

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

rest: why?

A

limits injury aggravation

  • should be total during immediate care
  • transition to “relative” rest
  • different from, but related to, stabilization
20
Q

ice: why?

A

most think it’s to control swelling
-important but only part of immediate care
limiting secondary injury and neural inhibition are most important than controlling swelling

21
Q

decreased blood flow theory

A

older, traditional theory
cold decreases blood flow
logic of theory
-cold causes vasoconstriction
-vasoconstriction decreases blood flow
decreasing blood flow decreases hemorrhaging
decreased hemorrhaging = swelling reduced

22
Q

decreased blood flow theory flaw

A

clotting occurs before tissue is cooled enough to substantially decrease blood flow

23
Q

decreased secondary injury theory

A

cold decreases cellular metabolism

  • reduces oxygen demand (consumption)
  • less secondary hypoxic injury
  • less total injury
  • less free protein generated by phagocytosis
  • less edema
24
Q

metabolism and cooling

A

direct relationship

-greater cooling = greater decrease in metabolism

25
cryotherapy and swelling
decreases edema, not hemorrhage -most occurs hours after injury, as free protein accumulates from phagocytosis cold reduces edema formation -by preventing secondary injury and thus less free protein accumulation
26
compression: why?
controls edema beyond normal volume (i.e. after tissue spaces are filled) slows swelling apply constantly until swelling is over
27
compression during immediate care | -RICES added benefit
enhances cooling
28
elevation: why?
decreases capillary hydrostatic pressure
29
stabilization: why?
controls neural inhibition -process in which neural pathway is blocked so impulses cannot pass --intended function does not occur -partial: function is diminished -total: function is abolished temporarily allows muscles to relax, thus -decreases pain-spasm-pain cycle -numerous braces and splints for stabilization
30
cryotherapy physics and physiology
cold = the absence of heat conduction -heat exchange until two substances have exchanged energy and meet in the middle
31
rate of conduction factors
``` temperature differential regeneration of body heat and modality cooling heat storage capacity size of the cold modality amount of tissue in contact with pack length of application individual variability ```
32
heat storage capacity principles
specific heat | latent heat of fusion
33
specific heat
amount of heat energy required to raise 1 kg of a substance 1 degree C
34
latent heat of fusion
amount of heat energy need to convert a substance from a solid state to a liquid state -phase change ice to water = 80 cal/kg
35
crushed ice pack vs. gel pack
``` crushed ice is better -gel pack approx. 22 kcal --water from -17 C to 5 C crushed ice pack approx. 86 kcal -phase change -from -1 C to 0 C ```
36
factors that affect tissue cooling
``` type of cold pack duration of application size of cold pack amount of tissue in contact with pack depth of target tissue method of application ```
37
factors affecting temperature changes after cold pack application
depth of tissue amount of heat available to rewarm area activity level before cryotherapy amount of heat removed from body during application (magnitude and duration of cold application)
38
types of cold packs
four general types - crushed ice - gel - artificial ice - chemical
39
crushed ice
most effective because of heat of fusion excellent for on-the-field use should not be used if kept in a freezer (too cold)
40
gel pack
water, alcohol, and gel substance cooled to 1 F -dangerous should only be applied with barrier between skin and pack
41
artificial ice
water in vinyl pouches, surrounded by nylon cover (Dura Kold) better than gel because of heat of fusion not as good as crushed ice because of insulation from nylon cover good for home use
42
chemical: double bags, crushable
chemical reaction when inner bag is crushed and contents react with outer chemicals least cooling
43
frozen peas
``` popular; cheap and convenient effects -better than nothing and gel packs -worse than crushed ice packs or ice water immersion -little phase chang with peas avoid if possible ```
44
cold application protocol
apply directly to skin, not over a towel or elastic wrap - except for gel packs - except for crushed ice from freezer
45
cold: length of application
intermittent -30-60 minutes every two hours depth of target tissue adipose tissue - leave on longer
46
average ice times based on area
finger: 20 minutes ankle or arm: 30 minutes thigh: 45 minutes +5 minutes for each millimeter of skin-fold >1 mm