Modalities Ch. 8 Flashcards

1
Q

what is pain?

A

subjective
#1 reason an athlete seeks tx
physiological
modulates response to a pain inducing stimulus

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2
Q

types of subjective pain

A

emotional
psychological
social

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3
Q

why is pain physiological

A

it is associated w/ tissue damage

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4
Q

cause and effect types of pain

A

simple pain
response aimed at protecting and healing
simple pain can become chronic pain (> 3 months)

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5
Q

simple pain

A

acute pain
tissue damage
most sports-related pain
alarm system alerts brain to actual or potential tissue damage

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6
Q

how does pain protect and heal the body?

A

it signals the brain that there is something wrong

plays a role in initiating the healing processes

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7
Q

how can simple pain become chronic?

-% of pop. that suffers from chronic pain

A

untreated
incorrect treatment
20% suffer

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8
Q

consequences of chronic pain

A
depression
decreased activity/disability
atrophy
loss of skill
anxiety
circulatory deficiency
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9
Q

how is pain described?

A

subjectively
-burning, sharp, achey, deep
we try to make it objective
-1-10 pain scales

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10
Q

nociception

A

ability to feel pain

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11
Q

nociceptors

A

specialized nerve endings that respond to stimulus - mechanoreceptors, thermoreceptors, chemoreceptors

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12
Q

how are nociceptors classified

A

by location

  • cutaneous
  • somatic
  • visceral
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13
Q

which nociceptors are most common?

A
mechanical nococeptors (a-delta, force)
polymodal nociceptors (C, heat, mechanical pressure)
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14
Q

function of A-delta fibers

A

pain transmission
crude touch
pressure
temperature

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15
Q

function of C-fibers

A

pain
touch
pressure
temperature

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16
Q

nociception vs. pain

A
nociception
-specific neurophysiological activity
pain
-interpretation of nociception
--subjective and emotional
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17
Q

impacts on pain

A

past pain experience
general life stress
heredity
personality differences

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18
Q

common noxious stimuli

A

mechanical - pressure directly on a nerve (swelling, muscle spasm)
thermal
electrical
chemical - mediators that are present when injured, bradykinin

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19
Q

pathway of pain

A

slam hand in car door
pain signal - chemicals released
chemicals signal receptor to generate a signal
signal travels up the arm on first-order neurons
transmit to the dorsal horn of the spinal cord
signals relayed to a second order neuron
neurons transmit the signal up the spinal cord
received at the thalamus
pain signal transmitted to the cerebral cortex by the third order neuron
pain signal is processed

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20
Q

pain terminology

A

perception threshold
-when it starts to hurt
tolerance threshold
-when someone acts on the pain

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21
Q

Specific Theory of Pain

A

specific nociceptors
specific nerve pathways
specific sensation

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22
Q

Pattern Theory of Pain

A

no existence of specific pain receptors
slow-conducting nerve fibers carry pain
fast-acting nerve fibers carry other sensations
normally, the fast-conduction fibers override the pain
under pathological conditions, the intensity of the stimulation in the slow system becomes much greater and dominates the fast system, resulting in pain

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23
Q

Pattern Theory of Pain flaw

A

too general - doesn’t account for receptor specialization

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24
Q

Gate Control Theory

A

specificity + pattern theory
operates at spinal level
proposes a gate mechanism
central control

25
gate mechanism
-pain and other sensory stimuli travel along both large and small nerve fibers -both stimulate T-cells which pass onto brain action system includes all responses to the pain inhibitory mechanism located in the substantia gelatinosa SG inhibits the T-cell differential stimulation of SG cells
26
what types of pain do the large and small fibers carry?
large: sharp, stinging small: dull, aching
27
how do large and small fibers stimulate the SG?
large: facilitates SG small: inhibits SG
28
Gate Control Theory problems
does not account for phantom limb pain | cannot explain influence of drugs on pain
29
types of pain relieving drugs
``` morphine naloxone enkephalin endorphin serotonin ```
30
morphine function
blocks pain - fills neurotransmitter sites - neorutransmitter blocked
31
naloxene function
morphine antidote: reverses effects
32
enkephalin
- mixture of two peptides - similar in shape and similar analgesic properties as morphine - found in brain, spinal cord, gut - half-life very short thus operates at spinal cord level - block the gate by interfering w/ A-delta and C fiber signal transmission to T-cells - released through non-painful sensory stimulus - -sensory TENS, gentle massage
33
endorphin - similar to... - where is it produced? - half-life - where does it act? - how does it work? - how are they released?
endogenous molecule similar to morphine produced in pituitary and circulated through body half-life = four hours acts in several areas of CNS (including dorsal horn) inhibits pain signal transmissions and decreases chemical irritants in CNS may be released by acupuncture and intense exercise
34
serotonin
biochemical messenger and regulator found primarily in CNS, GI tract, and blood platelets mediates several physiological function, including neurotransmission may influence pain perception via descending tract (brain to spinal cord), block signals from peripheral nociceptors
35
dopamine
neurotransmitter in extrapyramidal system of brain important in regulating movement may also be used to synthesize norepinephrine and epinephrin affects brain processes ___________
36
opoids
endogenous opiates term used to denote body's internal pain killers such as enkephalin, endorphin, serotonin, and dopamine bind to opiate receptors in the brain operate in different parts of nervous system effective for varying lengths of time
37
levels of pain control
level I: ascending influence pain control level II: descending influence pain control level III: beta-endorphin-mediated pain
38
level I: ascending influence pain control
traditional gate control pain relief theory gating mechanism induce pain relief TENS, massage, cryotherapy
39
level II: descending influence pain control
pain relief occurs in higher brain centers release of enkephalin modulates pain possible explanation for C-fiber activation relieving pain ex.: acupuncture or noxious TENS
40
level III: beta-endorphin-mediated pain
prolonged stimulation of A-delta fibers triggers the release of beta endorphins longer term pain relief due to the 4hr half-life of endorphins
41
dimension of pain
``` social emotional psychological physiological therapy is an art ```
42
function of social, emotional, and psychological dimensions of pain
reasons why therapy doesn't work | affect the rate of therapy
43
how to make a patient the owner of their pain
``` communication distraction education use it as a positive responsibility -pain meds. -treatment ```
44
when do we ignore pain
``` minimal chronic sore vs. pain denial inconvenient/annoying conditioning ```
45
when do we listen to pain
``` when it is significant interferes with everyday life visceral familiar -past experience ```
46
what does it mean to respect pain
``` understand when it needs attention knowing when to stop/continue -past experience -type of pain -understanding consequences ```
47
Dehne's Spinal Adaptation Syndorme
afferent nociceptive impulses from traumatized tissue alter the integration of central nervous excitation at the spinal cord -decreased response to volitional stimuli -increased response to otherwise subliminal peripheral stresses results in involuntary muscle action alters repair responds adversely to addition stress, favorably to reestablishing central control nociceptive impulses from traumatized tissue inhibit motor function and tissue repair voluntary activity can reestablish CNS control and prevent this inhibition prolonged inactivity after injury will lead to neural inhibition that may become permanent
48
resetting central control
not enough to just get rid of the pain sensation must also get rid of the effects of the pain -reset the system
49
Chicago Plane Analogy
a tire blowout occurs during takeoff the runway is shut down so the damaged plane and debris can be romoved the runway reopens (pain removed) however, thousands of passengers are stranded because of canceled flights (Chicago and elsewhere) meetings must be rescheduled, which takes time after the reopening of the airport
50
resetting central control after injury
progression exercise
51
source of athletic injury pain
``` nociceptor pain -cause of most sports injury pain relay impulse to spinal cord nociceptor is stimulated by -injured tissue (mediator release) -edema pressure -stretching injured tissue -otherwise normal activity in a tissue that is sensitized from disuse after injury ```
52
source of athletic injury pain
neuropathic pain -complex, chronic pain state accompanied by tissue damage to nerve fibers occurs due to -acute nerve damage -disuse following injury -surgery that involves neural tissue -condition that cause nerves to become dysfunctional
53
sources of athletic injury pain
``` learned pain -learned, or conditioned, response --"pain memories" -_________________ idiopathic pain -unknown origin --no identifiable pathology ```
54
placebo - latin - ___% receive relief
``` latin-I shall please mock intervention patient belief psychological effects 35% of postoperative, diabetes, or chronic headache patients receive relief from placebos ```
55
placebos | -why?
``` power of the mind believing something will work may increase effects -educate patients -be positive -set goals ```
56
pain relief types
direct | indirect
57
direct
get rid of the pain source
58
indirect
treating the pain but not the source
59
tools for relieving pain
``` immobilization therapeutic exercise counter-irritants thermotherapy electrotherapy cryotherapy psychological tools ```