acute GE - SA Flashcards
(33 cards)
most cases of acute GE are fatal y/n
n, most self limiting
name 2 common causes of acute, non-fatal self limiting GE
- parasites
2. dietary indiscretion
name 4 general causes of severe, pos fatal GE
- enteric infection (parvo, bacterial)
- surgical dz
- HGE (acure haemorhagic d+ syndrome..)
what is the major sign of sx dz wrt GE
V+
how much fluid goes IN /day of a 20kg dog
2,700ml/d
what % of the total fluid IN is absorbed by the GIT
98%, the rest into poo
what are the remaining factors which must be considered before a tx and dx is made
what supportive tx is needed
are there underlying dz causing this - pos non-enteric ones
then all the hx, and signalment etc
there are 3 categories of GE of which are often combined in various ways
- gastritis
- enteritis
- colitis
what is haematochezia
fresh blood in poo (colitis suggested)
both gastritis and enteritis have similar causes and often combined what are they
dietary indiscretion foreign body bezoar self-limiting intoxication/drugs etc
colitis has a number of other causes - name some
whipworms, garbage ing, protozoa (giardia, crypto)
how does colitis present differently to GE
tenesmus, not vomiting
rare in cat
after symptomatic tx how long should you give a case to imprve
48hrs
what is the min database from a lab that should be gained to get a dx for suspected GE
haematology
serum biochem
urinalysis (to check systemic dz)
faaecal exam for para
what procedures are carried out to check for para in poo
virology - ELISA for ag parasitology - smears and flotation (giardia) - wet-prep for tritrichomonas (C) or PCR serology - snap test for giardia and parvo
why are radiographs a good dx tool in GE cases
assess if obstruction
u/sound
intussusceptin and forign bodies
contract - rarely req
in a per-acute emergency cases - what would your dx tools/actions be
PCV/TP - assess DH, anaemia, protein losses
blood smear for inflam, haemolysis, anaemia, PLTs
glucose - if very young, often poor gluc control
urea
urinalysis - haemolysis
what are the pros/cons of withholding food in animals w d+ and V+
- less to V+ or d+ (management easier), will make more nauseous
- good to feed - less likely to be septic
- speedier recovery if feed through d+
how do anti-emetics act?
centrally at CTZ
- metoclopraminde, chlorpromazine
anticholinergics
- atropine,
what is the generic name for cerenia
maropitant
what is maropitant - cerenia
NK-1 R antago 1xd dose (s/c inj or oral) SE = v+; CI = if sx dz or obstruction
why woudl you use gastric mucosal protectants
V+ persists or ulceration
what receptors and how do drugs act to prevent ulceration
H2-R antagonists = cimetidine (v freq dosing and SE), sucralfate (antacid and mucosal protection), antacids like MgOH
whya re NSAIDs CI?
damage GI mucosa and damage kidney if they are DH
