eq colic Flashcards

(48 cards)

1
Q

what are the signs of colic

A
flank watching
lying down
pawing ground
rolling
restless
thrashing
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2
Q

why is colic so common

A

LI is large and very loosely/not at all attached! so it goes wandering

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3
Q

where are the common places that obstructions ocur

A

sternal, diaphragmatic, pelic flexures

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4
Q

how can colic lead to shock

A

loss of vascular supply
abs of endotox into circulation
SIRS

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5
Q

name the 7 main diff colic classifications

A
spasmodics
impaction
distention
obstruction
infarction
inflam
idiopathic
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6
Q

what advice would you give an O about a colicing horse before you arrive

A
  • wellbedded stable
  • remove buckets/feed/anything it could hurt itself on
  • let it roll
  • walk it for 10 mins max if you really want
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7
Q

if the horse is violently painful - how do you assess it safely??

A

assess HR (takes a LOT of pain to increase this!!) and RR from distance if poss - check mm colour
administer IV xylazine (220mg for 500kg horse) to analgese and sedate
key aspects of Hx and performe CE

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8
Q

what are impt Hx questions pertaining to colic

A
  • steroptypies eg windsucking, crib biting
  • management (stables, pasture)
  • parasite tx?
  • changes to feeding or turnout
  • hx of colic?
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9
Q

what are the impt q regarding the colic episode to ask?

A
  • when started
  • duration
  • how severe/actions
  • feed in and poo out? d+?
  • hx of grass sickness on the yard
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10
Q

late stages of acute colic may present as trauma - dont forge to think. but what other forms of ‘fase colic’ are there?

A

EVERYTHING possible practically..

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11
Q

what is the norm RR and HR and temp

A

HR - 28-44 (30)
RR = 12-15 (15)
T = 37.5-38.4

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12
Q

describe what major features can be felt in rectal palpation of the horse - in quadrants

A
LD = SI
LV = L colon
RD = caecum
RV = R colon
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13
Q

why is it impt to ascultate the abdo in horses

A

external palp impossible

listen for 1 min and record whether borborygmi -, ++ (norm), + (less) or hyper +++

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14
Q

how can you ID the SI from small colon (asc)

A

inner tubes feel

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15
Q

how can you ID the LI sections

A

taenia number and arrangement

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16
Q

what ID features does the caecum have

A

vertical band

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17
Q

name some common abnormalities in acute colic pres

A

dist SI
pelvic flexure, small colon, caecal impaction
LD/RD displacement
LI torsion

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18
Q

why should abdominocentesis be conducted in acute colic cases

A

any GI wall changes = this will chnage too

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19
Q

desc basic techn of abdominocentesis

A
  • V midline
  • clip and scrub
  • 18g, 1.5inch needle
    CI in foals and if v distended
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20
Q

why is it that if there is an intestinal blockage, the stomach can rupture

A

they cant vomit so still prod fluid –> put in a n-gasto tube. if you get >2L then thats significant!!

21
Q

why is colic in the foal difficult to assess

A

they dont like to show pain at all!
meconium impaction common
consider developmental abn and bladder rupture

22
Q

do donkeys usually show signs of colic?

A

no but impaction common. spinal needle req for abdominocentesis as fat bellied

23
Q

what is equine dysautonomia

A

grass sickness

24
Q

desc the dz of grass sickness

A

paralytic ileus
a, s/a and chronic
susp c. botulinum type c infection

25
what is the definition of chronic colic
signs of variable intensity for 48hrs+
26
what is the defin of recurrent colic
short episodes, variable intervals
27
name som eGI causes of recurrent colic
intermittient/paritial obstruction inflam motility disorders mesenteric traction/displacment
28
what factors can you ask O to record/keep diary of for a case of recurr colic
- number and nature of colic episodes - faecal output/d, kg loss, d+ - diet and intake - FEC - stereotypies, dental problems - type of turnout
29
what are the most likely cause og hypoalbuminaemia and hypoglobulinaemia?
GI losses
30
what is the likely cause of hyperglobulinaemia
chronic infection/inflammation (cyathostominiasis)
31
what is the likely reason for hyperfibrinogenaemoa
inf, inflm or neoplasia
32
what acute phase protein is a good marker for colic progression
serum amyloid a
33
after you have taken any serum biochem and a hx etc, what measurements need to be taken during stay in hosp?
temperature - several times a day. if varies consider abscess/neoplasia. take peritonal fluid
34
desc process and value of oral glucose abs test
- only assessing SI ability - starve overnight 1g/kg in 20% sol via n-g tube - dont sedate with a-2 as will dec motility
35
why would rectal and duodenal bsy be wanted in the weight loss chronic colic case
IS any pathology there - assess villi etc
36
what can you assess from trans rectal u/s
wall thickness of intestines lumen size motilty abnalities
37
name some ddx for protein-losing enteropathy / malabs
``` cyathostomins strongyles idiopth infiltrative bowel dz neoplasia lawsonia in folas ```
38
the aetiology of infiltrative bowel diseases are unknown, but they are granulomatous, eosinphillic or lymphocytic enteritis'es. whats the tx?
predn dex anthelmintics
39
what is a multisystemic infiltrative bowel dz?? name 2
1. multisystemic eosinophillic epitheliotropic dz = coronary bnds, pancreas and liver. tx with dex 2. systemic granulomatous dz = skin and other affected
40
name the 5 locations of equine lymphoma
1. alimentary = young horses 2. generalised = old horses 3. solitary = any age 4. cr. mediastinal = any age 5. cutaenous = any age
41
what are the clinical clues of lymphosarcoma
- fever, kg loss - peritonitis, pleural effusion, abdo distention - hypercalcaemia, haemolysis, cachexia
42
what is an 'inflammatory' haemogram
neutrophillia hyperfibrinogenaemia anaemia
43
what are the most common causes of chronic bacterial infection
strep equi equi | rhodoco. equi
44
how does the parasitic haemogram differ in horses to other mammal
NO eosinophillai | it is: neutrophillia, hypoalbuminaemia, hyperglobulinaemia
45
what 3 parasites are the most common cause of coli
strongylus vulg = verminous arteritis and thromboembolic colic cyathos = submucosal inflam parascaris equorum
46
gastric ulcers are a cause of kg loss and poor performance. what are the RF/
stress acid injury - too much hCHO--> VFAs horses continuously prod acid low fibre diet = less saliva produced to neutralise
47
signs of gastric ulcer =
kg loss poor performance selective appetite (roughage not grain) cranky
48
which of squamous ulcer or glandular ulcer responds better at low doses of H+ pump inhib
squamous - @ 2mg/kg/d for 4wks whereas glandular - 4mg/kg/d 6wks