Chapter 16 Flashcards

1
Q

Cardiac Output (CO)

A

CO = SV (Stroke Volume) x HR (heart rate)

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2
Q

End- diastolic volume

A
  • the preload

- amount of blood returned to the heart

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3
Q

Systemic Vascular Resistance (SVR or afterload)

A
  • determined by the radius of arteries and the degree of vessel compliance
  • SVR = BP/CO
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4
Q

Blood Pressure (BP)

A

BP = CO x SVR

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5
Q

Hypertension

A
  • increases morbidity and mortality associated with heart disease, kidney disease, peripheral vascular disease, and stroke
  • Is determined by the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure
  • is classified as a higher than usual Diastolic and/or Systolic blood pressure
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6
Q

Prehypertension

A
  • a range of pressures between normal and stage 1 hypertension in an effort to initiate interventions early enough to prevent or deter progression of the disease process
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7
Q

Numbers the classify Hypertension

A

Normal: SBP < 120 and DBP < 80
Prehyp: SBP 120-139 and DBP 80-89
Stage 1 Hyp: SBP 140-159 and DBP 90-99
Stage 2 Hyp: SBP >or= 160 and DBP >or+ 100

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8
Q

Primary Hypertension

A
  • also called essential hypertension
  • is an idiopathic disorder
  • most common form
  • Systolic BP: major risk factor of CV disease
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9
Q

Subtypes of Primary Hypertension

A

Isolated systolic: SBP >or= 140 while DBP < 90
Isolated Diastolic: DBP >or= 90 while SBP < 140
Combined: Both exceed hypertension levels

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10
Q

Nonmodifiable risk factors of Primary Hypertension

A
  • family history
  • age
  • ethnicity/genetics
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11
Q

Modifiable Risk Factors of Primary Hypertension

A
  • diet/weight
  • Metabolic issues
  • high blood glucose levels/diabetes
  • high cholesterol
  • alc and cigs
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12
Q

Outcomes of Primary Hypertension

A
  • sometimes called the silent killer as damage to organs is done before diagnosis
  • Results in end-organ damage
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13
Q

Treatment of Primary Hypertension

A
  • lifestyle changes such as weight loss and exercise, decrease in sodium intake, moderation of alcohol
  • Drug therapy but will affect heart rate, SVR, and/or SV
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14
Q

Secondary Hypertension

A
  • hypertension attributed to a specific identifiable pathology or condition
  • most common in infants and preschoolers
  • most common cause is renal disease and coarctation of the aorta
  • other causes are obstructive sleep apnea
  • Adult secondary can be attributed to : Renal artery stenosis, Pheochromocytoma, Pregnancy, Obesity/sleep apnea, and Hyperaldosteronism
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15
Q

Hypertensive Emergency

A
  • a sudden increase in either both SBP or DBP with evidence of end organ damage
  • treated with rapid but controlled reduction of blood pressure using parenteral antihypertensive agents under close monitoring (ICU)
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16
Q

Hypertensive Urgency

A
  • similar blood pressure elevation without evidence of end organ damage
  • treated with oral medications to bring blood pressure under control over 24-48 hrs
17
Q

Orthostatic (postural) Hypotension

A
  • an extreme response to the change from supine to upright position; activation of the short term control mechanisms is slow or inadequate
  • causes a decrease in SBP (>or= 20 or >or=10 in 3min) when moving to an upright position
  • excessive increase in heart rate may also be diagnostic (20-30 BPM)
  • results in dizziness(presinkapy), blurred vision, confusion, and possible syncope
18
Q

Orthostatic Hypotension may be the result of

A
  • problem with vasomotor or baroreceptor responese
  • adverse effect of drug therapy
  • arterial stiffness
  • volume depletion
  • secondary disease process
  • Vasovagal reaction
  • cardiac dysrhythmias
19
Q

Treatment for Orthostatic Hypotension

A
  • review medical history
  • slow positional changes
  • avoid high temps
  • avoind large carbs/heavy metals
  • squatting /bending forward or crossing legs at onset can reduce effects
  • elastic compression stockings, abdominal binders, elevate head of bed
  • increase salt and fluid intake
20
Q

Renin-Angiotensin-aldosterone system (RAAS)

A
  • important regulator of BP
  • juxtaglomerular cells when stimulated by low arterial pressure release renin which activates angiotensinogen to angiotensin I
  • Angiotensin I when in contact with ACE activates angiotensin II, a potent vasoconstrictor and stimulates release of aldosterone
  • Aldosterone, a hormone, causes reabsorption of sodium and water passively follows
21
Q

Monitoring Blood Pressure (measurement)

A
  • components of monitoring include SBP, DBP,, SV, SVR, and Mean Arterial Pressure (MAP)
  • ## there are both direct and indirect methods
22
Q

Mean Arterial Pressure (MAP)

A

the calculated average pressure within the circulatory system throughout the cardiac cycle
- MAP = (2 x DBP) + SBP/ 3

23
Q

Direct BP Monitoring

A
  • requires an intraarterial catheter and specialized equipment to transduce arterial fluid pulsations into electrical signals (waveforms)
  • catheder placed in radial artery
  • most accurate method
24
Q

Indirect BP Monitoring

A
  • commonly measuredd via the brachial artery using a stethoscope and sphugmomanometer or automated oscillometric system
  • Auscultation of Korotkoff sounds (SBP is onset of Korotkoff sounds) (DBP is disappearance of Korotkoff sounds)