Chp 18 class recording Flashcards

1
Q

Differences between cardiac muscle action potential

A

steep/fast depolarization
has a plateau
and then we repolarize

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2
Q

why does cardiac muscle plateau during action potential

A

we need to make sure is filling up all the ventricles

also because of calcium channels opening in the cardiac muscle fibers

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3
Q

cardiac muscle at rest is what milivolt

A

-90

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4
Q

what is the plateau for the cardiac muscles action potential

A

a maintained/sustained depolarization

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5
Q

what causes the plateau

A

the calcium channels being slow to open

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6
Q

does the heart gradually or quickly repolarize

A

gradually

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7
Q

does the cardiac muscle have hyperpolarization

A

no, but we do have a period of rest

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8
Q

depolarization happens because of

A

Na

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9
Q

repolarization happens because of

A

K

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10
Q

cardiac muscle action potential ion stages

A

Na
Ca
K

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11
Q

Everytime you see ca you know what is happening in the heart

A

contraction and plaeua is happening

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12
Q

even though we have no hyperpolarization we still need a protective mechanism so we aren’t sending messages to the sanode repeadtly. So when is that happening

A

in the middle of the plauteu is our absolute refractory period (when the heart is contracting)

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13
Q

the slow depolarization is only happening at the SA node until we get to

A

threshold

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14
Q

what kind of memebranes does the heart have and why

A

leaky, so I dont have to put as much effort into contraction

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15
Q

why does the heart have leaky membranes

A

to increase it’s own control

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16
Q

what can take over our pacemaker

A
sympathetic nervous system 
lack of calcium 
electrolyte balance 
caffeine, smoking, any kind of stimulant  
hypoxia (low oxygen) from anemia
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17
Q

why does hypoxia speed up the heart overriding the sa node

A

because the heart has to work harder to give every tissue he same oxygen

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18
Q

ectopic pacemakers are

A

outside factors overriding the pacemaker, stimulants, sanode damage hypoxia

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19
Q

EKGs do what

A

measure the electrical activity at each junction

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20
Q

electrical pathway of cardiac contractions

A

sa node
atrial muscle
av node
ventricular muscle

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21
Q

contractions only happen where in the heart

A

the ventricular walls

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22
Q

how long does it take the cardiac electrical muscle to send messages

A

.05 secs sa node to av node

.1 secs av node onwards (slows down)

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23
Q

why does conduction slow in the heart

A

to allow the atria to fill with blood, send to ventricles

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24
Q

three phases of the heart on EKG

A

p, QRS, T

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25
the most important phase in detecting actual contraction of the ventricles
QRS
26
T wave on EKG is going where
back to rest
27
p wave represents
sanode | atrial depolarization
28
if I have an inverted p wave what can you assume?
they need a pace maker | and something is wrong with the sanode
29
what does it mean if your QRS complex is drawn out?
contraction of the ventricles is slower
30
what does the T wave represent
ventricular repolarization
31
are invertted t waves common or uncommon... meaning what?
common, that we're not able to go back to rest and each contraction is not as strong
32
what makes the heart sounds
when valves close
33
the first heart sound we hear is made by
when our aortic and pulmonary valves close
34
second heart sound is heard when what
av valves are closed
35
what valves have to open to fill the ventricles with blood. And which valves are closed
av valves | exit valves
36
blood drops down and ventricles
contract
37
systole means
contraction of ventricles
38
diastole means
relaxation of the ventricles
39
isovolumetric contraction and relaxation is referring to
at the end of systole and diastole how much blood is left over
40
the cardiac cycle always starts with
blood coming back to the heart
41
ventricular contraction happens because
the av valves close
42
the beginning of the ventricular systole
when the av valves close
43
period of isovolumic contraction
systole (how much volume is in the ventricles at the end of systole)
44
diastole happens
immediately after we contract
45
period of isovolumic relaxation
diastole
46
what valves have to be closed during diastole
semilunar
47
at the end of diastole what is happening
the ventricles are completely filled and the av valves are open
48
in the middle of diastole what is happening
the ventricles are filling
49
initial filling of blood is called
isovolumetric relaxation | beginning of diastole
50
then the cusps are completely open allowing blood to fill up
complete relaxtion | middle of diastole
51
maxed out at that filling capacity of ventricles
end of diastole
52
isometric contraction. just now sending the message to the purkinges fibers sending message to contract. av valves are closed and semilunar valves are open
initial phase of systole
53
ventricular ejection of blood is what phase
middle of systole
54
semilunar valves close means
end of systole
55
what is stroke volume
how much blood is put out of the heart at each contraction
56
how well this container is able to hold all of us before it explodes is called
preload
57
contractility is exactly at the end of
systole
58
how well the ventricular cardiac cells contracting is what
preload and contractility
59
the pressure that the ventricles need to overcome because of volume in the atrium
afterload
60
how does norepinephrine from the sympathetic nervous system how does it override the heart
opens calcium channels bypassing the sanode
61
the heart increases the force contraction without increasing the length of the muscle is called
inotropic effect
62
if the heart is overworked it starts to build more
myocradium
63
an enlarged heart due to muscle growth
hypertrophy
64
intrinsic effect of your pacemaker is
the sanode
65
if your sanode is messed up who takes over
av node