Path V Flashcards

1
Q

what is the ring around infarcts seen microscopically around 2-3 weeks after incident

A

grnaulation tissue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

what is the syndrome called with posterior inferior cerebellar artery syndrome

A

lateral medullary syndrome

wallenberg

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Sx lateral medullary sydrome

A

loss of pain and temp on contra side
ipsi face
diagnostic!!!!!!!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what tract is damaged in lateral medullary syndrome causing loss of pain and temp to contra body

A

spinothalamic tract

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what part of brain is damaged in lateral medullary syndrome that causes ataxia

A

cerebellum or inferior cerebellar peduncle

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

what fibers can be damaged in lateral medullary syndrome that lead to horner like signs

A

hypothalamiospinal fibers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what nuclei involvement can cause nystagmus vertigo and nausea and vomiting

A

deiters nucleus and vestibular nuclei

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

what is a very bad complicaiton lateral medullary syndrome

A

increased P in posterior fossa that can cause tonsillar herniation affecting the respiratory center

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

what causes the fatal gastroeneteritis with lateral medullary syndrome

A

if vesticular and deitersnuclei involved

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

onset lateral medullary syndrome assoc with what

A

severe vertigo

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

widespread white matter hemorrhages

A

bone marrow embolization

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what can cause bone marrow embolizaiton

A

long bone fractures cause embolie

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

widespread white matter hemorrhages are seen in bone marrow embolization and what type

A

fat emboli

like in burn patient

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

TIA

A

transient ischemic attack

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

TIA lasts how long

A

1 hr leaves small infarcts

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

TIA important warning sign for what

A

stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

majority strokes caused by

A

HTN, antigoagulatns, subarachnodi hemorrhage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

HTN causes hemorrhage where

A

basalganglia area and thalamus and pons!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

hemorrhagic stroke occurs in what aa

A

lenticulostriate and recurrent a of heubner

both from MCA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

why do many patients with same stroke origination present differently

A

collateral circulation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

striatum

A

caudate and putamen

22
Q

lentiform nucleus

A

putamen and globus pallidus

23
Q

recurrent a of heubner suplies what

A

caudate head and ant putamen and globus pallidus

24
Q

all inputs basal ganglia arrive via what

A

striatum and nuc accumbens

25
outputs of basal ganglia leav how
internal segment globus palludus | close to substantia nigra pars reticulata
26
basal ganglia control what
general motor control eye movements cognitive functions emotional functions
27
pure motor stroke
contra pons or internal capsule
28
pure sensory stroke
contra thalamus
29
MCA syndrome
``` hemiparesis face and arm >leg aphasia sensory loss hemianopia eye deviation ```
30
ACA syndrome
lower extremity weakness, sensory loss, incontinence
31
PCA syndrome
homonymous hemianopia | sensory loss
32
imaging more sensitive to Dx small ischemic areas
MRI | but not as fast
33
what imaging is used to evaluate carotid aa and heart as sources of embolus
US
34
Tx TIA
preservation of tissue in penumbra t-PA and recanalizaiton if embolus watch for inc intracranial P
35
jet bleed into brain tissue
subarachnoid hemorrhage
36
where do intracranial aneurysms form
where there are gaps in media and internal elastica
37
majority berry aneurysms
circle of willis and 1st bifurcation MCA
38
if find one berry sneurysm must do what
look for others
39
patients at increased risk for intracranial aneurysms
W>M coarctation aorta (marfans) polycystic kidney disease since ciliopathy smoking and alcohol
40
what are arteriovenous malformations
aa and vv proliferation | inc risk of bleeding
41
what can occur intracranially in sturge weber syndrome
abnormal vessels in the subarachnoid space | can cause microcalcifications that cause seizures and neuro deficits
42
hemangioma in opthalmic division V
sturge weber
43
hypertensive encephalopathy
severe HA, nausea, vomiting, papilledema, visual disturbances, seizures, confusion and coma
44
fibrinoid necrosis small a
hypertensive encephalopathy
45
cerebral amyloid angiopathy
deposition of beta amyloid into small vessels | congo red +
46
cerebral amyloid angiopathy assoc with what
alzheimers because AD also deposits amyloid beta
47
what type of hemorrhage in Cerebral amyloid angiopathy
lobar
48
what phenotype assoc with cerebral amyloid angiopathy
ApoE
49
what stain for CAA
beta amyloid immunostain
50
the ischemic lesions in CAA can cause what clinical featues
dementia