Abnormal labour Flashcards

1
Q

too early

A

preterm birth <37 weeks of gestation

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2
Q

too late

A

beyond 42 weeks

induction of labour

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3
Q

too painful

A

requires anaesthetic input

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4
Q

too long

A

failure to progress

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5
Q

fetal distress can mean what

A

with every uterine contraction the blood supply to the fetus is cut off
leading to hypoxia/sepsis

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6
Q

inhalation agents meaning what

and for who

A

entonox for both high and low risk women

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7
Q

TENS is what

A

electrodes

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8
Q

IM opiate analgesia

SE

A

diamorphine

ECG changes, respiratory distress

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9
Q

what can be given as an intermediate between IM opiate analgesia and regional anaesthesia

A

IV remifentanil PCA

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10
Q
how effective is epidural anaesthetsia
does it impair uterine activity 
what may it inhibit 
what does it consist of
complications
A

complete pain relief in 95%

does not impair uterine activity

during stage 2

levobupivacaine +/- opiate

hypotension
dural puncture
headache
back pain - mainly mechanical
atonic bladder
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11
Q

what can an epidural anaesthetic lead to

A

puncture of the dura mate which leads to severe headaches due to CSF leak and photophobia - can’t stand up due to severe headaches

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12
Q

bladder and epidural anaethesia

A

interferes with the nerve supply to the bladder - ensure the bladder is not over distended

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13
Q

stages at which an epidural anaesthesia is given

A

test dose is given first

then it is regularly topped up

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14
Q

why can an epidural anaesthesia lead to misposition of the baby

A

relaxation of the pelvic floor so the head may not flex sufficiently so there may not be internal rotation

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15
Q

how is progress assessed in labour

A

cervical dilation
descent of the presenting part
signs of obstruction - caput/moulding

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16
Q

suspected delay in stage one for a nulliparous woman and a porous woman

A

nulli - <2cm dilatation in 4 hours

porous <2cm in 4 hours or slowing in progress

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17
Q

causes of failure to progress (3 ps)

A

powers - inadequate contractions: frequency and/or strength

passangers - short stature/trauma/shape

passenger - big baby, malposition

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18
Q

what does partogram assess

A
fetal heart
amniotic fluid
cervical dilatation 
descent 
contractions 
obstruction 
maternal observations
19
Q

what are the different ways of fetal assessing intra partum

A

doppler auscultation of the fatal heart during stage 1 during and after a contraction, every 15 mins and during stage 2 every 5-10 mins

CTG

colour of amniotic fluid

20
Q

risk factors for fetal hypoxia

what do they all require

A
small fetus
preterm/post dates
antepartum haemorrhage 
hypertension/ PET
DM
meconium 
epidural analgesia 
VBAC - vaginal birth after C sec
PROM > 24 hours
sepsis (temp >38c)
induction/augmentation of labour

continuous monitoring of the fetal heart

21
Q

what does VBAC have a risk of

A

uterus rupture

22
Q

whats does PROM have a risk of

A

sepsis and baby is at risk of septic brain injury

23
Q

how does induction lead to a risk for fetal hypoxia

A

hypersitmualated - more contractions - baby will be more stressed

24
Q

acute causes of fetal distress

A
abruption 
vasa praevia
cord prolapse
uterine rupture
feto-maternal haemorrhage 
uterine stimulation 
regional anaesthesia
25
sub acute causes for fetal distress and how is this assessed
hypoxia | using a CTG
26
what does a CTG monitor
baseline fetal heart rate variability accelerations/decelerations
27
what else does the CTG monster other than the fetal heart rate
contractions checked every 10 mins can't tell the strength - only the number
28
bradychardic fetal heart causes
hypoxia opiate analgesia malposition of the baby
29
tachycardia fetal heart causes
``` fetal stress active baby maternal dehydration intra uterine sepsis maternal sepsis ```
30
exaggerated variability means what | loss of variability means what
hypoxia opiate analgesia, sleep phase
31
early decelerations are caused by what | late decelerations are caused by what
physiological due to head compression pathological - mark of fetal hypoxia
32
what are complicated variable decelerations due to
mainly due to cord compression
33
What things need to be assessed and documented when reviewing the CTG
baseline fetal heart rate baseline variability presence or absence of decelerations presence of accelerations
34
how should a CTG be classed as
normal/non reassuring/abnormal
35
CTG interpretation | dr c bravado
``` determine risk contractions baseline r ate variability accelerations decelerations overall impressions ```
36
normal fetal heart rate
110-160bpm
37
management of fetal distress
change fetal position - take pressure off the aorta IV fluids stop syntocin and consider tocolysis (terbutaline 250 mcg s/c) to stop uterine contractions scalp stimulation maternal assessment fetal blood sampling - check acid base status
38
scalp pH normal boderline and abnormal pH and actions
>7.25 normal no action 7.20-7.25 boderline repeat in 30 mins <7.20 abnormal deliver baby
39
indications for operative (or not) labour
delay - failure to progress to stage 2 fetal distress maternal cardiac disease severe PET/eclampsia intra partum haemorrhage umbilical cord prolapse stage 2
40
what is failure to progress during stage two during prim/multips
prims 3 hour with epidural and 2 without multips 2 hour with epidural and 1 without
41
what are the risks of ventouse good things
increased risk of failure cephalohaematoma retinal haemorrhage maternal worry anaesthesia decrease vaginal trauma decrease perineal pain decrease
42
main indications for C sec
``` previous CS fetal distress failure to progress in labour breech presentation maternal request ```
43
risk with C sec
4 x greater maternal mortality
44
morbidity with C sec
``` sepsis haemorrhage VTE trauma TTN sub fertility regret complications in future pregnancy ```