cvs 1 Flashcards

1
Q

right dominant PDA arises from

A

RCA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

left dominant PDA arises from

A

LCX

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

coronary flow peaks in

A

early diastole

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

supply of SA node

A

RCA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

supply of AV node

A

PDA (depends on dominance)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

most dangerous MI

A

INFERIOR wall MI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

compensatory mechanism of HF

A

tachycardia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

decrease mortality drugs

A
SARB
Spirinilactone
ACE
RRB
BETA
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

stages of diastolic filling

A

Passive (70%)

Active (30%)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

S3 heard in

A

passive

due to overloading /pregnancy/ iv fluid

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

s4

A

active filling

late diastolic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

pathology of s4

A

HTN
HOCM
AS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

watershed area heart

A

subendicardial

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

brain

A

AM (aca mca)

PM (mca pca )

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

liver

A

zone III

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

colon

A

splenic flexure

rectosigmoid

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

kidney

A

medullary of PCT — has max atp pumps

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

sarcomeres in series

A

eccentric

volume overload

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

severity of MS depends on

A

opening snap time

short interval means severe stenosis

20
Q

sarcomeres in parallel

A

concentric

due to increase afterload

21
Q

ortner syndrome

A

LA enlargment

left recurrent laryngeal nerve of vagus —- hoarsness

22
Q

normal PCWP

23
Q

plop sound in

A

tumor c. myxoma

24
Q

increased PCWP

A

MS
MR
myxoma

25
virchows triad
hypercoag stasis injury to endothelium
26
a wave
atrial contraction S4
27
c wave
bulging of tricuspid valve into atrium
28
x descent
ejection
29
v
venous return against close TV
30
y dscent
early filling S3
31
prominent y
constrictive pericarditis
32
absent y
cardiac tamponade
33
absent a wave
a fib
34
cavo tricuspid
role in a flutter between IVC and tricuspid ablation done here
35
PANTT
``` PGI2 ---- # PLT agg ATIII NO TpA Thrombmodulin ```
36
PGI2 analog
epoprostenol pulm HTN can also use iloprost
37
iloprost SE
jaw painflushing
38
PTN DOC
bosentan | endothelin rec antagonist
39
sildenafil moa
pde5 # | increase cGMP
40
PGE1 analog
alprostadil smooth muscle relax mantains PDA P HTN (vasodilatory effect) erectyle dys
41
PGE2
DInoprostone | cervical ripening / abortion
42
PGF2alpha
smooth muscle contraction carboprost latanoprost abortion
43
latanoprost role in glaucoma
increases uveosacral outflow
44
increase NO in endothelium
``` ach bradykinin 5HT ANP BNP subs p ```
45
nitroprusside affect on veins + arteries
decrease preload | decrease afterload