TBI Flashcards

(41 cards)

1
Q

Risk factors for TBI

A

Ages 15-24 & 64+, men >women, ETOH, drug abuse, prior brain injury, SES/education, unemployment, divorce, LD/ADHD, psych illness, heart disease & HTN

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2
Q

Glasgow Coma Scale (GCS)

A

Assesses degree of impaired consciousness via responsiveness level in eye opening, motor mvmt, & verbal communication

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3
Q

What are the advantages of using GCS score for measuring TBI severity?

A

scores can be determined with 1st 24 hours, predictive of early important outcomes & later functional outcomes

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4
Q

What are the disadvantages of using GCS score for measuring TBI severity?

A

which GCS score to use (post-resuscitation, ER admission, etc.), can be affected by early management (intubation, sedating meds), limited in pts w/ aphasia or facial injuries

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5
Q

What are factors that complicate the use of initial GCS as a prognostic indicator?

A

Drugs/ETOH, sedation, intubation, facial injuries, echymosis, delayed deterioration (e.g., slow bleeding SDH)

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6
Q

Time to follow commands following TBI is predictive of

A

Global outcomes, NP functioning, personal independence, employment

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7
Q

What are the advantages to using time to return to conscious state as a marker for TBI severity?

A

takes into account early complications, can be obtained during early stages of recovery

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8
Q

What are the disadvantages to using time to return to conscious state as a marker for TBI severity?

A

interval can affected by sedation, difficult to interpret behaviors & fluctuations in MS, not immediately available for early prediction of outcome, no commonly agreed upon classification scheme, often not available to later-treating physicians

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9
Q

Posttraumatic amnesia

A

Phase of recovery from TBI during which the patient is responsive, but acutely confused, disoriented, & unable to form & retain new memories

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10
Q

PTA is predictive of

A

NP outcome, independent living states, return to work

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11
Q

Using duration of unconsciousness, what is a mild, mod, sev TBI?

A

Mild: 24 hrs

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12
Q

Using GCS scores, what is a mild/mod/sev TBI?

A

Mild: 13-15
Mod: 9-12
Sev: 3-8

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13
Q

Using duration of PTA, what is a mild/mod/sev TBI?

A

Mild: 7 days

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14
Q

Areas of the brain that are most vulnerable to diffuse axonal injury

A

Frontal & temporal lobes, BG, perinventricular zones, CC, brainstem fiber tracts, superior cerebellar peduncles

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15
Q

Glasgow Outcome Scale

A

Death
Vegetative state
Severe disability (requires assistance to meet basic needs)
Moderate disability (unable to return to non-sheltered work or resume other major roles)
Good recovery

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16
Q

Factors predictive of functional outcome after TBI

A

Pre-injury employment status, demographic variables, injury severity, CC atrophy, physical impairments, early cognitive status, post-injury depression, impaired self-awareness, early fx status, family support, post-acute brain injury rehab

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17
Q

Risk factors for substance abuse following TBI

A

pre-injury history, onset of depression since injury, better physical functioning, male gender, young age, uninsured, single

18
Q

Negative predictors of return-to-work following TBI

A

severity of injury, age >40, low education, greater physical or cognitive impairment, personality change, substance abuse

19
Q

Positive predictors of return-to-work following TBI

A

multidisciplinary team approach during acute rehab, socially inclusive work environment, environmental modifications, focus position on strengths of the individual

20
Q

ACRM mild TBI definition

A

A traumatically induced physiological disruption of brain fx: any LOC, any loss of memory for events before/after accident, any alteration in MS at time of accident, focal neuro deficits that may/may not be transient

Severity of injury does not exceed: LOC of 30 mins or less, initial GCS of 13-15 after 30 mins, PTA not >24 hrs

21
Q

Psychological causes of chronic postconcussion syndrome

A
Attributions/misattributions
Diagnosis threat
Expectation as etiology
"Good old days" bias
Iatrogenesis
Nocebo affect
22
Q

Tetany

A

Repetitive muscle stimulation by electricity that prolongs contact during electrocution

23
Q

Second Impact Syndrome

A

When a 2nd TBI is sustained before the physiological reactions to the prior injury have resolved; diffuse swelling within hours of even mild injury that is likely the result of compromised cerebral autoregulation

24
Q

What constitutes primary, secondary, tertiary, & quaternary damage secondary to blast injuries?

A

Primary - blast wave
Secondary - flying debris
Tertiary - blast winds throw individuals into other things
Quaternary - anything else, like burns, toxic inhalations, crush injuries

25
The younger the child at the time of TBI, the more likely to display
Brain swelling, hypoxic-ischemic insult, diffuse rather than focal injuries
26
Academic performance in older children following TBI
Academic decline in behavioral terms with increased risk of retention, special ed placement, etc. Achievement tests tend to be insensitive to their academic changes
27
Academic performance following TBI in younger children
More likely to demonstrate deficits on standardized achievement tests than older children
28
Academic performance following TBI in children is predicted by
Premorbid academic fx, postinjury NP fx, postinjury bx adjustment, family environment, age at injury (preschool injury assoc. w/ school failure & slower acquisition of academic skills)
29
Factors related to pediatric TBI outcome
Severity, pre- & post-injury family status, treatment received, premorbid fx
30
Social functioning following pediatric TBI
Rated as less socially competent & lonelier than peers, poor social outcomes persist over time, relationship b/t injury & social outcome moderated by poor family fx, lower SES, lack of family resoources Children w/ frontal lobe injury more likely to have problems
31
There is empirical support for what treatments following pediatric TBI?
Operant conditioning in decreasing aggressive behaviors, school-based social interventions, cognitive remediation for attention skills, involvement of family member as member of treatment team
32
Galveston Orientation & Amnesia Test (GOAT)
Test of orientation; starts at 100 & failing items of orientation result in subtractions
33
Contusions are seen most in what areas?
Orbitofrontal cortex, anterior temporal lobe, posterior portion of superior temporal gyrus & adjacent parietal opercular area
34
Diffuse axonal injury (DAI)
Breaking/shearing/stretching of myelinated axons due to acceleration/deceleration & rotational injuries
35
Excitatory amino acids that are produced after DAI have an affinity for receptors that are especially prevalent in the _____ & ______.
Hippocampus & thalamus
36
Increase in risk in dementia with aging after history of severe TBI
4-5x higher
37
Risk of 2nd TBI following 1st
8x
38
Low velocity GSW injuries create contusions at
Entry & contrecoup points
39
High velocity GSW injuries are more likely to create
Diffuse injury secondary to shock waves & pressure effects
40
Pathway of electrical current most likely to result in CNS disturbance
Head to foot
41
Most damage caused by primary blast wave injuries occurs in
Organs that contain air (ears), fluid (stomach/intestines), tissue/air, tissue/fluid interface