Unit 2 - Small Ruminant Johne's to end Flashcards

1
Q

What clinical signs are associated with small ruminant Johne’s disease?

A

Wasting, loss of body fat, and about 20% have diarrhea

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2
Q

How young can small ruminants be to be affected by Johne’s disease?

A

1 year of age

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3
Q

What causes Johne’s in small ruminants?

A

M. avium subsp. paratuberculosis

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4
Q

How is Johne’s diagnosed?

A
Clinical signs
DTH skin test
Serology
Histopath
Culture
PCR
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5
Q

What differentials should be considered with chronic weight loss?

A

Chronic parasitism, OPP, starvation, caseous lymphadenitis, bad teeth, and plastic disease

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6
Q

How is Johne’s prevented?

A

Extensive culling and segregation of replacement ewes

Total de-pop and re-pop

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7
Q

In what population of small ruminants does hemorrhagic enteritis cause high mortality rates?

A

Neonatal lambs

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8
Q

What usually causes hemorrhagic enteritis?

A

Clostridium perfringens type C

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9
Q

What toxin causes hemorrhagic enteritis?

A

Beta toxin

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10
Q

What does beta toxin do?

A

Causes severe damage to the intestinal epithelium and hemorrhagic necrosis

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11
Q

How is hemorrhagic enteritis prevented?

A

Immunization and sanitation of the lambing barn

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12
Q

What is the etiologic agent of Yellow Lamb Disease?

A

C. perfringens type A

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13
Q

What lambs are more commonly affected by yellow lamb disease?

A

Best doing lambs of 2-6 months of age usually due to overeating

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14
Q

What lesions does yellow lamb disease cause?

A

Massive intravascular hemolysis

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15
Q

What clinical signs does yellow lamb disease cause?

A

Anemia, icterus, hemoglobinuria, and jaundice

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16
Q

When is yellow lamb disease immunization recommended?

A

In endemic areas

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17
Q

What differentials should be considered with yellow lamb diseasE?

A

Leptospirosis, babesiosis, and copper poisoning

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18
Q

What is the main intestinal parasite that effects small ruminants?

A

Haemonchus contortus

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19
Q

What clinical syndrome does Haemonchus contortus cause?

A

Severe anemia

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20
Q

What lesion does Haemonchus contortus cause?

A

Bottle jaw

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21
Q

What are the ‘parasites’ of secondary importance?

A

Ostertagia circumcincta

Trichostrongylus axei, T. colubriformis, nematodirus

22
Q

What do the parasites of secondary importance cause?

A

Diarrhea, loose stools, and weight loss

23
Q

How are intestinal parasites prevented/controlled?

A

Pasture management vs. Dry lot management
Correct dosing of anthelmintics
Over-reliance on anthelmintics
Don’t switch anthelmintics often

24
Q

Is a low or high number on the FAMACHA test indicative of anemia?

A

High number

25
What are the benefits to using the FAMACHA system?
Can be rapidly applied and is easier than FEC High correlation between anemia score and severity of disease Allows for good records and identification of genetically resistant animals
26
What is refugia?
Maintaining a low population of anthelmintic-sensitive parasites
27
What cautions need to be taken with the FAMACHA system?
Must be done correctly or will lead to problems Only addresses Haemonchus Must be used in combo with other parasite surveillance Must use the card Other conditions can affect the color of the 3rd eyelid
28
What lambs and kids get coccidiosis?
Lambs and kids usually over 3 weeks of age
29
T/F: Coccidiosis is only a problem in lambs and kids.
False - adult dairy goats get it too
30
What are the predisposing factors for coccidiosis?
Standing water, muddy lots, and stress
31
What clinical signs are associated with small ruminant coccidiosis?
Weak, down, or dead lambs or kids | Scouring, black feces, depression, and anorexia
32
How is Coccidiosis prevented?
Lambing and nursing areas should be dry and well bedded Prevent overcrowding Coccidiostats Rumensin
33
How is Coccidiosis treated?
Neonates - Amprolium or Trimethoprim-sulfa | Feeder lambs - Amprolium + Sulfamethazine
34
What causes black disease in small ruminants?
C. novyi type B and Fasciola hepatica
35
When should small ruminants be vaccinated against black disease?
Prior to placing on pasure in spring or summer
36
How is black disease treated?
There is no effective treatment
37
What causes neonatal colibacillosis?
F5 E. coli
38
What is neonatal colibacillosis also known as?
Watery mouth or rattle-belly
39
In what conditions is neonatal colibacillosis more common in?
Crowded, dirty conditions
40
How is colibacillosis prevented?
``` Shearing before lambing Crutching of ewes Make sure lambs get sufficient colostrum Sanitation Immunization ```
41
How is colibacillosis treated?
Rehydration Keep lambs warm Antibiotics
42
What causes club lamb fungus?
Trichophyton verrucosum
43
What predisposes lambs to club lamb fungus?
Lots of washing, shearing, and grooming - happens in sow lambs Infected equipment and facilities
44
T/F: Club lamb fungus is easily transmitted and lots of treatments are needed
True
45
What predisposes small ruminants to dermatophilosis?
Moisture and minor trauma
46
Where can dermatophilosis localize?
Back, legs, ears, etc.
47
How is dermatophilosis treated?
penicillin
48
T/F: Peste des Petit ruminant is rapidly transmitted and not a problem in the US.
True
49
What clinical signs are associated with Peste des Petit?
High fever Mucopurulent ocular and nasal discharges High mortality
50
What lesions are associated with Peste des Petit?
Extensive erosions on oral and pharyngeal mucosa | Enteric lesions - zebra stripes
51
What vaccinations should ewes get?
``` C. jejuni and C. fetus fetus CDT Chlamydia abortus Soremouth Caseous lymphadenitis E. coli F5 bacterin Bluetongue OR no vaccination for a closed herd ```
52
What vaccinations should lambs get?
2x CDT 4 and 6 weeks of age Bluetongue in endemic areas Perfringens type A in endemic areas +/- Haemonchus contortus vax