Lecture 27 Flashcards

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1
Q

what occurs with acquired amusia?

A

there is high degree of heterogeneity

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2
Q

where the damage located in the brain in acquired amusia?

A

great severity in right hemisphere

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3
Q

Sihvonen tested stroke patients damage to the brain…what parts of the brain were damaged?

A
  • right STG
  • MTG
  • insula
  • putamen
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4
Q

what occurred to the gray and white matter in the Sihvonen that studied stroke patients?

A
  • gray matter decreased in right STG and right MTG
  • white matter decreases in right MTG
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5
Q

gray matter decreasing in the right temporal area causes what kind of damage?

A
  • posterior damage = pitch-based deficits
  • anterior = beat finding disorder
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6
Q

was gray matter volume changes in one area of the brain?

A

no -> widespread areas of the right cortex

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7
Q

where are white matter abnormalities found?

A

various regions -> corpus callosum too

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8
Q

acquired amusia shows damage in what areas?

A
  • damage extended beyond auditory regions and higher-level cognition
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9
Q

along with amusia what other impairment appears?

A

language
- worse performance on verbal expression/comprehension tests

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10
Q

acquired amusia is associated with deficits related to _________ ________

A

visuospatial processing

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11
Q

t or f: deficits are found in other domains other than visuospatial processing

A

true
- working memory
- verbal learning
- attention
- executive functioning

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12
Q

what do amusics not seek out?

A

do not seek out music for recreational purposes

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13
Q

what is musical anhedonia?

A

lack of pleasure derived from listening to music

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14
Q

is there an association or dissociation between the ability to perceive music and the ability to experience pleasure in response to music?

A

dissociation

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15
Q

what can musical anhedonia co-occur with?

A

depression

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16
Q

what kind of problem occur did case study IR acquire?

A
  • cerebral aneurysm
17
Q

IR lost the ability to perceive slight variations in pitch but continued to listen and dance, what did this indicate?

A

apparent dissociation between pitch perception and potential reward related to musical experiences
- social setting/shared activities = rewarding

18
Q

Hirel et al. study about the stroke patient having acquired amusia showed what results?

A
  • able to perceive emotional content in diff domains
  • worse than controls at identifying the intensity of emotion within musical pieces
  • loss of emotional engagement and interest with music
    -> proves the disconnection b/w perception and experience of emotion
19
Q

what area of the brain show reduced levels of activity in musical anhedonia?

A

nucleus accumbens

20
Q

what does the nucleus accumbens do and what task is associated with it?

A
  • reward
  • gambling task -> more investment if you know you are going to walk away with earnings
21
Q

where does functional connectivity decrease in people with musical anhedonia?

A

right auditory cortex and ventral stream

22
Q

what happens to connectivity between the right auditory cortex and ventral stream in people with above avg responses to music?

A

connectivity is enhanced

23
Q

the connection between the auditory areas and subcortical structures is associated with what kind of activity?

A

reward-network-relevant activity in response to musical activity

24
Q

what is amusia an example of?

A

perception without awareness

25
Q

the abnormalities found in the brain for amusia are found in what areas?

A

recurrent/re-entrant/feedback processing between sensory modules and higher order frontal areas

26
Q

abnormalities in recurrent/re-entrant/feedback processing can explain what other disorders?

A

developmental dyslexia
congenital prosopagnosia

27
Q

what does developmental dyslexia affect?

A

accuracy and fluidity of reading

28
Q

phonetic representations are processed in what area of the brain?

A

left STG
- connectivity between STG and IFG -> prevents conscious access to representations

29
Q

what is congenital prosopagnosia?

A

deficit related to facial recognition
- right FFA relatively normal responses but there is a reduction in white matter connecting FFA to frontal/temporal regions

30
Q

what was the commonality did Royal, Paquette and Tranchant find b/w dyslexia, prosopagnosia and amusia?

A

breakdown in communication pathways b/w core sensory area and higher-order regions that are required for the conscious detection of subtle perceptual differences

31
Q

what did Vuvan et al. find about deficits in amusia patients?

A

amusia patients have deficits more in domain-general acoustic deficit applying to both musical and non-musical representations
- double dissociation
- amusia may be more heterogenous

32
Q

Costanzo et al. used what to improve reading behavior in dyslexia patients?

A

tDCS

33
Q

what did Schaal et al use to improve pitch memory in amusics?

A

tACS