thalamus, limbics, and cerebellum Flashcards Preview

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Flashcards in thalamus, limbics, and cerebellum Deck (12):
1

VPL of thalamus: input, info, and destination

input: spinothalamic and dorsal columns/medial lemniscus.
info: pain and temp, pressure, touch, vibration, and proprioception
goes to the primary somatosensory cortex

2

VPM of thalamus: input, info, and destination

input: tigeminal and gustatory pathways
info: face sensation and taste
goes to the primary somatosensory cortex

3

LGN: input, info and destination

input: CN II. input: vision
destination: calcarine sulcus. lateral = light

4

MGN: input, info, and destination

input: superior olive and inferior colliculus of the tectum
gives info about hearing
goes to the auditory cortex of the temporal lobe
(medial = music)

5

nucleus VL of the thalamus: input, info, and destination

input: basal ganglia, cerebellum
gives motor info
goes to the motor cortex

6

What are the structures of the limbic system?

hippocampus, amygdala, fornix, mammillary bodies, and cingulate gyrus.

7

What does the limbic system do?

feeding, fleeing, fighting, feeling, sex

8

What is the input to the cerebellum?

contralateral cortex via the middle cerebellar peduncle
ipsilateral proprioceptive info via inferior cerebellar peduncle from the spinal cord. these are climbing and mossy fibers

9

What is the output of the cerebellum?

sends info to the contralateral cortex to modulate movement.
output nerves: purkinje cells to the deep nuclei of the cerebelum to the contralateral cortex via the superior cerebellar peducnle.`

10

What are the deep nuclei of the cerebellum?

dentate, emboliform, globose, fastigial ( lateral to medial)

11

Manifestations of lateral lesions to the cerebellum? What side will a patient fall to?

affects the voluntary movements of the extremities. When the cerebellum is injured, the patient is likely to fall toward the injured side

12

Manifestations of medial lesions to the cerebellum

vermis and fastigial nuclei lesions cause truncal ataxia
floculonodular lobe causes nystagmus, head tilting
patients may have a wide-based gait and deficits in truncal coordination. midline lesions result in bilateral motor deficits of the axial and proximal limb musculature.

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