Traumatic head and spinal injury Flashcards

(45 cards)

1
Q

Which structures may the direct effects of trauma be seen in? Which types of trauma affect each structure?

A

Scalp - lacerations
Skull - fractures
Meninges - vascular injury, lacerations
Brain/spinal cord - contusions, lacerations, diffuse axonal injury, diffuse vascular injury

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is concussion?

A

A clinical term to describe instantaneous loss of consciousness, temporary respiratory arrest and loss of reflexes.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What causes concussion?

A
Sudden change in the momentum of the head
Pathology unknown (maybe RAS)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

How is a head injury assessed clinically?

A

Glasgow coma scale
13 or above: mild brain injury
9-12: moderate brain injury
8 or less: severe brain injury

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Why is the bony casing around the brain and spinal cord both a blessing and a curse?

A

Blessing: high energy transfer required to breach layer
Curse: Soft tissue against hard bone can cause problems

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

How is damage caused in penetrating injuries and in closed injuries?

A

Penetrating: direct disruption of tissue
Closed: movement and compression of neural and vascular structures within bony casing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What are some of the secondary effects of traumatic head injury?

A
Ischaemia
Hypoxia
Cerebral swelling
Infection
Epilepsy
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

How do skull fractures generally behave?

A

Radiate from point of impact

May be depressed

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What are the different classes of skull fractures?

A

Open: communicate with surface
Closed: do not communicate with surface
Comminuted: splintering of bone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What does blood or CSF coming from the nose and/or ears indicate?

A

Basal skull fractures

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Why is it important to diagnose skull fractures?

A

They are an indicator of high energy transfer energy

Therefore, potential for direct brain sequelae is high

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Why are extradural haematomas less common in elderly people?

A

Dura becomes more adherent to the skull with age, so that blood cannot track through it

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Why are subdural haematomas more common in elderly people?

A

Brain shrinks > veins stretched> more susceptible to rupture

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Why can subdural haematomas be acute or chronic?

A

Can be self-limiting, as it is very low pressure blood

Can continue to accumulate unnoticed until it causes raised ICP

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What are contusions?

A

Haemorrhagic necrosis or bruising of the brain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What are coup injuries?

A

Those that occur at the site of impact

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What are contrecoup injuries?

A

Injuries that occur on the opposite side of the brain when the head is not immobilised at the time of injury

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Why do contusions often occur at the base of the brain?

A

Many irregularities in the cranial floor which the brain may rub against

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

What are some common sites for contusions at the base of the brain?

A

Inferior frontal lobes

Inferolateral temporal lobes

20
Q

Describe the appearance of old cerebral contusions?

A

Abnormal gyri
Collapsed brain tissue
Yellowish

21
Q

Why do old cerebral contusions appear yellowish?

A

Macrophages have removed necrotic tissue and blood

Leave behind haemosidirin

22
Q

What causes a traumatic cerebral haematoma?

A

Intra-cerebral haemorrhage when a large blood vessel is ruptured

23
Q

Define a laceration?

A

Penetration by foreign body or skull fragments

24
Q

Describe the damage caused by missile injuries?

A

Brain at either side of bullet path has shockwave effect

So, actual injury to brain can be much greater in diameter than the bullet itself

25
What is required for cerebral tissue to tear?
Severe enough impact with sufficient energy transfer
26
What is the most vulnerable site for cerebral tissue tearing?
Ponto-medullary junction
27
Which part of the brain is most susceptible to diffuse axonal injury?
Corpus callosum
28
Describe the microscopic appearance of diffuse axonal injury lesions in the corpus callosum?
Silver stain > axonal spheroids (area of swelling in axon, marker of transection)
29
Describe the macroscopic appearance of diffuse vascular injury?
Spotty haemorrhages
30
Describe the longterm effects of diffuse axonal injury?
Brain atrophy Enlarged ventricles Thin corpus callosum Thin white matter
31
Describe the toothpaste effect in cord-compressive acute traumatic injuries?
Spinal cord squashed by bone and cartilage > cord tissue squeezed both proximally and distally
32
List some of the longer term sequelae after brain trauma?
Infections Hydrocephalus Epilepsy Chronic traumatic encephalopathy
33
What is hydrocephalus?
Ventricles become dilated and brain tissue is compressed as a consequence
34
Why may hydrocephalus occur in the long term after brain trauma?
Exit for CSF may be blocked off by scar tissue
35
What are the three major components of the cranium?
Brain tissue CSF Blood
36
How much blood and CSF usually exists in the cranium
150mL each
37
How does the brain initially respond to an expanding brain lesion?
Expulsion of as much venous blood and CSF as possible
38
Where can herniations of brain tissue occur due to raised ICP?
Through dural openings
39
What happens as ICP approaches arterial pressure?
Brain perfusion ceases | CPP=MAP-ICP
40
What is a major structural sequelae of raised ICP?
Herniation of brain tissue
41
What are the causes of raised ICP?
``` Trauma Tumor Infarction Haemorrhage Infection Cerebral oedema Increased CSF ```
42
What are the two main types of cerebral oedema?
Vasogenic | Cytotoxic
43
Describe vasogenic cerebral oedema?
Due to BBB disruption with increased vascular permeability Predominantly involves white matter Responds to normal treatment
44
Describe cytotoxic oedema?
Increased intracellular fluid to cell membrane injury Involves grey and white matter Non-steroid responsive
45
Which three brain areas are prone to herniation with raised ICP?
Medial temporal lobe Cingulate gyrus Cerebellar tonsil