Ch. 36 GI - Book/6 Flashcards

(36 cards)

1
Q

hypovolemia, hypotension, and shock are sx of

A

acute pancreatitis

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2
Q

pancreatitis develops because of

A

obstruction to the outflow of pancreatic digestive enzymes caused by bile and pancreatic duct obstruction

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3
Q

goal of tx for acute pancreatitis is to

2

A

stop process of autodigestion and prevent systemic complications

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4
Q

pancreatitis that is usually mild and resolves spontaneously

A

acute

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5
Q

epigastric or midabdominal constant pain that radiates to back

A

pancreatitis

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6
Q

common age for pancreatitis

A

50-60

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7
Q

chronic pancreatitis pain is associated with

5

A
  1. increased intraductal pressure
  2. ischemia
  3. neuritis
  4. intra-abdominal HTN
  5. ongoing injury
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8
Q

abdominal distention with bowel hypomotility is to

A

acute pancreatitis

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9
Q

risk factor for pancreatic cancer

A

chronic pancreatitis

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10
Q

characteristics lab finding for acute pancreatitis

A

elevated amylase

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11
Q

elevated serum lipase levels is to

A

primary dx marker for acute pancreatitis

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12
Q

what increase the risk for chronic pancreatitis (besides alcohol)
3

A
  1. obstruction from gallstones
  2. smoking
  3. genes
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13
Q

pancreatitis pain is caused by

4

A
  1. edema
  2. chemical irritation and inflammation of peritoneum
  3. irritation or obstruction of the biliary tract
  4. inflammation of nerves
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14
Q

acute pancreatitis complications can include

6

A
  1. ARDS
  2. heart failure
  3. renal failure
  4. coagulopathies
  5. intra abdominal HTN
  6. systemic inflammatory response syndrome SIRS
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15
Q

chronic pancreatitis is a process of

A

progressive fibrotic destruction of the pancreas

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16
Q

pancreatitis risk factors

8

A
  1. obstructive biliary tract disease
  2. alcoholism
  3. obesity
  4. peptic ulcers
  5. trauma
  6. hyperlipidemia
  7. hypercalcemia
  8. smoking
17
Q

progressive fibrotic destruction of the pancreas

A

chronic pancreatitis

18
Q

chronic pancreatitis - what contribute to the destruction of acinar cells and islets of langerhans
2

A
  1. toxic metabolites

2. chronic release of inflammatory cytokines

19
Q

elevated amylase is to

A

acute pancreatitis

20
Q

pancreatitis - what accompany the inflammatory response

2

A

fever

leukocytosis

21
Q

chronic pancreatitis - may be required to relieve pain and prevent cystic rupture
2

A

endoscopic or surgical drainage of cysts or partial resection of pancreas

22
Q

develops because of obstruction to the outflow of pancreatic digestive enzymes caused by bile and pancreatic duct obstruction

23
Q

inflammation of the pancreas aka

24
Q

which pancreatitis can lead to DM

25
systemic effects of acute pancreatitis are related to
release of proinflammatory cytokines into the bloodstream
26
acute pancreatitis can result from direct cell injury from | 3
alcohol drugs virus
27
most common cause of chronic pancreatitis
chronic alcohol abuse
28
acute pancreatitis tx - to decrease pancreatic secretion and rest the gland, what may be done initially
stop oral intake
29
because of obstruction, what happens to the pancreas
activated enzymes cause autodigestion of pancreatic cells and tissues, resulting in inflammation
30
the cardinal manifestations of acute pancreatitis is
epigastric or midabdominal constant pain that radiates to back
31
primary dx marker for acute pancreatitis
elevated serum lipase levels
32
acute pancreatitis - what occurs because plasma volume is lost as inflammatory mediators release into the circulation and dilate vessels 5
1. hypovolemia 2. hypotension 3. tachycardia 4. myocardial insufficiency 5. shock
33
chronic pancreatitis is a risk factor for
pancreatic cancer
34
common sx of chronic pancreatitis | 2
1. continuous or intermittent abdominal pain | 2. weight loss
35
chronic pancreatitis - to correct enzyme deficiencies and prevent malabsorption, what is done
oral enzyme replacements are taken before and during meals
36
manifestations of what are present in late stages of chronic pancreatitis
pancreatic enzyme deficiency