38. Acquired and developmental disturbances of the teeth shape, position and morphology. Clinical manifestation, diagnosis, prevention, treatment Flashcards

(19 cards)

1
Q

Acquired and developmental disturbances of the teeth

Gemination

A
  • Aka schizodontia
  • Developmental anomaly=>single tooth bud divides, into tooth w/ bifid crown and common root
  • Primary and permanent dentitions affected
  • Prevalence in maxillary anterior region
  • Trauma suggested as possible cause, but exact etiology unknown
  • Tooth larger than normal w/ notched or clefted crown
  • Diagnosis=>
  • Radiographic Features:
  • Enlarged, notched crown
  • Two pulp chambers
  • Single root and pulp canal
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2
Q

Acquired and developmental disturbances of the teeth

Fusion (synodontia)

A
  • Aka synodontia
  • Two normally separated tooth buds unite, forming single large tooth
  • Can involve entire length of teeth or just roots, where dentine and cementum shared
  • Affected tooth larger than usual, and tooth count reveals missing tooth
  • Frequently in mandibular region and can be associated w/ conditions like thalidomide-induced embryopathy
  • Diagnosis=>
  • Radiographic Features:
  • Two pulp chambers
  • Two root canals
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3
Q

Acquired and developmental disturbances of the teeth

Taurodontism

A
  • Elongated crown and an apically displaced furcation=>
  • Pulp chambers w/ increased apical-occlusal height
  • May be seen as isolated incidents within families or associated w/ syndromes such as Down syndrome and Klinefelter’s syndrome
  • Diagnosis=>
  • Radiographic Features:
  • Elongated crowns
  • Apically displaced furcation
  • Treatment=>
  • Generally, no treatment required unless associated w/ other dental conditions
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4
Q

Acquired and developmental disturbances of the teeth

Dens evaginatus

A
  • Aka Talon’s cusp
  • Well-delineated additional cusp forms on surface of anterior tooth=>
  • Extends at least halfway from cementoenamel junction (CEJ) to incisal edge
  • Additional cusp may interfere w/ occlusion
  • Treatment=>
  • If occlusion affected, additional cusp removed
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5
Q

Acquired and developmental disturbances of the teeth

Dens invaginatus

A
  • Aka dens in dente
  • Deep surface invagination of crown or root that is lined w/ enamel
  • Types=>
  • Type I: Confined to crown
  • Type II: Extends below CEJ and ends in blind sac, which may or may not communicate w/ adjacent pulp
  • Type III: Extends through root and perforates apical area without immediate communication w/ pulp
  • Depth of invagination varies, ranging from slight enlargement of cingulum to deep infolding extending to apex
  • Treatment=>
  • Type I: Restoration of invagination to prevent caries
  • Type II: Temporary placement of calcium hydroxide (Ca(OH)₂) w/ endodontic treatment
  • Type III: Closure of openings before pulpal necrosis occurs
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6
Q

Acquired and developmental disturbances of the teeth

Hutchinson’s incisors and Mulberry molars

A
  • Dental anomalies associated w/ congenital syphilis
  • Hutchinson’s Incisors=>
  • Peg-shaped or screwdriver-shaped lateral incisors
  • Widely spaced, w/ notched biting surfaces and crescent-shaped deformities
  • Mulberry Molars=>
  • First permanent molar presents multiple rounded rudimentary enamel cusps=>
  • “Mulberry” appearance
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7
Q

Acquired and developmental disturbances of the teeth

Concrescence

A
  • Union of two teeth by cementum without confluence of dentin
  • Often in posterior maxillary region, especially involving second molar and adjacent impacted third molar
  • Classification=>
  • True Concrescence: Developmental origin
  • Acquired Concrescence: Occurs after tooth development
  • Diagnosis=>
  • Radiographic Features:
  • Cementum of two adjacent teeth appears fused
  • Often, no treatment required unless union interferes w/ tooth eruption=>
  • Surgical removal
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8
Q

Acquired and developmental disturbances of the teeth

Enamel pearls

A
  • Ectopic droplets of enamel that occasionally form on roots=>
  • Particularly in bifurcation or trifurcation areas, w/ maxillary molars more commonly
  • Minor abnormalities=>nodule of enamel attached to dentine, sometimes containing a pulp horn
  • Diagnosis=>
  • Visual and Radiographic Features:
  • Enamel pearls small, round radiopacities on radiographs
  • Treatment=>
  • Flattening or removal of enamel pearl if poses risk for plaque accumulation and periodontal disease
  • Good oral hygiene essential in affected area
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9
Q

Acquired and developmental disturbances of the teeth

Dilaceration

A
  • Abnormal angulation/sharp bend in root or crown
  • Etiology=>
  • Trauma to developing tooth common cause=>
  • Root forms at angle
  • In some cases, hereditary factors may be involved
  • Angulation may cause complications in tooth eruption or endodontic treatment
  • Diagnosis=>
  • Radiographic Features:
  • A pronounced curve or angulation in tooth root observed.
  • Treatment=>
  • Often not required unless dilaceration interferes w/ tooth function or alignment
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10
Q

Acquired and developmental disturbances of the teeth

Amelogenesis imperfecta and its different types

A
  • Group of conditions caused by defects in genes encoding enamel matrix proteins=>
  • Amelogenin and enamelin
  • Primary and permanent dentitions
  • Types=>
  • Hypoplasia
  • Hypomaturation
  • Hypocalcification
  • Treatment=>
  • Cosmetic management, such as improving appearance of teeth
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11
Q

Acquired and developmental disturbances of the teeth

Amelogenesis imperfecta-Hypomaturation

A
  • Enamel appears normal in form on eruption=>
  • Opaque, white to brown-yellow
  • Softer than normal
  • Radiographic Features=>
  • Radiodensity similar to dentine
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12
Q

Acquired and developmental disturbances of the teeth

Amelogenesis imperfecta- Hypoplasia

A
  • Inadequate formation of enamel matrix
  • Randomly pitted, grooved, thin, translucent enamel
  • Radiographic Features=>
  • Enamel appears reduced in bulk, w/ thin layer over occlusal and interproximal surfaces=>
  • Normal dentine and pulp chambers
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13
Q

Acquired and developmental disturbances of the teeth

Amelogenesis imperfecta-Hypocalcification

A
  • Enamel matrix formed in normal quantity but poorly calcified=>
  • Weak, chalky, easily worn enamel
  • Radiographic Features=>
  • Enamel and dentin w/ similar radiodensities
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14
Q

Acquired and developmental disturbances of the teeth

Dentinogenesis imperfecta

A
  • Aka hereditary opalescent dentin
  • Genetic disorder caused by mutations in dentine sialophosphoprotein (DSPP) gene
  • Primary and permanent dentitions
  • Abnormal dentine formation, leading to discolored and fragile teeth
  • Teeth exhibit blue-brown discoloration w/ distinctive opalescent translucency
  • Enamel tends to separate easily from underlying defective dentin, leading to rapid wear
  • Radiographic Features=>
  • Bulbous crowns
  • Cervical constriction
  • Thin roots
  • Early obliteration of root canals and pulp chambers
  • Type I-III
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15
Q

Acquired and developmental disturbances of the teeth

Dentinogenesis imperfecta classification

A
  • Classification
  • Type I=>
  • In association with osteogenesis imperfecta
  • Primary teeth more severely affected
  • Radiographically, partial or total obliteration of pulp chambers and root canals, roots may be short and blunted
  • Type II=>
  • Does not occur in association w/ osteogenesis imperfecta
  • Teeth present only dentin abnormalities without bone disease
  • Radiographically similar to Type I
  • Type III=>
  • “Brandywine type,” primarily seen in racial isolate in Maryland
  • Multiple pulp exposures common in deciduous teeth=>
  • Not seen in Types I and II
  • Radiographically, large pulp chambers, and enamel appears normal
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16
Q

Acquired and developmental disturbances of the teeth

Dentinogenesis imperfecta Treatment

A
  • Preventing loss of enamel and subsequent dentin through attrition crucial
  • For posterior teeth, cast metal crowns recommended
  • For anterior teeth, jacket crowns placed to improve aesthetics and protect underlying dentin
17
Q

Acquired and developmental disturbances of the teeth

Dentin dysplasia

A
  • Rare genetic disorder aka “rootless teeth,”
  • Abnormal dentin formation, leading to malformed roots and pulp morphology
  • Type I (Radicular Type)=>
  • Primary and permanent dentitions normal in color
  • Premature tooth loss due to short roots or periapical inflammatory lesions
  • Radiographic Features=>
  • Extremely short roots
  • Almost complete obliteration of pulp chambers
  • Periapical radiolucencies, including granulomas, cysts, and chronic abscesses
  • Type II (Coronal Type)=>
  • Primary dentition may appear opalescent, while permanent dentition typically normal color
  • Radiographic Features:
  • Deciduous Teeth: Extremely short roots and almost complete obliteration of pulp chambers
  • Permanent Teeth: Abnormally large pulp chambers in coronal portion of tooth=>
  • Often filled w/ globules of abnormal dentin
  • Treatment=>
  • Preventing complications related to short roots and ensuring integrity of the teeth, especially in Type I
18
Q

Acquired and developmental disturbances of the teeth

Regional odontodysplasia

A
  • Aka “ghost teeth”
  • Affects structure and formation of enamel and dentin in one or several teeth
  • Usually localized to specific region of mouth
  • Affected teeth may exhibit delayed eruption or fail to erupt
  • Shape of teeth altered=>
  • Irregular and malformed
  • Radiographic Features:
  • Marked reduction in radiodensity, “ghost-like” appearance
  • Enamel and dentin very thin, pulp chambers large
  • Treatment=>
  • Due to poor cosmetic appearance and structural integrity of affected teeth, extraction often=>
  • Restoration using prosthetic appliances
19
Q

Acquired and developmental disturbances of the teeth

Shell tooth

A
  • Normal enamel thickness but extremely thin dentin=>
  • Enlarged pulp chambers
  • Entire tooth or isolated to root
  • Frequently in deciduous teeth
  • Tooth appears normal size, but radiographs reveal an enlarged pulp chamber with very thin dentin surrounding it
  • Treatment=>
  • Protecting thin dentine from damage
  • Careful dental care and sometimes restorative procedures to prevent fractures