12. Diseases of the oesophagus in dogs and cats. Regurgitation and vomitus. Flashcards

(62 cards)

1
Q

causes of oesophagitis

A

caustic substances
gastro- oesophageal reflux
motility disorders
megaoesophagus
doxycycline
clindamycin
anaesthesia
froeign body

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2
Q

clinical signs of oesophagitis

A

odynophagia (pain when swallowing)
regurgitation
salivation
food refusal
coughing
fever
dyspnoea

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3
Q

diagnosis of oesophagitis

A

increased WBC
endoscopy
xray

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4
Q

treatment of oesophagitis

A

omeprazole + prokinetics
sucralfate suspension
total parenteral nutrition

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5
Q

gastro oesophagwal reflux (GOR)
definition

A

poor closure of the cardia
inflammation due to HCL, pepsin, trypsin, bile, HCO3

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6
Q

causes of gastro oesophagwal reflux (GOR)

A

delayed gastric emptying
upper airway obstruction
hiatal hernia
anaesthesia
chronic vomiting

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7
Q

clinical signs of gastro oesophagwal reflux (GOR)

A

reverse sneezing
chronic bronchitis
laryngitis
rhinitis
erucation

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8
Q

diagnosis of gastro oesophagwal reflux (GOR)

A

clinical signs - history
endoscopy

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9
Q

treatment of gastro oesophagwal reflux (GOR)

A

low fat diet
H2 receptor blockers
PPI
prokinetics
sucralfate

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10
Q

what durgs enhance the closure of the cardia

A

metoclopramide
erythromycin

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11
Q

clinical sings of oesophageal foreign body

A

regurgitation
salivation
abdominal distension
coughing, fever, dyspnoea

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12
Q

diagnosis of gastro oesophagwal reflux (GOR)

A

clinical signs
thoracic xray
endoscopy
increased WBC

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13
Q

treatment of oesophageal foreign body

A

endoscopic removal
surgery
gastric tubing
euthansia

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14
Q

types of dysmotility/ oesophageal weakness

A

megaoesophagus
partial oesophageal dysmotility
dystonia
diverticulum

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15
Q

causes of dysmotility/ oesophageal weakness

A

idiopathic
muscle atrophy
myasthenia gravis
Autoimmune
post anaesthesia condition
oesophagitis

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16
Q

clinical signs of dysmotility/ oesophageal weakness

A

salivation
cough (bronchopneumonia)
fever
dyspnoea
weight loss

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17
Q

diagnosis of dysmotility/ oesophageal weakness

A

fluroscopy, radiography

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18
Q

treatment of dysmotility/ oesophageal weakness

A

special feeding
PEG (percutaneous endoscopic gastrotomy)

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19
Q

Megaoeasophagus definition

A

persisten decrease of motility of the oesophagus

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20
Q

what animal is Megaoeasophagus more common in

A

dogs > cats

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21
Q

cause of Megaoeasophagus

A

normally idiopathic
may be caused by laryngeal paralysis
NB to check for aspiration pneumonia

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22
Q

congenital Megaoeasophagus

A

mini schnauzer, great dane, dalmatian
shar pei, irish setter, lb

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23
Q

clinical signs of congenital Megaoeasophagus

A

usually develops during the weaning phase
no treatment
poor porgnosis

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24
Q

acquired Megaoeasophagus
Primary form

A

diagnosis by exclusion
large > toy breeds
treat using Bailey chair; sucralfate + bethanechol

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25
acquired Megaoeasophagus secondary form causes
myasthenia gravis Addison's lead poisoning oesophagitis hypothyroidism neuropathy snake bit
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what is myasthenia gravis
immune response against Ach receptors Focal/ generalised treat with pyridostigmine
27
Spirocercosis cause
spirocercosis lupi - nematode causes parasitic nodules in the oesophagus, aortic aneurysms & spondylitis
28
clinical signs of Spirocercosis
regurgitation vomiting weight loss coughing dyspnoea sudden death
29
diagnosis of Spirocercosis
faecal examination endoscopy
30
treatment of Spirocercosis
imidacloprid / moxidectin surgery
31
prevention of Spirocercosis
milbemycin
32
persistent right aortic arch definition
extraluminal compression of the oesophagus by the lig arteriosum a vascular ring anomaly
33
cause of persistent right aortic arch
congenital g. shepherds
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clinical signs of persistent right aortic arch
regurgitation coughing dyspnoea weight loss
35
diagnosis of persistent right aortic arch
survey & contrast radiography endoscopy
36
differential of persistent right aortic arch
stricture intraluminal obstructiontr
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treatment of persistent right aortic arch
surgery
38
Regurgitation vs Vomiting comes from
Regurgitation - from mouth / pharynx / oesophagus Vomiting - from stomach / small intestine
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Regurgitation vs Vomiting activity
Regurgitation - passive, no nausea Vomiting - active, wretching, heaving
40
Regurgitation vs Vomiting content
Regurgitation - undigested food, no bile (eating content) Vomiting - un / digested food, chyme, bile (no eating of content)
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expectoration
expulsion of material from the respiratory tract
42
how to distinguish Regurgitation and Vomiting
history clinical exam palpation of distended oesophagus th. xray endoscopy fluroscopy
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causes of vomiting
metabolic diseases endocrine diseases toxins / drugs dietary causes abdominal diseases gastric diseases small intestine diseases large intestine diseases
44
metabolic diseases causing vomiting
renal disease hepatobiliary disease electrolyte / acid base derangements
45
endocrine diseases causing vomiting
hypoadrenocorticism hyperthyroidism diabetic ketoacidosis
46
toxins / drugs causing vomiting
heavy metals ethylene glycol NSAIDS, AB, chemotherapy
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dietary causes causing vomiting
allergy intolerance
48
abdominal diseases causing vomiting
pancreatitis peritonitis neoplasia
49
gastric diseases causing vomiting
gastritis GDV foreign body delayed gastric emptying neoplasia
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small intestine diseases causing vomiting
IBD obstruction parasites neoplasia infection
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large intestine diseases causing vomiting
constipation colitis
52
history of vomiting patient
duration food in vomit > 8hrs after ingestion acute / chronic
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physical exam of vomiting patient
oral exam mucosa cardiac arrhythmia abdominal palpation rectal examination
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lab d of patient with vomiting
CBC neutrophilic leucocytosis biochemical tests acid base status hyperkalaemia anaemia neutropenia eosinophilia hypoproteinaemia
55
what to see in xray of vomiting patient
foreign body distention displacement delayed gastric emptying penetrating ulcers masses deep ulceration pyloric obstruction
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symptomatic treatment of vomiting
antiemetics fluid therapy dietary antacids protectives
57
antiemetics
maropitant phenothiazines metoclopramide ondansetron
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fluid therapy
0.9% saline K supplementation
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dietary treatment for vomiting
NPO for 24hrs low fat, single protein source, chcicken and rice
60
antacids
H2 receptor antagonists - ranitidine PPI - omeprazole
61
protectives
sucralfate - provide barrier to acid penetration. inactivates pepsin, absorbs bile acids, stimulated PG synthesis Misoprostol - contraindicated in pregnant animals
62