peds82 Flashcards

1
Q

reactive lymphadenitis

A

occurs in response to infections in the pharynx, teeth, and soft tissues of the head and neck

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2
Q

viral infections that can cause cervical lymphadenitis

A

EBV, CMV, and HIV, among others

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3
Q

kawasaki disease and cervical lymphadenitis

A

may present with cervical lymphadenitis (in fact, enlarged cervical lymph node is one of the diagnostic criteria)

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4
Q

toxoplasma gondii and cervical lymphadenitis

A

can cause a mono-like disease with cervical lymphadenopathy

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5
Q

how can structural lesions in the neck cause a cervical lymphadenitis

A

can become infected and drain to the lymph nodes in the neck

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6
Q

what do the lymph nodes in cervical lymphadenitis look like?

A

mobile, tender, warm, and enlarged, and the overlying skin is erythematous. Fluctuance may be present

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7
Q

management of cervical lymphadenitis

A

empiric antibiotics directed at strep and staph; initial treatment may include a first gen cephalosporin or an anti-steph penicillin for 7-10 days

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8
Q

parotitis

A

inflamm of the parotid salivary glands

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9
Q

etiology of parotitis

A

mumps and other viruses; bacterial parotitis

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10
Q

difference in presentation between bacterial and viral parotitis

A

viral has bilateral involvement; bacterial is unilateral

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11
Q

causes of bacterial parotitis

A

aka acute suppurative parotitis; caused by staph aureus, strep pyogenes, and M tuberculosis

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12
Q

clincial features of parotitis

A

swelling centered above the angle of the jaw and fever; exam of the pharynx may reveal pus expressed from stenson’s duct

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13
Q

most common cause of viral parotitis

A

mumps (before vaccination)

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14
Q

complications of acute suppurative parotitis

A

formation of an abscess anad osteomyelitis of the jaw

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15
Q

impetigo

A

superficial skin infection involving the upper dermis

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16
Q

causative organisms in impetigo

A

stap aureus and GABHS or strep pyogenes

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17
Q

clinical features of impetigo

A

honey colored crusted or bullous lesions are present, commonly on the face, esp around the nares; fever usually absent; infection is easily transmitted

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18
Q

management of impetigo

A

treatment may include topical mupirocin or oral antibiotics like dicloxacillin, cephalexin, or clindamycin

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19
Q

complications of impetigo

A

bacteremia, post-strep glomerulonephritis (tx of impetigo does not prevent this), and staph scalded skin syndrome (SSSS)

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20
Q

erysipelas

A

skin infection that involves the dermal lymphatics

21
Q

causative organism in erysipelas

A

GABHS

22
Q

clinical features of erysipelas

A

tender, erythematous skin with a distinct border. Face and scalp are common locations

23
Q

management of erysipelas

A

systemic abx aganst GABHS

24
Q

complications of erysipelas

A

bacteremia, post-strep glomerulonephritis, and necrotizing fasciitis

25
Q

cellulitis

A

skin infection within the dermis

26
Q

causes of cellulitis

A

staph aureus and GABHS

27
Q

infected skin border in erysipelas vs cellulitis

A

in cellulitis border is indistinct

28
Q

management of cellulitis

A

oral or IV abx (first gen cef or anti staph penicillins)

29
Q

buccal cellulitis

A

unilateral bluish discoloration of the cheek of a young immunized child

30
Q

causative agent in buccal cellulitis

A

HIB

31
Q

what does a child with buccal cellulitis look like?

A

toxic, and blood cultures usually pos

32
Q

management of buccal cellulitis

A

IV antibiotics against H influenza, usually a second or third gen cephalosporin; perform LP

33
Q

perianal cellulitis

A

well-demarcated erythema involveing the skin around the anus

34
Q

how might kids with perianal cellulitis present

A

constipation

35
Q

cause of perianal cellulitis

A

GABHS

36
Q

managmenet of perianal cellulitis

A

oral antibiotics (cephalaxin, dicloxacillin)

37
Q

necrotizing fasciitis

A

potentially fatal form of deep cellulitis that extends into the muscle; pain and systemic findings out of proportion to physical findings;

38
Q

causative organisms in necrotizing fasciitis

A

polymicrobial; may involve GABHS and anaerobic bacteria

39
Q

therapy for necrotizing fasciitis

A

IV antibiotics and surgical debridement are essential components of therapy

40
Q

staph scalded skin syndrome

A

staph aureus produces a toxin that caues fever, tender skin, and bullae; large sheets of skin slough off; Nikolsky sign is present

41
Q

Nikolsky sign

A

extension of bullae when pressure is applied to the skin (skin pulls away w lateral pressure)

42
Q

management of SSSS

A

good wound care and IV antibiotics against Staph aureus

43
Q

scarlet fever

A

toxin-mediated bacterial illness that results in a characteristic skin rash

44
Q

cause of scarlet fever

A

GABHS that produces an erythrogenic toxin

45
Q

transmission of scarlet fever

A

large respiratory droplets or by infected nasal secretions

46
Q

clinical features of scarlet fever

A

exanthem tha develops during any GABHS infection (impetigo, cellulitis, pharyngitis); before or during exanthem, fever, chills, often exudative pharyngitis

47
Q

what does the exanthem of scarlet fever look/feel like?

A

begins on trunk and moves peripherally; skin is erythematous with tiny skin colored papules (“scarlitiniform”) and has texture of sandpaper; rash blanches; petechiae localized within skin creases in a linear distrib (Pastias lines)

48
Q

pastia’s lines

A

petechiae are often localized within skin creases in a linear distribution; seen in scarlet fever;

49
Q

desquamation of dry skin occurs as scarlet fever rash resolves

A

right; so do not worry if you see it