Spinal Neurosurgery Flashcards

(86 cards)

1
Q

A 37-year-old man, normally active and otherwise healthy, develops a sudden pain in his back and an ‘electric shock’ down his left leg. Over 5 weeks the back pain resolves but he is left with intermittent severe pain in the left leg which radiates down his calf into the sole of the foot and little toe. His right leg has been unaffected and he has normal sphincter function.

What is the likely diagnosis? Is there a differential diagnosis?

A

The history is classical for a prolapsed L5/S1 intervertebral disc and left S1 dermatomalsciatica (see ‘ Nerve roots affected by a prolapsed disc ’, p. 328).
Hip and knee pathology can both mimic sciatica, especially in older people. Many patients will have back pain, and there may be dual pathology. Recent exacerbation of back pain with sciatica suggests the two are linked. Rarely, acute leg pain can be a presentation of vascular events such as limb ischaemia. Calf pain may be a symptom of DVT which should be considered in a patient with severe immobility due to back pain

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2
Q

A 37-year-old man, normally active and otherwise healthy, develops a sudden pain in his back and an ‘electric shock’ down his left leg. Over 5 weeks the back pain resolves but he is left with intermittent severe pain in the left leg which radiates down his calf into the sole of the foot and little toe. His right leg has been unaffected and he has normal sphincter function.

The patient walks with a mildly antalgic gait. What other signs would you look for on examination?

A

Most common finding will be restriction of passive straight leg raise

May have senosry change in the left S1 dermatome and absent or diminished ankle jerk.

In more severe cases there is S1 numbness and weakness of ankle plantarflexion.

He has restricted straight leg raise at 40 degrees, despite mild senosry loss and weak ankle jerk, he is sensorily intact.

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3
Q

What is the diagnosis and how would you manage him at this stage?

A

There are two degenerate discs at L4/5 (A) and L5/S1 (B)

The L5/S1 bulges posteriorly a bit more than the L4/5 and the axial slices show a left posterolateral disc prolapse compressing the left S1 nerve root (C). The left S1 root is not visible separate from the disc.

Most disc prolapses settle with conesrvative management- rest and analgesia int he first instance.

If there is a neurological deficit there is a stronger case for early surgery, especially if foot drop is present.

The point at which surgery is considered depends on degree of pain and impact on QoL

6/52 typically is what is trialled for conservative Mx

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4
Q

37-year-old man, normally active and otherwise healthy, develops a sudden pain in his back and an ‘electric shock’ down his left leg. Over 5 weeks the back pain resolves but he is left with intermittent severe pain in the left leg which radiates down his calf into the sole of the foot and little toe. His right leg has been unaffected and he has normal sphincter function.

What surgery would you offer?

A

As leg pain is the main complaint- lumbar discectomy for the L5/S1 fragment.

Discectomy for leg pain with typical symptoms and clinical-radiological correlation has a high chance (>90% of improving his leg pain)

There may be secondary improvement in his back pain

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5
Q

Risks of lumbar discectomy

A

Haemorrhage

Infection

Infective discitis- results in progressive back pain and rarely, sepsis in the weeks following surgery which would require prolonged Abx

The risk of neurological complications e.g. cauda equina should be small.

Risk of S1 weakness should be mentioned as a rare occurence.

Early recurrence in the first few weeks following surgery can be treated with repeat surgery with good outcome

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6
Q

A 38-year-old woman presents to the emergency department with 2 weeks of back pain radiating down her left leg. On the previous day, she noticed difficulty passing urine and partial numbness of her buttocks.

What is the diagnosis?

A

Cauda equina until proven otherwise

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7
Q

Def: Cauda equina syndrome

A

Clinical syndrome resulting from the compression of the cauda equina.

Presents with a variable combination of:

leg pain (classically bilateral)

sacral anaesthesia

urinary retention with overflow incontinence.

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8
Q

Causes of cauda equina compression

A

Prolapsed lumbar intervertebral disc.

Tumours, abescess, trauma can also cause the syndrome

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9
Q

Important questions in cauda equina?

A

Characterise the exact nature of urinary symptoms as urinary retention can occur due to back pain.

Timing of onset of symptoms is also critical as it informs the timing of surgery.

Patient with incomplete cauda equina syndrome have better prognosis

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10
Q

A 38-year-old woman presents to the emergency department with 2 weeks of back pain radiating down her left leg. On the previous day, she noticed difficulty passing urine and partial numbness of her buttocks.

On examination there was some weakness of plantarflexion and
hip extension on the left. The left ankle reflex was absent.
Pinprick sensation was preserved throughout the lower limbs
but was absent in the perineum. However, when more pressure
was applied to the pin, the patient reported that she could feel
the sharp pin in the perineum. The rest of the examination
was normal.

Level of lesion

A

Left ankle is affected suggesting S1 involvement

Ankle plantar flexion and hip extension also have contributions from S1.

Level of compression is likely to be at L5/S1

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11
Q

A 38-year-old woman presents to the emergency department with 2 weeks of back pain radiating down her left leg. On the previous day, she noticed difficulty passing urine and partial numbness of her buttocks.

On examination there was some weakness of plantarflexion and
hip extension on the left. The left ankle reflex was absent.
Pinprick sensation was preserved throughout the lower limbs
but was absent in the perineum. However, when more pressure
was applied to the pin, the patient reported that she could feel
the sharp pin in the perineum. The rest of the examination
was normal.

Are the clinical findings consistent with the suspected diagnosis?

A

Examining perineal sensation can be challenging if the patient’s response is inconsistent. As a general rule, if perineal pinprick sensation is less than reported elsewhere when the same pressure is applied, it should be assumed that there is impairment. If a sharp sensation is elicited when the pin is pressed harder, impairment is not excluded. If a sharp sensation is not elicited at all, impairment is confirmed. Therefore these findings are consistent with cauda equina compression.

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12
Q

Ix in ?cauda equina

A

MRI lumbar spine

other options include CT myelogram or MRI under GA

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13
Q

?Dx

A

Large disc prolapse at L5/S1 filling the width of the canal (1).

Axial view shows the disc is laterally sited to the left. (2)

The thecal sac which appears white on the T2 image is displaced to the right.

There is a samlller left sided protrusion at the level above where the canal is more capacious,.

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14
Q

Mx of cauda equina

A

Discectomy involving mid-line posterior approch, laminectomy and removal of the prolapse segment of the disc.

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15
Q

A 38-year-old woman presents to the emergency department with 2 weeks of back pain radiating down her left leg. On the previous day, she noticed difficulty passing urine and partial numbness of her buttocks.

She undergoes surgery to decompress the cauda equina.

The patient is reviewed the following morning when
she complains of recurrent left leg pain. How should
her symptoms be managed?

A

DDx:

Residual disc

Post-operative haematoma

Infection

Post-surgical oedema.

Repeat MRI is required to exclude persistent neural compression

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16
Q

In the presence of what particular feature should you have a low threshold for emergency scanning in ?cauda equina?

A

Sensory changes in the sacral deramtomes, S2, 3 or 4 even if only subjective or unilateral.

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17
Q

Relationship between nerve root and intervertebral dsic

A

Situated just above the level of the numbered disc.

Unilateral lumar disc prolapse tends to ocompression the transversing nerve roots whereas a far lateral prolapse will compress the exiting nerve root at the levl.

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18
Q

A 43-year-old woman has a 1-year history of right arm pain felt around the elbow and radiating into the thumb and index finger. It has settled a little but she has a continu- ous tingling feeling in the same region. There is no history of neck pain or trauma.

Ddx and key features of Hx or exam

A

Neurological or MSK pain.

The tingling suggests the symptoms are more likely to be neurological in origin.

2 most likely are CTS which may radiate proximally beyond the wrist to the elbow or shoulder. And radiculopathy, classically C6 if the thumb and index finger are involved.

MSK causes such as lateral epicondylitis and tenosynovitis should be considered and can be excluded by point tenderness at the site of inflammation exacerbated by passive movement.

CTS usually repsonds to Tinel’s/Phalen’s test.

Cervical radicular pain can be reproduced by asking the patient to look up and turn their head to the contralateral side, to reduce the calibre of the affected nerve root foramen and reproduce symptoms

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19
Q

Spurling’s sign

A

Reproduction of radicular pain on looking up and turning head to contralateral side, which narrows the foramen.

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20
Q

A 43-year-old woman has a 1-year history of right arm pain felt around the elbow and radiating into the thumb and index finger. It has settled a little but she has a continu- ous tingling feeling in the same region. There is no history of neck pain or trauma.

On closer assessment she confirms that the thumb and index finger, rather than the thumb and the lateral two and a half fingers, are affected. Spurling’s sign is positive and you arrange an MRI. The T2 sagittal images of the midline and to the right of the midline and the axial image of the C5/6 disc are shown in Fig. 48.1. Comment on the findings.

A

There is loss of cervical lordosis, which is very common in patients with acute and chronic cevical spine pathology.

The discs apppear healthy on the midline image and there is no loss of disc height or osteophyte formation.

The saggital image to the right of the midline shows some loss of CSF signal adjacent to the disc at the C5/6 level.

The axial scans of C5/6 show the left C6 nerve root with the CSF surrounding it, but there is narrowing of the right C6 root due to lateral disc or osteophyte

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21
Q

A 43-year-old woman has a 1-year history of right arm pain felt around the elbow and radiating into the thumb and index finger. It has settled a little but she has a continu- ous tingling feeling in the same region. There is no history of neck pain or trauma.

MRI shows narrowing of the C6 nerve root and the surrounding CSF due to osteophyte or canal narrowing.

What are the options?

A

She does not have a progressive deficit and surgery should be offered if pain is intractable or intolerable.

A radiologically guided nerve root infiltration with steroids may offer temproary relief.

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22
Q

Surgical options for C6 nerve root compression

A

Options are anterior cervical discectomy (ACD) or foraminotomy (posterior approach to de-roof the right C6 neural foramen) as the aim is to decompress the C6 nerve root at the C5/6 neural foramen

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23
Q

What are the benefits of ACD

A

Will remove primary pathology (disc prolapse) and has a higher success rate in alleviating the brachalgia (95%)

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24
Q

What are the possible complications of ACD

A

Significant potential morbidity from complications such as RLN palsy, oesophageal, carotid injury and neck wound haematomas

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25
What is the rate of success in foraminotomy to decompress cervical nerve roots
80%
26
When is ACD preferred?
When there is a greater compressive component from anteriorly, where the nerve root may be bowstringed over the disc/osteophyte and therefore still remain under tension were performed. For instance in the MRI shown, there is a large C4/5 disc prolapse with probable compression of theC5 nerve root which would be inappropriate for foraminotomy as neural compression is very marked and exclusively from the front.
27
A 55-year-old woman has experienced left arm and neck pain for 18 months. Her usual pain radiates from the shoulder through the elbow into the thumb and index finger. However, a recent severe exacerbation involved the whole hand and lasted for 2 months. During that time she underwent an MRI and was referred for a surgical opinion. On examination she describes slight sensory change in her left index finger but has no deficit in terms of power or deep tendon reflexes. DDx
DDx of lateral forearm pain includes a number of common condiitons Cervical radiculopathy Lateral epidoncdylitis CT syndrome
28
Cervical radiculopathy
Nerve root irritation in the exiting foramen of the SC Usually due to disc bulge or facet joint arthropathy and osteophyte formation. It may be reproduced by asking the patient to look up and to the side of the pain (Spurling's sign)
29
Lateral epicondylitis
Inflamation of the comon extensor origin at the head of the radius. Pressure over this area during passive supination/pronation is painful
30
Carpal tunnel syndrome
Median nerve compression at the wirst, this typically involves rthe radial two and a half fingers of the thumb but may often have forearm pain and easily be confused with C6 radiculopathy. Forced flexion of the wrist in pronation may reproduce the symptoms as may tapping onthe volar surgace of the wrist in mild exentsion
31
A 55-year-old woman has experienced left arm and neck pain for 18 months. Her usual pain radiates from the shoulder through the elbow into the thumb and index finger. However, a recent severe exacerbation involved the whole hand and lasted for 2 months. During that time she underwent an MRI and was referred for a surgical opinion. On examination she describes slight sensory change in her left index finger but has no deficit in terms of power or deep tendon reflexes. The patient’s MRI is shown in Fig. 49.1 (sagittal T2, to the left of the midline, and axial T2 through the C5/6 and the C6/7 discs. What are the findings and how do they fit with the history?
There are two degenerate discs at C5/6 and C6/7 with posterior bulges on the sagittal images. On the axial images there is bilateral foraminal stenosis at C5/6 which is worse on the left due to low signal bulge posteriorly fromthe C5/6 disc. The C6/7 image also shows posterolateral bulge although it is higher signal than hte level above. This suggests higher water content and therefore may be a more recent disc prolapse than the less impressive protrusion above. This is in keeping with her symptoms- chronic and hence fairly stabel entrapment of C6 at C5/6, her recent exacerbation involving the whole hand due to an acute prolapse at C6/7 which has subsequently settled.
32
What are the managament options for cervical radiculopathy caused by disc degeneration.
Surgery should be directed at present symptoms rather than previous symptoms that have resolved or to prevent future disease.
33
What is the success of nerve root decompression of C6 root in the C5/6 foramen
Carries a high \>90% chance of improving pain if the correct level has been identified preoperatively.
34
What are the options for investigation if uncertainty remains over the culprit level for symptoms in cervical radiculopathy
Repeating MRI Electrophysiology CT-guided root infiltration of steroid and local anaesthetic.
35
What are the two surgical options for decompression in cervical radiculopathy
Anterior cervical discectomy. Posterior cervical foraminotomy.
36
A 72-year-old man is referred by his GP. He is normally healthy with a 6-month pro- gressive deterioration in mobility. He reports that his legs are strong and have normal sensation, but when he walks they feel heavy and become numb. He slows down after walking 300m and has to sit before continuing. He has no sphincter disturbance and no back pain. What other features of the history are important?
DDx is vascular or spinal claudication Vascular claudication is exercise dependent whereas spinal caludication is worse when lumbar lordosis is exaggerated by standing upright, causing buckling of the hypertrophic ligamentum flavum and nerve root compression. Vascular claudicants will still have symtpoms when riding on a bike whereas spinal claudicants will. Patients with lumbar stenosis will be able to walk for longer if leaning forward and may find walking uphill easier
37
A 72-year-old man is referred by his GP. He is normally healthy with a 6-month pro- gressive deterioration in mobility. He reports that his legs are strong and have normal sensation, but when he walks they feel heavy and become numb. He slows down after walking 300m and has to sit before continuing. He has no sphincter disturbance and no back pain. What would you look for on examination
There are frequently no positive findings on physical examinations. There may be dull lower limb reflxes. Peripheral foot pulses should be checked to rule out PVD
38
A 72-year-old man is referred by his GP. He is normally healthy with a 6-month pro- gressive deterioration in mobility. He reports that his legs are strong and have normal sensation, but when he walks they feel heavy and become numb. He slows down after walking 300m and has to sit before continuing. He has no sphincter disturbance and no back pain. There are no signs of vascular disease and the patient undergoes an MRI scan
There is evidence of marked degenerative disease of the lumbar spine. There is narrowing of the spinal canal at L4/5. On the axial scans the central part of the canal is patent but there is marked facet joint hypertrophy and hypertrophy of the ligamentum flavum causing stenosis of the lateral recesses of the spinal canl
39
What are the treatment options for spinal stenosis?
Symptoms usually worsen slowly in a predictable manner and surgery should be offered electively when the patient's symptoms become intolerable and oral analgesia is no longer sufficient. Surgery typically involves a laminectomy with undercutting of hypertrophic facet joints. The posterior midline elements are removed- spinous process, laminae and underlying ligamentum flavum and the anteromedial part of the facet joints that contacts the theca and compresses the nerve roots is removed to improve the dimensions of the spinal canal Severe monoradiculopathy in a patient with spinal stenosis may be managed with analgesia and nerve root injections
40
What are the risks of operative intervention for spinal stenosis
Hameorrhage and infection are uncommmon The risk of dural tear is higher than in lumbar disc surgery as the canal is narrowed and the dura may be stuck to the degenerate ligament. The risks of nerve root injury are also high, although still small. The risks of iatrogenic cauda equina syndrome are very low but would be severely disabling if occurred.
41
What is an alternative option to decompressive surgry in patients with spinal stenosis in whom symptoms are better with lumbar flexion
Surgical insertion of an interspinous spacer. Act at individual levels of the spine to wedge one or two segments of the lumbar spine in forward flexion thus preventing extension and buckling of the hypertrophic ligamentum flavum responsbile for stenotic symptoms. Efficacy can be limited but the procedure can be done under local.
42
What are the components innervating the bladder?
Efferent Afferent Central
43
Components of efferent innervation of the bladder
Sympathetic Parasympathetic Somatic
44
Sympathetic input on bladder
Relaxes detrusor, contract internal sphincter, allows filling
45
Parasympathetic input on bladder
Contracts detrusor, relaxes internal sphincter, empties bladder Parasympathetic- pisses
46
Nerve roots of sympathetic bladder innervation
T9-12
47
Nerve roots of parasympathetic innervation of bladder
S2-4
48
Somatic input on bladder
Voluntary control of EUS
49
Central control of micturition
Fibres from the cerebral cortex, BG and pontine micturition centre travel down the spinal cord in the medial and lateral reticulospinal tracts
50
Which bladder sphincter is under voluntary control
EUS
51
Impact of spinal cord lesions on micturition
Can interfere with central control of micturition by interrupting the micturition pathway. This may result in detrusor instability which results from disinhibition of detrusor muscle leading to urgency, urge incotninence and low residual volume or detrusor sphincter dyssnergia which results from involuntary detrusor contraction without relxation of the EUS leading to urgency with incomplete emptying. Detrusor-sphincter dyssynergia is most often seen with spinal cord lesions whereas detrusor hyper-reflexia can be seen in both SC and cortical or BG pathology including cerebrovascular diseaes, NPH and frontal lobe tumours
52
Disorders of micturition in cauda equina
Classically there is painless urinary retention and overflow incontinence due to interruption of the parasympathetic and somatic pathways to the bladder. Absent bladder sensation (can patient feel bladder distension or tug on catheter) suggets impaired sacral outflow. Intact sensation does not exclude impaired outflow as bladder efferents also travel with the sympathetic nerves. A full bladder with incontinence is consistent with cauda euqina. whereas an empty bladder is not. There are varying types of bladder dysfunction that have been noted to occur in cauda equina so there should be a low threshold for performing MRI. Acute back pain in itself can also cause urinary retention
53
A 55-year-old woman is referred by her GP with acute severe lumbar pain which had started 6 weeks previously whilst walking a friend’s dog when it lurched suddenly. Her symptoms were settling but there had been an exacerbation a week prior to this con- sultation, and she feels that she is as bad as she was initially. She has an antalgic gait and sits awkwardly, is not overweight, and localizes the pain to her lower lumbar area. Are there any red flag symptoms?
Not from the history
54
A 55-year-old woman is referred by her GP with acute severe lumbar pain which had started 6 weeks previously whilst walking a friend’s dog when it lurched suddenly. Her symptoms were settling but there had been an exacerbation a week prior to this con- sultation, and she feels that she is as bad as she was initially. She has an antalgic gait and sits awkwardly, is not overweight, and localizes the pain to her lower lumbar area. She is keen to have an MRI, what would you adivse?
The most recent NICE guidelines suggest non-sepcific low back pain (without red flag symptoms) is not well-evaluated with XR. MRI scanning is most unlikely to change her management which should be non-surgical in the expectation that her symptoms will improve
55
A 55-year-old woman is referred by her GP with acute severe lumbar pain which had started 6 weeks previously whilst walking a friend’s dog when it lurched suddenly. Her symptoms were settling but there had been an exacerbation a week prior to this con- sultation, and she feels that she is as bad as she was initially. She has an antalgic gait and sits awkwardly, is not overweight, and localizes the pain to her lower lumbar area. You advise her that an MRI is not appropriate. What would you suggest otherwise?
She should be advised that she will get better in time and conservative treamtnet. Exercise Analgesia PT structured exercise Non-weight bearing exercises e.g. swimming may be of value
56
A 55-year-old woman is referred by her GP with acute severe lumbar pain which had started 6 weeks previously whilst walking a friend’s dog when it lurched suddenly. Her symptoms were settling but there had been an exacerbation a week prior to this con- sultation, and she feels that she is as bad as she was initially. She has an antalgic gait and sits awkwardly, is not overweight, and localizes the pain to her lower lumbar area. She returns 2 weeks later with an MRI scan that her GP arranged (Fig. 51.1). What does it show and what are the options for her management?
The MRI shows loss of height of L5/S1 with a suggestion of posterior disc prolapse. This is confirmed on the axial image shows central disc herniation without neural compression. The options for her management would not change in view of this finding. The prolapse disc is likely to settle with conservative treatment. A lumbar discectomy would carry a much lower chance of improving her back pain than the chance of improving leg pain in association with disc prolapse
57
What is the use of spinal fusion in lower back pain
There is variable evidence to support it. The largest trial of surgery versus structured therapy found a minimal difference between the two groups. Well-selected patients may benefit from fusion surgery for persistent discogenic back pain that may require a discogram. Lumbar disc replacement has been used as an alternative to fusion.
58
A 77-year-old woman is referred to the outpatient clinic with back pain for 3 years and recent pain radiating from her back through her hips into her legs and feet. The pain is intermittent and occurs when she walks or stands. Within 5 minutes of walking she will be unable to continue because of pain. She does not have any bowel or bladder disturbance. Her past medical history includes hypertension and osteoporosis. An MRI scan of the lumbar spine is performed. The MRI is shown in Fig. 52.1. What does it show, and does it explain the symptoms?
There is spondylolisthesis: anterior displacement of L4 on the L5 vertebra. The L4/5 disc has lost height and protrudes posteriorly and there is marked thickening of the ligamentum flavum at this level. Patients often present with back pain and neurogenic claudication from compression of the exiting nerve roots. Symtpoms are worse on walaking or standing and are relieved by leaning forward which increases the diameter of the canal. A slip at L4/5 commonly affects L5 which is consistent with the patient's symptoms Significant compression of the thecal sac may cause compression of the cauda equina. The axial view at the level of the slip shows obliteration of the spinal canal though the patient does not have symptoms of that.
59
What is the use of the Meyerding and Wiltse-Newman-MacNab classifications?
Used for classification of spondylolisthesis Meyerding- degree of displacement Wiltse-Newman-MacNab- aetiology
60
Meyerding 1
25% displacement over vertebra below
61
Meyerding 2
50% displacement over verebra below
62
Meyerding 3
75% displacement over vertebra below
63
Meyerding 4
100% displacement
64
Wiltse-Newman-MacNab 1 grading of spondylolisthesis
Dysplastic (congenital)
65
Wiltse-Newman-MacNab 2 grading of spondylolisthesis
Isthmic- pars atricularis fracture
66
Wiltse-Newman-MacNab 3 grading of spondylolisthesis
Degenerative
67
Wiltse-Newman-MacNab 4 grading of spondylolisthesis
Traumatic
68
Wiltse-Newman-MacNab 5 grading of spondylolisthesis
Pathological
69
Management of spondylolisthesis
Operative or non-operative dependent on grade and symptoms. Non-operative mangement consists of analgseia, muscle relaxants and graded exercise. Surgery may be considered for persistent symptoms lasting over at least several weeks- the aim is to relieve pain and neurological deficit and prevent deformity progression. Posterior decompression alone or decompression with fusion/instrumentation
70
Myotomes Shoulder abduction
C5 6 Deltoid Axillary nerve
71
Myotomes Elbow flexion Supinated
C5 6 Biceps Musculocutaneous
72
Myotomes Elbow flexion (half pronated)
C5 6 Brachioradialis Radial
73
Myotomes Elbow extension
C6-C8 TRICEPS RADIAL
74
Myotomes Wrist extension
C7 Long extensors Radial nerve
75
Myotomes Finger flexion
C8 FDP Median 1-3, ulnar 3-4
76
Myotomes Finger abduction
T1 Abductor digiti minimi Ulnar
77
Myotomes Hip flexion
L1 2 Iliopsoas Femoral nerve
78
Myotomes Knee extension
L3 L4 Quadriceps Femoral nerve
79
Myotomes Dorsiflexion
L4 Tibialis anterior Deep peroneal nerve
80
Myotomes Big toe extension
L5 EHL Deep peroneal
81
Myotomes Plantar flexion
S1 2 Gastrocnemius, soleus Tibial nerve
82
Myotomes Knee flexion
L5-S1 Hamstrings Sciatic nerve
83
Myotomes Hip extension
L5-S2 Gluteus maximus Inferior gluteal nerve
84
Myotomes Hip adduction
L2-3 Adductors Obturator nerve
85
Myotomes Inversion
L4 5 Tibialis posterior Tibial nerve
86
Myotomes Eversion
L5, S1 Peroneus longus and brevis (lateral compartment of lower leg) Superficial peroneal nerve- eversion will be week in superficial peroneal nerve palsy but other L5 muscle groups will be strong, in an L5 lesion all groups will be weak including ankle eversion