Spinal cord injury CC4 Flashcards

(51 cards)

1
Q

acute consequences of sci

weakness with a neck injury leads to

A

quadriparesis/plegia

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2
Q

acute consequences of sci

weakness with a caudal injury leads to

A

paraparesis/plegia

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3
Q

htn or hypotension in acute phase of sci?

A

hypotension

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4
Q

urinary ___ in acute phase of sci

A

retention

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5
Q

Acute consequences of SCI? (5)

A

quadriparesis/plegia (neck) paraparesis/plegia (caudal)

Sensory loss or abnormalities

Hypotension

Urinary retention

Orthopedic pain

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6
Q

chornic consequencies of sci? (10

A
  1. involutnary mvoements
  2. bladder problems
  3. decreased bowel motiblity
  4. sexual dysfunction
  5. increased risk fo blood clots
  6. pressure ulcers
  7. autonomic dysfunction
  8. metabolic disorders
  9. musculoskeltal breakdown/fracures
  10. psych
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7
Q

above T10 injury , bladder problems are

A

spastic/neurogenic bladder (most common).

Can’t voluntarily relax the external urethral sphincter, so it stays tight as bladder fills.

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8
Q

below T12, bladder sx are

A

flaccid bladder, can’t contract for emptying

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9
Q

management of flaccid bladder

A

manage using…intermittent catheterization, condom catheter, indwelling/Foley
catheter, or suprapubic catheter

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10
Q

male sexual dysfunction?

A

Male, sperm viability is impaired / fertility is impaired.

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11
Q

female sexua problems?

A

Female, fertility is mostly unaffected but C-section delivery strongly recommended (b/c of autonomic dysreflexia)

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12
Q

Pressure ulcers from __

A

cutaneous ischemia

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13
Q

Autonomic Dysfunction: sympathetic damage = impaired ___

A

thermoregulation

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14
Q

* Autonomic Dysreflexia with SCI =

A

extreme blood pressure swings (240/160) + low heart rate

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15
Q

what causes autonomic dysreflexia?

A

occurs in response to strong afferent input, ex. pain or overly full bladder

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16
Q

what can happen with repeated occurenced of autonomic dysrefelxia?

A

chronic hypertension

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17
Q

metabolic disorders with spi are often due to

A

lifestyle difficuluties

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18
Q

___ sp injuries/year

A

10,000-12,000

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19
Q

> 50% of injuries are to ___ spine

A

cervical

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20
Q

> 50% are neurologically “incomplete” - means there is sensation

A

in S4-S5 region.

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21
Q

“complete” means bilateral injury with total loss of

A

sensation below injury level

22
Q

ASIA scale: A is ___ injury

A

complete/worst injury

23
Q

ASIA sale B-D is ___

24
Q

ASIA scale e is __

25
Three approaches to SCI treatment:
1. Neuroprotection of **surviving cells/axons from toxic injury environment** 2. Neurorestoration **via replacement cells, neurotrophins, and establishing a growth-** permissive environment 3. Neurorehabilitation by **strengthening existing systems and retraining circuits**
26
methylpredinisolone (steroids) trials results
1. found higher mortality in higher tx group 2. another found asia scale improvement in pts who were incomplete, 24 hr dosing 3. 48 hr dosing beter
27
parapesis/plegia occurs caudal to \_\_
t1
28
for\_\_\_that are crushed by the intial truamtic injury nothing can be done those are gone for food
nerve cell bodies
29
among pts who started tx ___ of injury the 48 MP group receovered signfiicantly more funciton at 6 weeksn and 6 months than those with 24 MP
3-8
30
Spontaneous ____ movements seen in patient with C5 incomplete SCI for 17 years
stepping
31
Spontaneous stepping movements seen in patient with C5 incomplete SCI for 17 years Caused by arthritic degeneration and subluxation in R. hip ---\> ___ triggeered CPG
pain upon hip extension
32
Characteristics of all these walking CPG manifestations: Always associated with\_\_\_nput to the spinal cord
pathology / noxious i
33
Can be “trained” to improve ___ walking in persons with incomplete SCI
voluntary
34
Studies on Walk-Training with Body-Weight Support Most subjects show some improvement, but
no greater than seen with physical therapy
35
Studies on Walk-Training with Body-Weight Support is wlaking cpg being trained?
no evidence
36
Studies on Walk-Training with Body-Weight Support where were impovements seen?
better balance, stronger leg muscles, improved fitness
37
Autonomic Dysreflexia Typically seen after ___ or ___ SCI
cervical or high-thoracic
38
Autonomic Dysreflexia is more common after ___ sci
compelte
39
Potential causes of autonomic dysreflexia? (4)
1. loss of brain/brainstem inhibition to symps 2. excessive sensory response to stimuli 3. excessive sympathetic response to normal sensory input 4. excessive vascular response to normal sympathetic activity
40
in all cases of cpg walking all were rhythmic but not reciprocal
between agonists and anatogists
41
electrical stim to ___ can give rise to leg emg comparable to cpg results
lumbar enlargement
42
what si the cpg in human characterized by?
43
interlimb reflexes are observed in all persons with
chronic cervical sci
44
interlimb reflex is more common in
distal upper limb muscles
45
is interlibm reflex found in able bodied subjects?
no
46
early interlimb reflexes from
conenctions already established (latents syanspes)
47
late interlimb reflexes are from ___ connections
novel (regenerative sproutin)
48
do interlimb reflexes disappear?
no
49
what happens to interlimb reflexes once they appear
50
interlimb reflexes are liekly due to new growth within the spinal cord \_\_\_
caudal to the lesion
51
interlimb reflexes are possibly the basis for
autonomic dysreflexia