Neonatal emergencies Flashcards

1
Q

The most common congenital defect of the esophagus is

A

esophageal atresia

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2
Q

A key diagnostic indicator tor tracheoesophageal fistula is

A

maternal polyhydramnios

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3
Q

The most common type of tracheoesophageal fistula is

A

type C (90%)

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4
Q

Tracheoesophageal fistula may occur as part of

A

the VACTERL association

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5
Q

Approximately 20% of neonates with esophageal atresia have

A

a significant cardiac defect

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6
Q

Anesthetic considerations for esophageal atresia & tracheoesophageal fistula include

A

head up position and frequent suctioning minimizes the risk of gastric aspiration
maintain spontaneous ventilation during induction (PPV can cause gastric distension)
a precordial stethoscope placed on the left chest will immediately detect a right mainstem intubation

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7
Q

Where should the ETT be placed for a tracheoesophageal fistula?

A

place it below the fistula but above the carina

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8
Q

Most kids with esophageal atresia will have

A

a tracheoesophageal fistula

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9
Q

Diagnosis of esophageal atresia is confirmed by

A

the inability to pass a gastric tube into the stomach

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10
Q

Other symptoms of esophageal atresia include

A

choking
coughing
cyanosis during oral feeding

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11
Q

What is VACTERL association?

A

vertebral defects
imperforated anus
cardiac anomalies
tracheoesophageal fistula
esophageal atresia
renal dysplasia
limb anomalies

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12
Q

If an ETT is placed too high with tracheoesophageal fistula then

A

respiratory gas is delivered to the stomach

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13
Q

If an ETT is placed too low with tracheoesophageal fistula then

A

endobronchial intubation is likely

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14
Q

Placement of a g-tube with tracheoesophageal fistula allows for

A

gastric decompression with induction

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15
Q

Which lecithin/sphingomyelin ratio suggests fetal lung maturity?
a. 0.5
b. 1.0
c. 1.5
d. 2.0

A

d. 2.0

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16
Q

Type 2 pneumocytes begin producing surfactant between ___________ with peak production occurring at _______________

A

22-26 weeks; 35-36 weeks

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17
Q

Neonates who do not produce enough surfactant are at risk for

A

respiratory distress syndrome

18
Q

Patients with respiratory distress syndrome are at high risk for

A

hypoxemia
hypercarbia
mixed acidosis
hemodynamic instability
death

19
Q

Positive pressure ventilation in the patient with poor lung compliance

A

increases the risk of pneumothorax

20
Q

Before delivery, treatment for respiratory distress syndrome includes

A

maternal corticosteroids to hasten fetal lung maturity

21
Q

After delivery, treatment for respiratory distress syndrome includes

A

CPAP
mechanical ventilation
exogenous surfactant

22
Q

Preductal and postductal oxygen saturation monitoring should be used to assess for

A

pulmonary hypertension
a right-to-left cardiac shunt and the return to fetal circulation via the PDA

23
Q

The tendency of an alveolus to collapse is directly proportional to

A

surface tension

24
Q

The tendency of an alveolus to collapse is inversely proportional to

A

alveolar radius (smaller radius= more likely to collapse)

25
Q

The steroid used to hasten lung maturity is

A

betamethasone

26
Q

Risk factors for respiratory distress syndrome include

A

low birth weight
low gestational age
barotrauma from positive pressure ventilation
oxygen toxicity
endotracheal intubation
maternal diabetes

27
Q

Anesthetic considerations for respiratory distress syndrome include that SpO2 should be

A

maintained while keeping in mind that hyperoxia can contribute to retinopathy of prematurity and poor lung development

28
Q

Where should an arterial line be placed in the neonate with RDS?

A

preductal artery preferred

29
Q

Where should the preductal and postductal pulse oximetry be placed?

A

preductal: right upper extremity
postductal: lower extremity

30
Q

What cells produce surfactant?

A

type 2 pneumocytes

31
Q

________________ allows the abdominal contents to enter the thoracic cavity

A

Congenital diaphragmatic hernia

32
Q

The most common site of herniation is

A

the foramen of Bochdalek (usually on the left)

33
Q

The mass effect of the abdominal contents within the chest

A

impairs lung development leading to pulmonary hypoplasia

34
Q

Consequences of poor lung development related to CDH include

A

poor pulmonary vascular development
increased pulmonary vascular resistance
pulmonary hypertension
impaired airway development
airway reactivity

35
Q

What should be avoided in terms of anesthetic management for the patient with congenital diaphragmatic hernia?

A

keep PIP <25-30 cm H2O to minimize barotrauma and the risk of pneumothorax in the “good” lung
avoid conditions that increase PVR (hypoxia, acidosis, hypothermia)

36
Q

When is surgery performed for congenital diaphragmatic hernia?

A

delayed 5-15 days to allow for stabilization of pulmonary, cardiac, and metabolic status

37
Q

What is necessary for anesthetic management of the patient with a congenital diaphragmatic hernia?

A

monitor preductal oxygen saturation
one-lung ventilation with a single lumen ETT advanced into the good lung may be required

38
Q

Other sites of herniation for a congenital diaphragmatic hernia include

A

foramen of Morgagni and around the esophagus

39
Q

CDH is usually diagnosed

A

at birth

40
Q

The newborn with CDH will experience

A

respiratory distress
scaphoid abdomen (sunken in)
barrel chest
cardiac displacement
fluid-filled GI segments in the thorax

41
Q

With CDH, a pulse oximeter placed on a lower extremity can

A

warn of increased intra-abdominal pressure

42
Q

With CDH, preductal SpO2 should be

A

> 90%