Neonatal emergencies Flashcards

(42 cards)

1
Q

The most common congenital defect of the esophagus is

A

esophageal atresia

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2
Q

A key diagnostic indicator tor tracheoesophageal fistula is

A

maternal polyhydramnios

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3
Q

The most common type of tracheoesophageal fistula is

A

type C (90%)

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4
Q

Tracheoesophageal fistula may occur as part of

A

the VACTERL association

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5
Q

Approximately 20% of neonates with esophageal atresia have

A

a significant cardiac defect

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6
Q

Anesthetic considerations for esophageal atresia & tracheoesophageal fistula include

A

head up position and frequent suctioning minimizes the risk of gastric aspiration
maintain spontaneous ventilation during induction (PPV can cause gastric distension)
a precordial stethoscope placed on the left chest will immediately detect a right mainstem intubation

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7
Q

Where should the ETT be placed for a tracheoesophageal fistula?

A

place it below the fistula but above the carina

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8
Q

Most kids with esophageal atresia will have

A

a tracheoesophageal fistula

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9
Q

Diagnosis of esophageal atresia is confirmed by

A

the inability to pass a gastric tube into the stomach

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10
Q

Other symptoms of esophageal atresia include

A

choking
coughing
cyanosis during oral feeding

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11
Q

What is VACTERL association?

A

vertebral defects
imperforated anus
cardiac anomalies
tracheoesophageal fistula
esophageal atresia
renal dysplasia
limb anomalies

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12
Q

If an ETT is placed too high with tracheoesophageal fistula then

A

respiratory gas is delivered to the stomach

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13
Q

If an ETT is placed too low with tracheoesophageal fistula then

A

endobronchial intubation is likely

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14
Q

Placement of a g-tube with tracheoesophageal fistula allows for

A

gastric decompression with induction

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15
Q

Which lecithin/sphingomyelin ratio suggests fetal lung maturity?
a. 0.5
b. 1.0
c. 1.5
d. 2.0

A

d. 2.0

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16
Q

Type 2 pneumocytes begin producing surfactant between ___________ with peak production occurring at _______________

A

22-26 weeks; 35-36 weeks

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17
Q

Neonates who do not produce enough surfactant are at risk for

A

respiratory distress syndrome

18
Q

Patients with respiratory distress syndrome are at high risk for

A

hypoxemia
hypercarbia
mixed acidosis
hemodynamic instability
death

19
Q

Positive pressure ventilation in the patient with poor lung compliance

A

increases the risk of pneumothorax

20
Q

Before delivery, treatment for respiratory distress syndrome includes

A

maternal corticosteroids to hasten fetal lung maturity

21
Q

After delivery, treatment for respiratory distress syndrome includes

A

CPAP
mechanical ventilation
exogenous surfactant

22
Q

Preductal and postductal oxygen saturation monitoring should be used to assess for

A

pulmonary hypertension
a right-to-left cardiac shunt and the return to fetal circulation via the PDA

23
Q

The tendency of an alveolus to collapse is directly proportional to

A

surface tension

24
Q

The tendency of an alveolus to collapse is inversely proportional to

A

alveolar radius (smaller radius= more likely to collapse)

25
The steroid used to hasten lung maturity is
betamethasone
26
Risk factors for respiratory distress syndrome include
low birth weight low gestational age barotrauma from positive pressure ventilation oxygen toxicity endotracheal intubation maternal diabetes
27
Anesthetic considerations for respiratory distress syndrome include that SpO2 should be
maintained while keeping in mind that hyperoxia can contribute to retinopathy of prematurity and poor lung development
28
Where should an arterial line be placed in the neonate with RDS?
preductal artery preferred
29
Where should the preductal and postductal pulse oximetry be placed?
preductal: right upper extremity postductal: lower extremity
30
What cells produce surfactant?
type 2 pneumocytes
31
________________ allows the abdominal contents to enter the thoracic cavity
Congenital diaphragmatic hernia
32
The most common site of herniation is
the foramen of Bochdalek (usually on the left)
33
The mass effect of the abdominal contents within the chest
impairs lung development leading to pulmonary hypoplasia
34
Consequences of poor lung development related to CDH include
poor pulmonary vascular development increased pulmonary vascular resistance pulmonary hypertension impaired airway development airway reactivity
35
What should be avoided in terms of anesthetic management for the patient with congenital diaphragmatic hernia?
keep PIP <25-30 cm H2O to minimize barotrauma and the risk of pneumothorax in the "good" lung avoid conditions that increase PVR (hypoxia, acidosis, hypothermia)
36
When is surgery performed for congenital diaphragmatic hernia?
delayed 5-15 days to allow for stabilization of pulmonary, cardiac, and metabolic status
37
What is necessary for anesthetic management of the patient with a congenital diaphragmatic hernia?
monitor preductal oxygen saturation one-lung ventilation with a single lumen ETT advanced into the good lung may be required
38
Other sites of herniation for a congenital diaphragmatic hernia include
foramen of Morgagni and around the esophagus
39
CDH is usually diagnosed
at birth
40
The newborn with CDH will experience
respiratory distress scaphoid abdomen (sunken in) barrel chest cardiac displacement fluid-filled GI segments in the thorax
41
With CDH, a pulse oximeter placed on a lower extremity can
warn of increased intra-abdominal pressure
42
With CDH, preductal SpO2 should be
>90%