Lower GI Flashcards

(105 cards)

1
Q

microbiome

A

vitamin K absorption

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2
Q

absorption and reabsorption - how many liters are produced in 24 hours?

A

Saliva, gastric, bile, pancreas, small bowel, all produce GI fluids
7 liters of fluids in 24/hours
All but 100 mL is reabsorbed terminal ileum

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3
Q

crohn’s disease - what side? (lee leans to the right)

A

right

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4
Q

diverticulitis - what side?

A

left

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5
Q

important labs (4, 3, 2)

A

K (at 4), Phos (at 3), mag (at 2)

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6
Q

lactic acid tells us if

A

someone is septic

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7
Q

pre-albumin is

A

recent nutrition

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8
Q

albumin is

A

nutrition over several months

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9
Q

pancreas enzymes

A

amalyse and lypase

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10
Q

ABG tests for

A

lactate

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11
Q

occult blood

A

blood you can’t see, the test is a guiac

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12
Q

barium enema (are clear liquids ok?)

A

pt needs to be able to swallow, but can be given via tube. clear liquids. may give laxatives, enema. if they can’t drink, give liquids IV. make sure barium comes out the other end.

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13
Q

gastroscopy - NPO for how long? And sedation?

A

NPO 6-8 hrs, bowel prep, conscious sedation, aspiration is a risk. ex. endoscopy, EGD. should be totally awake afterwards.

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14
Q

stool

A

FIT, FOBT, iFOBT, C and S, and O & P

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15
Q

constipation

A

Defined as fewer than three bowel movements weekly or bowel movements that are hard, dry, small, or difficult to pass

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16
Q

constipation causes

A

meds, chronic laxative use, immobility, fatigue, can’t increase abdomen pressure, diet, ignoring urge, lack of exercise

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17
Q

perceived constipation

A

subjective, when person’s elimination is not consistent w/ what is believed to be normal

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18
Q

constipation symptoms

A

less than 3 a week, distention and bloating, sensation of not evacuating, straining, small dry stools

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19
Q

cause of chronic constipation

A

usually idiopathic

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20
Q

diagnostics for constipation (constipation is defcon 1)

A

MRI, Defecography and colonic transit studies

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21
Q

constipation complications (constipation/mega)

A

decreased C/O, fecal impaction, hemorrhage, fissures, prolapse, megacolon (abnormal dilation)

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22
Q

types of diarrhea (diarrhea pac)

A

acute, persistent, or chronic

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23
Q

diarrhea causes

A

infections, medications, tube feeding formulas, metabolic and endocrine disorders, and various disease processes

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24
Q

manifestations of diarrhea

A

Increased frequency and fluid content of stools
Abdominal cramps
Distention
Borborygmus (grumbling)
Anorexia and thirst
Painful spasmodic contractions of the anus
Tenesmus (feeling like you need to have a bowel movement)

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25
C.diff
About 1 in 6 patients who get C. diff will get it again in the subsequent 2-8 weeks. One in 11 people over age 65 diagnosed with a healthcare-associated C. diff infection die within one month.
26
fecal incontinence - causes
anal sphincter weakness, trauma, neuropathies, pelvic floor disorders, CNS disorders, diarrhea, behavioral disorders
27
gastroparesis (is it painful?)
Motor and nerve functions of digestive organs are impaired Accompanied by pain
28
gastroparesis - Weakness of stomach
muscles cannot fully aid in the digestive process. Delayed stomach emptying.
29
gastroparesis - causes (paralyzed by diabetes and opioids)
DM, electrolyte imbalances (K+, Mg, Ca+), opioids, hypothyroid vagus nerve damage, pancreatitis, scleroderma
30
gastroparesis - S & Sx (think full)
bloating, abdominal distention, early satiety, abdominal pain, vomiting (large pieces undigested food), GERD, malnutrition
31
treatment for gastroparesis - how long to sit upright? and what type of diet? (slow needs low fiber and low fat)
Control N, V, abdominal pain Nutrition/Diet –low fiber, low fat, soft or liquid with good hydration, MVI to prevent malnutrition, position upright after meals 4-5 hours, eat early in the day
32
gastroparesis - diagnostics
gastric emptying study, antro-duodenal motility study (measures strength of contractions); electrogastrogram (measures electrical voltage of contractions, endoscopy, CT
33
meds for gastroparesis
Promotility medications in liquid form –metoclopramide, domperidone, erythromycin, SQ octreotide
34
lowe GI bleed - causes - but esp which one?
Diverticula IBD Crohn’s (some with crohns). UC (especially UC), gastroenteritis Perianal disorders Hemorrhoids Carcinoma AV malformation
35
Hemorrhoids - does bleeding hurt?
can be internal or external, bleeding is usually painless.
36
external hemmoroids usually
itchy. bleeding is like a streak. some can prolapse during defacation and then back to normal.
37
hemmoroids treatment
cold baths, topicals to shrink, stool softeners, topical nitroglycerin.
37
hemmoroids treatment
cold baths, topicals to shrink, stool softeners, topical nitroglycerin.
38
Diverticula - type of bleeding (D for dieverticula, D for dark)
Diverticula - Sustained dark bleeding
39
IBD - type of bleeding (I for IBD, I for intermittent)
IBD -Intermittent bleeding & frequent BM’s
40
Perianal disorders - type of bleeding (perianal is bright)
BRB per rectum
41
Cancer - type of bleeding(cancer is the occult)
CA - Occult bleeding with intermittent melena
42
AV malformations - type of bleeding (AVs go either way)
AV malformations - Intermittent BRB (bright red blood), dark with clots from cecum
43
perforation - symptoms
Symptoms include: severe abdominal pain, cramping, N/V, bleeding, tenderness to palpation, distention, rigidity, fever/chills, change in stool
44
Stomach perforation pain is
sudden
45
Colon perforation may come on (colon is slow)
more gradually
46
perforation etiologies
PUD, forceful vomiting, UC, Crohn’s, diverticulitis, toxic megacolon, strangulated hernia, bowel ischemia, bowel obstruction, abdominal surgery (anastamotic leaks), trauma, foreign body, corrosive substance ingestion, appendicitis
47
2nd most common cause of sepsis
peritonitis (#1 cause organ perforation)
48
peritonitis (peri inflames sara)
inflammation of the serosal membrane that lines the abdominal cavity
49
peritonitis - causes
perforation - bile from perforated gall bladder, liver laceration, gastric acid from perforated ulcer, surgical leak, foreign bodies, ectopic pregnancy, ruptured ovarian cyst or appendix, infected PD catheter or G-J Tube
50
peritonitis - s & sx - early and then later (and what relieves it?) (peri hurts my shoulder)
Early vague, diffuse pain, may become sharp and radiate to shoulder; progresses with intensity & is worse with movement, might be slightly relieved with fetal position
51
peritonitis - gold standard for diagnosis
CT scan
52
one way to define peritonitis is (peri has a high WBC - what number)
WBCs >500 with peritoneal lavage
53
peritonitis - interventions
Minimize complications-hypoxia, shock, AKI/ARF, sepsis, acidosis Supplemental oxygen Bowel rest NPO, NGT suction (low to continous suction) Maintain FEN/Acid-base balance IV hydration Electrolyte additives/replacements Caloric supplementation (PPN, TPN)
54
peritonitis surgeries
Exploratory lap, open lavage, I & D abscess, lysis of adhesions, resections of tumors or c=necrosis, temporary or permanent fecal diversions, drains
55
peritonitis - post op
ABC Prevent pulmonary complications, dehydration and malnutrition Positioning, oxygen, pulmonary toilet, mobility Identify infection early, antibiotics Hydration Acid-base/FEN Wound drainage/management (tubes, drains, positioning) Pain/anxiety management (motility compromise) Maintain mobility
56
sepsis - what is the mortality rate? (not quite 50)
distributive shock, 30% mortality. 50% of survivors have post-sepsis syndrom.
57
sepsis shock
Inadequate vessel tone Blood pressure is a function of cardiac output and systemic vascular resistance (SVR). If SVR decreases from vasodilation, blood pressure decreases. Massive vasodilation can occur from loss of sympathetic nervous system function or chemicals released within the body. Bacteria and toxins in the blood lead to vasodilation and increased capillary permeability. Treatment includes vasoconstriction and volume restoration.
58
post sepsis syndrome - causes
Causes of PSS DIC, decreased CO, amputations, ARDS, pulmonary edema, ATN/ARI, liver
59
post sepsis syndrome - symptoms (like ptsd)
Both physical and psychological long-term effects Sleep pattern disturbances, nightmares, hallucinations, panic attacks, PTSD Disabling muscle or joint pain Difficulty concentration, decreased cognitive ability Loss of self esteem, depression Hair loss Chronic fatigue High risk for viral respiratory infections
60
IBS - more common in men or women?
2x more in women
61
IBS triggers (not so much foods)
chronic stress, sleep deprivation, surgery, infections, diverticulitis, and some foods
62
IBS manifestions (IB bloated)
Alteration in bowel patterns Pain Bloating Abdominal distention
63
IBS diagnostics
Stool studies Contrast radiography studies Proctoscopy Barium enema Colonoscopy Manometry Electromyography
64
malabsorption - conditions
Mucosal (transport) disorders Infectious disease Luminal disorders Postoperative malabsorption Disorders that cause malabsorption of specific nutrients
65
malabsorption manifestations
Symptoms similar to irritable bowel syndrome Manifested by weight loss and vitamin and mineral deficiency
66
gluten is found in
Gluten is most commonly found in wheat, barley, rye, and other grains malt, dextrin, and brewer’s yeast.
67
celiac can lead to (Celia and hashi are friends)
hashimotos
68
celiac disease - manifestations
Diarrhea Steatorrhea Abdominal pain Abdominal distention Flatulence Weight loss
69
inflammatory bowel disease - 2 types
UC and Crohns
70
UC - where is it located? (U see it's just large)
Ulcerative IBD that is just in the large intestines
71
crohns - inflammation where? and where is it found? (Crohn's hangs out w/ illeana)
Also called regional enteritis Inflammation and erosion of the ileum Can be found throughout the GI tract and anywhere
72
S/Sx of IBD (IB rebound cheese w/ anemia)
N, V D Weight loss Cramping Rectal bleeding Dehydration Hematochezia Anemia Fever Rebound tenderness
73
can UC be cured?
yes, removal of part of colon
74
can Crohn's be cured?
no
75
more bleeding with UC or crohns?
UC
76
IBD treatment (nothing really)
Hydration Pain control Diet Correct anemia Education Support
77
IBD meds
Antidiarrheals (Only given with mildly symptomatic UC) Antibiotics Sulfonamides (for Peritonitis) Steroids (To decrease inflammation) Immune system suppressors Potentiate the effects of corticosteroids 6 mercaptopurine & azathioprine
78
IBD - Immune system suppressors
Potentiate the effects of corticosteroids 6 mercaptopurine & azathioprine
79
IBD meds (IB ending in mab)
Bind to TNF Remicade (infliximab) & Humira (adalimab), Stelara (ustekinumab)
80
Diverticular Disorders - what part of colon? (sigfried descends on his dive)
Major cause of lower GI bleed Diverticula are small herniations in the bowel wall caused by weakness in sigmoid or descending colon
81
dDiverticular Disorders - risk factors (just 2 things) (ol and constipated is the big d)
> 60 years, constipation
82
Diverticular Disorders - complications
Diverticulitis inflammation/infection Obstruction, rupture Bleeding, perforation Intra-abdominal abscess Adhesions, fistulas
83
divertula - treatment (divers need fiber)
keep things moving, give fiber, exercise
84
diverticulitis - what to eat? (dive softly w/ food)
soft diet, avoid inflammation
85
diverticular disorders - pathos
herniation of colonic mucosa.. outpouchings form..fecal material trapped…microperforation…contamination of surrounding tissues…inflammatory response…abscess formation… may erode vessels… bleeding…perforation
86
diverticular disorders - S/Sx - where is the pain? (Dive to the left, it's worse after you eat)
flares/remissions; constipation, diarrhea, flatulence; pain is acute, LLQ, and worst after meals; relieved with BM; rectal bleeding; fever/chills
87
diverticular disorders - diagnostics (dive cutie)
history/physical, CT, abdominal ultrasound, CBC with diff
88
diverticular disorders - prevention - fiber good or bad?
Prevention High fiber diet, increase bulk of stools with colloid laxative During “flare”: AVOID high fiber. Pain management (anticholenergics; minimize opiates ABX Bowel rest from clears for 48 hours to NPO and IV hydration
89
diverticular disorders - surgeries (drain the pool before diving)
abscess drainage, resections, temporary fecal diversion, repair/manage fistulas, relieve obstructions
90
Acute paralytic ileus (adynamic ileus)
results from loss of intestinal peristalsis
91
Acute paralytic ileus (don't touch a cute illeana)
Causes: handling of the bowel during surgery, trauma, electrolyte disturbances, intestinal ischemia, infections, peritonitis, sepsis, toxic metabolic conditions, pain, spinal cord trauma/lesions and medications that decrease gastric motility
92
Ogilvie’s syndrome (olga is dilated)
Massive dilation NO peristalsis
93
illeus - s/sx
Pain, dehydration, vomiting (not prominent), abdominal distention & tenderness, hiccoughs, decreased/absent bowel sounds, no flatus/stool
94
illeus - complications
Obstruction, delay/inability to ingest food/fluids, pain, delayed healing, peritonitis
95
illeus - does it resolve on its own? (Illeana helps herself)
Usually self limiting (2-3 days) Correct underlying cause Bowel rest (NPO, NGT LC/I WS (wall suction) fluids Increase mobility (ambulate) Increase GI motility (warm, medications, guided imagery and other integrative approaches Wean from opioids or oral naloxone
96
intestinal bowel obstruction - what about the kidneys?
Partial or complete Mechanical Non-Mechanical Vascular If unresolved can lead to AKI d/t 3rd spacing, hypovolemia; bowel perforations, bowel ischemia/infarction, `peritonitis, sepsis, death
97
most bowel obstructions occur in
the small intestines
98
bowel obstruction
Distention of gas and fluid occurs proximal to the distention.Trapped fluid and electrolytes leak out into the peritoneal cavity. inflammation causes na to get stuck. strangulation, perforation or cardiovascular collapse are are worst case scenarios.
99
bowel obstruction - fluid replacement
keep them euvolemic (equal fluids)
100
bowel obstruction - risks
Motility problems Elderly Postoperative Bedridden Multisystem dysfunction Multiple abdominal or bowel surgeries adhesions
101
bowel obstruction - treatments
FLUIDS Isotonic NPO NGT I & O Antibiotics for strangulation Surgical consultation KUB Abdominal CT
102
gastroparesis - who is at risk?
Patient with diabetes, surgical anastamoses and certain viral conditionsp are at greatest risk for developing gastroparesis
103
peritonitis - hypo or hypervolemia? any distention? (you've lost the fluid, so...)
Abdominal distention, A/N/V decreased bowel sounds Dehydration/hypovolemia (HR up, BP down) with hemoconcentration (elevated HCT, Na+, BUN) Acidosis, elevated WBC Fever, chills Free air on KUB, CT with perforation
104
gastroparesis symptoms (full and paralyzed)
Patients report feeling full and tired, nauseated and often vomit