3.2.4 Dysfunctional Families Evaluation (Sch) Flashcards Preview

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Flashcards in 3.2.4 Dysfunctional Families Evaluation (Sch) Deck (32):
1

(lack of research support) many studies highlight what?

communication difficulties

2

(lack of research support) there is lack of research to support what theory?

double bind communication

3

(lack of research support) Liem gave structured tasks to parents of who?

11 sons with schizophrenia
11 sons without

4

(lack of research support) Liem found what between the parents communication with schizophrenic and non schizophrenic sons?

communication was no more disordered

5

(lack of research support) Liem's research caused him to question what?

cause or effect relationship

6

(lack of research support) does double bind cause schizophrenia or does?

schizophrenia cause double bind

7

(lack of research support) if the theory was correct what would you expect?

all/most children in the family to develop schizophrenia

8

(origins) to be a complete explanation what needs to be offered?

the origins of dysfunctional communications

9

(origins) Koopmans - double bind communication arises at times of what?

family disruption

10

(origins) Koopmans - what is there little evidence of?

empirical research support between disruption and double bind communication

11

(origins) Koopmans - there is also an ethical issue of what?

blaming patients/their families

12

(origins) it has been suggested double bind communications are a symptom of what?

mental illness/pathology

13

(origins) it may be pathology that is a greater issue than what?

communications

14

(origins) what does the lack of origins do to this explanation?

weakens it

15

(origins) Bateson didn't claim double bind was a whole explanation rather he said it was what?

a risk factor

16

(EE research support) there is a lot of evidence to support the role of expression of emotion in what?

relapse of schizophrenia

17

(EE research support) Vaughan and Leff - in high EE homes with high contact and no medication, what was the rate of relapse?

90%

18

(EE research support) Vaughan and Leff - in low EE homes what was the rate of relapse?

10%

19

(EE research support) Vaughan and Leff - in high EE homes what was the rate of relapse?

50%

20

(EE research support) in prospective studies what were high risk children?

those with a parent with schizophrenia

21

(EE research support) high risk children who developed schizophrenia were more likely to come from where?

high EE homes characterised by negative relationships

22

(EE research support) Marcus - Israeli high risk study - none of the high risk children with good parenting did what?

developed schizophrenia

23

(EE research support) Kavanagh - people with schizophrenia in high risk families were how many times more likely to relapse than those in low EE families?

4x

24

(EE research support) not all research supports EE as a predictor of what?

relapse

25

(EE research support) McCreadie and Philips failed to find what?

increase in 6 and 12 month relapse rates among individuals with schizophrenia living in high risk homes

26

(EE research support) McCreadie and Philips research suggest although EE may be a significant relapse factor in some, it is not what?

the only one

27

(genes or environment) what is it difficult to separate?

whether it is the result of families having the same genes or same environment

28

(genes or environment) the schizophrenia working group of psychiatric genomics consitorium have found what?

108 genetic loci associated with schizophrenia

29

(genes or environment) which explanation has more supporting evidence?

genetics

30

(genes or environment) it may be dysfunctional communication is what?

a CV

31

(genes or environment) genetics leads to poor mental health which then leads to what?

poor communication

32

(conclusion) what would be a better way to look at schizophrenia?

diathesis stress model
genes and environment both contribute

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